Skin Barrier Defects Caused by Keratinocyte-Specific Deletion of ADAM17 or EGFR Are Based on Highly Similar Proteome and Degradome Alterations.

scientific article published on 11 April 2016

Skin Barrier Defects Caused by Keratinocyte-Specific Deletion of ADAM17 or EGFR Are Based on Highly Similar Proteome and Degradome Alterations. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1021/ACS.JPROTEOME.5B00691
P8608Fatcat IDrelease_xzo2ujmmivhmzbx635dfmofgka
P698PubMed publication ID27089454

P50authorOliver SchillingQ61159147
Jayachandran N KizhakkedathuQ88055357
P2093author name stringStefan Tholen
Martin L Biniossek
Claus-Werner Franzke
Bettina Mayer
Stefanie Löffek
Cristina Wolf
Yawen Qian
Julia D Knopf
P2860cites workProteomeXchange provides globally coordinated proteomics data submission and disseminationQ29614806
ProteoWizard: open source software for rapid proteomics tools developmentQ33349681
P433issue5
P921main subjectkeratinocyteQ1473931
P304page(s)1402-1417
P577publication date2016-04-21
P1433published inJournal of Proteome ResearchQ3186939
P1476titleSkin Barrier Defects Caused by Keratinocyte-Specific Deletion of ADAM17 or EGFR Are Based on Highly Similar Proteome and Degradome Alterations
P478volume15

Reverse relations

cites work (P2860)
Q89721924ADAM17 stabilizes its interacting partner inactive Rhomboid 2 (iRhom2) but not inactive Rhomboid 1 (iRhom1)
Q39683133ADAM17/EGFR axis promotes transglutaminase-dependent skin barrier formation through phosholipase C γ1 and protein kinase C pathways
Q53497674Differential effect of TGFβ on the proteome of cancer associated fibroblasts and cancer epithelial cells in a co-culture approach - a short report.
Q64105690EGFR Controls Hair Shaft Differentiation in a p53-Independent Manner
Q90754117Microbe-host interplay in atopic dermatitis and psoriasis
Q28069428Proteomic Substrate Identification for Membrane Proteases in the Brain

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