| scholarly article | Q13442814 |
| P50 | author | Lucila Guadalupe Caceres | Q79694964 |
| P2093 | author name string | Francisco Capani | |
| Juan I Romero | |||
| Laura R Guelman | |||
| Soledad L Uran | |||
| María A Zorrilla Zubilete | |||
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| The assessment of non-feminizing estrogens for use in neuroprotection | Q34624609 | ||
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| The antioxidant neuroprotective effects of estrogens and phenolic compounds are independent from their estrogenic properties | Q35586685 | ||
| Structure-nongenomic neuroprotection relationship of estrogens and estrogen-derived compounds | Q35867501 | ||
| Acute pretreatment with estradiol protects against CA1 cell loss and spatial learning impairments resulting from transient global ischemia | Q35870685 | ||
| Estrogen and the brain: beyond ER-alpha and ER-beta | Q35974805 | ||
| Quinol-based cyclic antioxidant mechanism in estrogen neuroprotection | Q36161592 | ||
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| Gonadal hormone modulation of neurogenesis in the dentate gyrus of adult male and female rodents. | Q36899285 | ||
| The organizational-activational hypothesis as the foundation for a unified theory of sexual differentiation of all mammalian tissues | Q36900866 | ||
| 17alpha-Estradiol is neuroprotective in male and female rats in a model of early brain injury | Q36961292 | ||
| Contribution of estrogen receptors alpha and beta to the effects of estradiol in the brain. | Q36971282 | ||
| Direct binding and activation of protein kinase C isoforms by steroid hormones | Q37105428 | ||
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| The antioxidant effect of estrogen and Selective Estrogen Receptor Modulators in the inhibition of osteocyte apoptosis in vitro | Q40194824 | ||
| 17beta-oestradiol up-regulates longevity-related, antioxidant enzyme expression via the ERK1 and ERK2[MAPK]/NFkappaB cascade | Q40415424 | ||
| Role of hippocampal signaling pathways in long-term memory formation of a nonassociative learning task in the rat | Q40416447 | ||
| Free radicals in biology: oxidative stress and the effects of ionizing radiation | Q40620886 | ||
| Neuroprotective effects of a novel non-receptor-binding estrogen analogue: in vitro and in vivo analysis | Q40699740 | ||
| Hippocampal-related memory deficits and histological damage induced by neonatal ionizing radiation exposure. Role of oxidative status. | Q42641274 | ||
| Estradiol therapy in adulthood reverses glial and neuronal alterations caused by perinatal asphyxia | Q43141483 | ||
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| Motor, cytoarchitectural and biochemical assessment of pharmacological neuroprotection against CNS damage induced by neonatal exposure to ionizing radiation | Q43657143 | ||
| WR-2721 (amifostine, ethyol) prevents motor and morphological changes induced by neonatal X-irradiation | Q44268136 | ||
| Involvement of insulin/phosphoinositide 3-kinase/Akt signal pathway in 17 beta-estradiol-mediated neuroprotection | Q44719217 | ||
| Deferoxamine antioxidant activity on cerebellar granule cells gamma-irradiated in vitro | Q44869071 | ||
| Neuroprotective effects of estradiol in newborn female rat hippocampus | Q44906432 | ||
| The 17alpha and 17beta isomers of estradiol both induce rapid spine synapse formation in the CA1 hippocampal subfield of ovariectomized female rats. | Q45108198 | ||
| P433 | issue | 4 | |
| P407 | language of work or name | English | Q1860 |
| P304 | page(s) | 626-635 | |
| P577 | publication date | 2011-06-24 | |
| P1433 | published in | Journal of Neurochemistry | Q6295643 |
| P1476 | title | An early treatment with 17-β-estradiol is neuroprotective against the long-term effects of neonatal ionizing radiation exposure | |
| P478 | volume | 118 |
| Q37184525 | Assessment of Neuroprotective Effects of Local Administration of 17- Beta- Estradiol on Peripheral Nerve Regeneration in Ovariectomized Female Rats |
| Q39020032 | GANRA-5 protects both cultured cells and mice from various radiation types by functioning as a free radical scavenger |
| Q30416021 | Protective actions of 17β-estradiol and progesterone on oxidative neuronal injury induced by organometallic compounds. |
| Q46007615 | Radioprotective agents to prevent cellular damage due to ionizing radiation. |
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