Improvement of cognitive deficits in SAMP8 mice by 3-n-butylphthalide.

scientific article published in March 2014

Improvement of cognitive deficits in SAMP8 mice by 3-n-butylphthalide. is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1179/1743132813Y.0000000280
P698PubMed publication ID24512016

P50authorZun-Ji KeQ41658092
P2093author name stringH Chen
F Wang
X J Sun
P2860cites workNeuropathological alterations in Alzheimer diseaseQ27003314
l-3-n-Butylphthalide improves cognitive impairment induced by chronic cerebral hypoperfusion in ratsQ28567272
Treatment of Alzheimer's diseaseQ33779004
Senescence-accelerated mouse (SAM) with special references to neurodegeneration models, SAMP8 and SAMP10 miceQ37387075
The senescence accelerated mouse (SAMP8) as a model for oxidative stress and Alzheimer's diseaseQ37964816
The SAMP8 mouse: a model to develop therapeutic interventions for Alzheimer's diseaseQ37979825
Senescence-accelerated mouse (SAM): a novel murine model of senescenceQ41411648
Pathobiology of the senescence-accelerated mouse (SAM).Q41411678
3-n-Butylphthalide (NBP) reduces apoptosis and enhances vascular endothelial growth factor (VEGF) up-regulation in diabetic ratsQ43059949
Oligomeric proanthocyanidins improve memory and enhance phosphorylation of vascular endothelial growth factor receptor-2 in senescence-accelerated mouse prone/8.Q43263201
Magnolol and honokiol prevent learning and memory impairment and cholinergic deficit in SAMP8 miceQ43265027
Reversal of thiamine deficiency-induced neurodegenerationQ44309699
Markers for biogenic amines in the aged rat brain: relationship to decline in spatial learning abilityQ44799559
Long-term ginsenoside consumption prevents memory loss in aged SAMP8 mice by decreasing oxidative stress and up-regulating the plasticity-related proteins in hippocampusQ46169706
Improved mitochondrial function and increased life span after chronic melatonin treatment in senescent prone mice.Q46591210
Acupuncture improves cognitive deficits and regulates the brain cell proliferation of SAMP8 miceQ46794420
Age-related spatial cognitive impairment is correlated with a decrease in ChAT in the cerebral cortex, hippocampus and forebrain of SAMP8 miceQ47839441
Effects of an acidic fibroblast growth factor fragment analog on learning and memory and on medial septum cholinergic neurons in senescence-accelerated miceQ48150175
Senescence-accelerated mouse (SAM) as an animal model of senile dementia: pharmacological, neurochemical and molecular biological approachQ48298991
Functional relationship between age-related immunodeficiency and learning deteriorationQ48315813
Cholinergic deficits in the septal-hippocampal pathway of the SAM-P/8 senescence accelerated mouseQ48360229
Lipid peroxidation in brain during aging in the senescence-accelerated mouse (SAM).Q48469494
Peripheral inflammatory mechanisms modulate microglial activation in response to mild impairment of oxidative metabolismQ48494614
The biological sciences section program at the 60th Annual Meeting of the Gerontological Society of America.Q51116075
A single nucleotide polymorphism in CHAT influences response to acetylcholinesterase inhibitors in Alzheimer's disease.Q51922147
Spatial learning in transgenic mice expressing human presenilin 1 (PS1) transgenes.Q52025498
Impairment of spatial memory and changes in astroglial responsiveness following loss of molar teeth in aged SAMP8 mice.Q52028632
Age-related changes in learning and memory and cholinergic neuronal function in senescence accelerated mice (SAM).Q52050540
Degenerative Changes in Forebrain Cholinergic Nuclei Correlate with Cognitive Impairments in Aged Rats.Q52118014
P433issue3
P304page(s)224-233
P577publication date2014-03-01
P1433published inNeurological ResearchQ15765622
P1476titleImprovement of cognitive deficits in SAMP8 mice by 3-n-butylphthalide.
P478volume36

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cites work (P2860)
Q34548561A Review of Recent Advances in Neuroprotective Potential of 3-N-Butylphthalide and Its Derivatives
Q90351907Long-Term DL-3-n-Butylphthalide Treatment Alleviates Cognitive Impairment Correlate With Improving Synaptic Plasticity in SAMP8 Mice

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