Changes in vimentin, lamin A/C and mitofilin induce aberrant cell organization in fibroblasts from Fanconi anemia complementation group A (FA-A) patients.

scientific article published on 2 July 2013

Changes in vimentin, lamin A/C and mitofilin induce aberrant cell organization in fibroblasts from Fanconi anemia complementation group A (FA-A) patients. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1016/J.BIOCHI.2013.06.024
P698PubMed publication ID23831462

P50authorMaurizio BruschiQ37841639
Giovanni CandianoQ37841656
Silvia RaveraQ42412845
Cristina CapanniQ43166626
Andrea PetrettoQ55325244
Enrico CappelliQ57065263
P2093author name stringCarlo Dufour
Paola Cuccarolo
Paolo Degan
Marta Columbaro
P433issue10
P921main subjectFanconi anemia complementation group AQ32147067
P304page(s)1838-1847
P577publication date2013-07-02
P1433published inBiochimieQ2904035
P1476titleChanges in vimentin, lamin A/C and mitofilin induce aberrant cell organization in fibroblasts from Fanconi anemia complementation group A (FA-A) patients.
P478volume95

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cites work (P2860)
Q35020589Dysregulated Ca2+ homeostasis in Fanconi anemia cells
Q34261778High-risk human papillomavirus E6 protein promotes reprogramming of Fanconi anemia patient cells through repression of p53 but does not allow for sustained growth of induced pluripotent stem cells
Q47262970Hypomorphic FANCA mutations correlate with mild mitochondrial and clinical phenotype in Fanconi anemia.
Q57590763Inhibition of Metalloproteinase Activity in FANCA Is Linked to Altered Oxygen Metabolism
Q91797290Mitochondrial inner membrane protein, Mic60/mitofilin in mammalian organ protection
Q99629112The passage from bone marrow niche to bloodstream triggers the metabolic impairment in Fanconi Anemia mononuclear cells
Q28541992Treatment of FANCA cells with resveratrol and N-acetylcysteine: a comparative study
Q41352828Why is an energy metabolic defect the common outcome in BMFS?

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