scholarly article | Q13442814 |
P356 | DOI | 10.1016/J.BBI.2017.06.002 |
P698 | PubMed publication ID | 28610747 |
P50 | author | Jessica L. Teeling | Q42325972 |
Junjun Ni | Q57653216 | ||
P2093 | author name string | Hiroshi Nakanishi | |
Zhou Wu | |||
Fumiko Takayama | |||
Yicong Liu | |||
Alex Collcutt | |||
Paul Ibbett | |||
P921 | main subject | lipopolysaccharide | Q421804 |
Porphyromonas gingivalis | Q3214147 | ||
P304 | page(s) | 350-361 | |
P577 | publication date | 2017-06-10 | |
P1433 | published in | Brain, Behavior, and Immunity | Q15716593 |
P1476 | title | Cathepsin B plays a critical role in inducing Alzheimer's disease-like phenotypes following chronic systemic exposure to lipopolysaccharide from Porphyromonas gingivalis in mice | |
P478 | volume | 65 |
Q93018574 | An impaired intrinsic microglial clock system induces neuroinflammatory alterations in the early stage of amyloid precursor protein knock-in mouse brain |
Q61807350 | Assessing the role of in periodontitis to determine a causative relationship with Alzheimer's disease |
Q57174872 | Can an Infection Hypothesis Explain the Beta Amyloid Hypothesis of Alzheimer's Disease? |
Q99711216 | Chronic Periodontitis and Alzheimer Disease: A Putative Link of Serum Proteins Identification by 2D-DIGE Proteomics |
Q57040842 | Chronic oral application of a periodontal pathogen results in brain inflammation, neurodegeneration and amyloid beta production in wild type mice |
Q64361136 | Genetic, Transcriptome, Proteomic, and Epidemiological Evidence for Blood-Brain Barrier Disruption and Polymicrobial Brain Invasion as Determinant Factors in Alzheimer's Disease |
Q90725224 | Implications of Systemic Inflammation and Periodontitis for Major Depression |
Q53704723 | Importance of heterogeneity in Porhyromonas gingivalis lipopolysaccharide lipid A in tissue specific inflammatory signalling. |
Q61814461 | Increased expression and altered subcellular distribution of cathepsin B in microglia induce cognitive impairment through oxidative stress and inflammatory response in mice |
Q41717940 | Infection of microglia with Porphyromonas gingivalis promotes cell migration and an inflammatory response through the gingipain-mediated activation of protease-activated receptor-2 in mice. |
Q55024687 | Inhibition of Cathepsins B Induces Neuroprotection Against Secondary Degeneration in Ipsilateral Substantia Nigra After Focal Cortical Infarction in Adult Male Rats. |
Q92358830 | Is there a link between genetic defects in the complement cascade and Porphyromonas gingivalis in Alzheimer's disease? |
Q51764702 | Lipopolysaccharide Associates with Amyloid Plaques, Neurons and Oligodendrocytes in Alzheimer's Disease Brain: A Review. |
Q64939013 | Menaquinone-4 Suppresses Lipopolysaccharide-Induced Inflammation in MG6 Mouse Microglia-Derived Cells by Inhibiting the NF-κB Signaling Pathway. |
Q90175392 | Microglial cathepsin B as a key driver of inflammatory brain diseases and brain aging |
Q98205070 | Molecular linkage between post-traumatic stress disorder and cognitive impairment: a targeted proteomics study of World Trade Center responders |
Q97882878 | Periodontitis Induced by P. gingivalis-LPS Is Associated With Neuroinflammation and Learning and Memory Impairment in Sprague-Dawley Rats |
Q47428289 | Periodontitis, Microbiomes and their Role in Alzheimer's Disease |
Q64114001 | Serotype b of triggers pro-inflammatory responses and amyloid beta secretion in hippocampal cells: a novel link between periodontitis and Alzheimer´s disease? |
Q92223760 | Systems-wide analysis unravels the new roles of CCM signal complex (CSC) |
Q94545415 | TLR4 Cross-Talk With NLRP3 Inflammasome and Complement Signaling Pathways in Alzheimer's Disease |
Q47194761 | The Porphyromonas gingivalis/Host Interactome Shows Enrichment in GWASdb Genes Related to Alzheimer's Disease, Diabetes and Cardiovascular Diseases. |
Q92315939 | The Possible Causal Link of Periodontitis to Neuropsychiatric Disorders: More Than Psychosocial Mechanisms |
Q89698133 | The Role of Periodontitis and Periodontal Bacteria in the Onset and Progression of Alzheimer's Disease: A Systematic Review |
Search more.