scholarly article | Q13442814 |
P50 | author | Matteo Bordoni | Q58952002 |
Lorenzo Drufuca | Q95990317 | ||
Cristina Cereda | Q37842077 | ||
P2093 | author name string | Stella Gagliardi | |
Mauro Ceroni | |||
Orietta Pansarasa | |||
Luca Diamanti | |||
Stefano Bernuzzi | |||
Sabrina La Salvia | |||
Daisy Sproviero | |||
Rosa Trotti | |||
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Inside out: the role of nucleocytoplasmic transport in ALS and FTLD | Q26748482 | ||
Rapid detection of octamer binding proteins with 'mini-extracts', prepared from a small number of cells | Q27860599 | ||
Cell death: critical control points | Q28240722 | ||
Mitochondrial dysfunction and oxidative stress in neurodegenerative diseases | Q28269333 | ||
Effect of nitric oxide on lymphocytes from sporadic amyotrophic lateral sclerosis patients: toxic or protective role? | Q28275312 | ||
SOD1 mRNA expression in sporadic amyotrophic lateral sclerosis | Q28279971 | ||
FUS Interacts with HSP60 to Promote Mitochondrial Damage | Q28547727 | ||
Enhancing Mitofusin/Marf ameliorates neuromuscular dysfunction in Drosophila models of TDP-43 proteinopathies. | Q50784802 | ||
Altered age-related changes in bioenergetic properties and mitochondrial morphology in fibroblasts from sporadic amyotrophic lateral sclerosis patients. | Q51597460 | ||
Skeletal Muscle Is a Primary Target of SOD1G93A-Mediated Toxicity | Q62397259 | ||
Morphological abnormalities in mitochondria of the skin of patients with sporadic amyotrophic lateral sclerosis | Q83178534 | ||
TDP-43 and FUS: a nuclear affair | Q84433614 | ||
Amyotrophic lateral sclerosis multiprotein biomarkers in peripheral blood mononuclear cells | Q28744223 | ||
El Escorial revisited: revised criteria for the diagnosis of amyotrophic lateral sclerosis | Q29619074 | ||
Abnormal mitochondrial transport and morphology are common pathological denominators in SOD1 and TDP43 ALS mouse models | Q30572264 | ||
Focal degeneration of astrocytes in amyotrophic lateral sclerosis. | Q33350786 | ||
In vivo accumulation of Helicobacter pylori products, NOD1, ubiquitinated proteins and proteasome in a novel cytoplasmic structure | Q33543097 | ||
Mitochondrial dysfunction and intracellular calcium dysregulation in ALS | Q34103782 | ||
Protein aggregation and degradation mechanisms in neurodegenerative diseases | Q34334363 | ||
Wild-type human TDP-43 expression causes TDP-43 phosphorylation, mitochondrial aggregation, motor deficits, and early mortality in transgenic mice | Q34664325 | ||
Altered intracellular localization of SOD1 in leukocytes from patients with sporadic amyotrophic lateral sclerosis | Q35025204 | ||
An over-oxidized form of superoxide dismutase found in sporadic amyotrophic lateral sclerosis with bulbar onset shares a toxic mechanism with mutant SOD1 | Q35882558 | ||
Early and gender-specific differences in spinal cord mitochondrial function and oxidative stress markers in a mouse model of ALS | Q35891904 | ||
TDP-43 and FUS en route from the nucleus to the cytoplasm | Q36336278 | ||
Nonnative SOD1 trimer is toxic to motor neurons in a model of amyotrophic lateral sclerosis | Q36498352 | ||
The ALS disease-associated mutant TDP-43 impairs mitochondrial dynamics and function in motor neurons | Q37286990 | ||
Mitochondria-associated membrane collapse is a common pathomechanism in SIGMAR1- and SOD1-linked ALS | Q37514803 | ||
Non-cell autonomous toxicity in neurodegenerative disorders: ALS and beyond | Q37642283 | ||
Protein misdirection inside and outside motor neurons in Amyotrophic Lateral Sclerosis (ALS): a possible clue for therapeutic strategies | Q37954610 | ||
Mitochondrial dynamics in neurodegeneration | Q38060435 | ||
The changing scene of amyotrophic lateral sclerosis | Q38086771 | ||
Role of protein misfolding and proteostasis deficiency in protein misfolding diseases and aging | Q38172142 | ||
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Disturbed mitochondrial dynamics and neurodegenerative disorders | Q38285228 | ||
Misfolded SOD1 Accumulation and Mitochondrial Association Contribute to the Selective Vulnerability of Motor Neurons in Familial ALS: Correlation to Human Disease | Q38675517 | ||
Gene-specific mitochondria dysfunctions in human TARDBP and C9ORF72 fibroblasts. | Q38772926 | ||
Mutant SOD1G93A triggers mitochondrial fragmentation in spinal cord motor neurons: neuroprotection by SIRT3 and PGC-1α. | Q38844786 | ||
Overexpression of nuclear FUS induces neuronal cell death | Q38929777 | ||
Decoding ALS: from genes to mechanism. | Q39004348 | ||
Metabolic Dysfunctions in Amyotrophic Lateral Sclerosis Pathogenesis and Potential Metabolic Treatments | Q39101800 | ||
TDP-43 and Cytoskeletal Proteins in ALS. | Q39279950 | ||
The role of mitochondria in amyotrophic lateral sclerosis | Q39409879 | ||
The ALS-linked E102Q mutation in Sigma receptor-1 leads to ER stress-mediated defects in protein homeostasis and dysregulation of RNA-binding proteins. | Q41636922 | ||
An EBV-transformed owl monkey B-lymphocyte cell line | Q42814431 | ||
In vitro studies in VCP-associated multisystem proteinopathy suggest altered mitochondrial bioenergetics | Q46148248 | ||
Lon protease: a novel mitochondrial matrix protein in the interconnection between drug-induced mitochondrial dysfunction and endoplasmic reticulum stress. | Q47757120 | ||
P275 | copyright license | Creative Commons Attribution 3.0 Unported | Q14947546 |
P6216 | copyright status | copyrighted | Q50423863 |
P4510 | describes a project that uses | ImageJ | Q1659584 |
P433 | issue | 3 | |
P921 | main subject | amyotrophic lateral sclerosis | Q206901 |
P577 | publication date | 2018-03-26 | |
P1433 | published in | Disease Models & Mechanisms | Q1524006 |
P1476 | title | Lymphoblastoid cell lines as a model to understand amyotrophic lateral sclerosis disease mechanisms | |
P478 | volume | 11 |
Q64072993 | Extensive epigenetic and transcriptomic variability between genetically identical human B-lymphoblastoid cells with implications in pharmacogenomics research |
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