Dose-related cytogenetic damage in pulmonary alveolar macrophages from mice exposed to cigarette smoke early in life.

scientific article published on 12 October 2011

Dose-related cytogenetic damage in pulmonary alveolar macrophages from mice exposed to cigarette smoke early in life. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1007/S00204-011-0765-3
P698PubMed publication ID21989788
P5875ResearchGate publication ID226425441

P2093author name stringVernon E Steele
Silvio De Flora
Francesco D'Agostini
Roumen Balansky
Rosanna T Micale
Sebastiano La Maestra
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Reactivity of mouse alveolar macrophages to cigarette smoke is strain dependentQ42467292
An in vitro micronucleus assay with size-classified micronucleus counting to discriminate aneugens from clastogensQ43277834
Systemic genotoxic effects produced by light, and synergism with cigarette smoke in the respiratory tract of hairless miceQ44502546
Birth-related genomic and transcriptional changes in mouse lung. Modulation by transplacental N-acetylcysteineQ44673260
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Budesonide and phenethyl isothiocyanate attenuate DNA damage in bronchoalveolar lavage cells of mice exposed to environmental cigarette smokeQ46201750
Genotoxicity of 10 cigarette smoke condensates in four test systems: comparisons between assays and condensatesQ46897969
Type II epithelial cells are critical target for hyperoxia-mediated impairment of postnatal lung development.Q50721829
Influence of glutathione S-transferase polymorphisms on genotoxic effects induced by tobacco smoke.Q51745307
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Human pulmonary alveolar macrophages metabolise benzo(a)pyrene to proximate and ultimate mutagensQ59099327
P433issue3
P304page(s)509-516
P577publication date2011-10-12
P1433published inArchives of ToxicologyQ635837
P1476titleDose-related cytogenetic damage in pulmonary alveolar macrophages from mice exposed to cigarette smoke early in life.
P478volume86

Reverse relations

cites work (P2860)
Q39336757Transcriptomics in developmental toxicity testing
Q34540931Whole cigarette smoke increased the expression of TLRs, HBDs, and proinflammory cytokines by human gingival epithelial cells through different signaling pathways

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