| scholarly article | Q13442814 |
| P2093 | author name string | MacDonald HR | |
| Rufer N | |||
| Migliaccio M | |||
| Bianchi T | |||
| P2860 | cites work | Intrathymic and intrasplenic oxidative stress mediates thymocyte and splenocyte damage in acutely exercised mice | Q77880320 |
| Mechanisms that limit the in vitro proliferative potential of human CD8+ T lymphocytes | Q81482588 | ||
| The INK4 family of cell cycle inhibitors in cancer | Q28145080 | ||
| Tumor suppression by Ink4a-Arf: progress and puzzles | Q28207959 | ||
| Glucocorticoids in T cell development and function*. | Q33932702 | ||
| A syndrome of multiorgan hyperplasia with features of gigantism, tumorigenesis, and female sterility in p27(Kip1)-deficient mice | Q34381923 | ||
| Putting the stress on senescence | Q34430830 | ||
| Two adjacent trimeric Fas ligands are required for Fas signaling and formation of a death-inducing signaling complex | Q34472898 | ||
| Studies of T-cell activation in chronic inflammation | Q34731209 | ||
| CDK inhibitors p18(INK4c) and p27(Kip1) mediate two separate pathways to collaboratively suppress pituitary tumorigenesis | Q35209756 | ||
| Control of T cell viability | Q35698562 | ||
| p16(Ink4a) interferes with Abelson virus transformation by enhancing apoptosis | Q37010207 | ||
| Loss of proliferative capacity and induction of senescence in oxidatively stressed human fibroblasts | Q40514053 | ||
| The tumor suppressor p16(INK4a) gene is a regulator of apoptosis induced by ultraviolet light and cisplatin | Q40539192 | ||
| Stress-induced premature senescence in BJ and hTERT-BJ1 human foreskin fibroblasts | Q40717611 | ||
| CD95/Fas signaling in T lymphocytes induces the cell cycle control protein p21cip-1/WAF-1, which promotes apoptosis | Q42832518 | ||
| Loss of p16Ink4a with retention of p19Arf predisposes mice to tumorigenesis | Q43729274 | ||
| BCL-2 is involved in preventing oxidant-induced cell death and in decreasing oxygen radical production. | Q43897073 | ||
| P16(INK4A) is implicated in both the immediate and adaptative response of human keratinocytes to UVB irradiation | Q43966862 | ||
| Oxidative-stress-induced T lymphocyte hyporesponsiveness is caused by structural modification rather than proteasomal degradation of crucial TCR signaling molecules. | Q44528095 | ||
| N-Acetyl-L-cysteine prevents exercise-induced intestinal lymphocyte apoptosis by maintaining intracellular glutathione levels and reducing mitochondrial membrane depolarization | Q44926931 | ||
| Rap1 signaling is required for suppression of Ras-generated reactive oxygen species and protection against oxidative stress in T lymphocytes | Q44968886 | ||
| Inhibition of CREB transcriptional activity in human T lymphocytes by oxidative stress. | Q46510475 | ||
| The role of phase II antioxidant enzymes in protecting memory T cells from spontaneous apoptosis in young and old mice | Q46664087 | ||
| Inactivation of the cyclin-dependent kinase inhibitor p15INK4b by deletion and de novo methylation with independence of p16INK4a alterations in murine primary T-cell lymphomas. | Q48052432 | ||
| Expression of the p16INK4a tumor suppressor versus other INK4 family members during mouse development and aging. | Q52526599 | ||
| The tumor-suppressive functions of the human INK4A locus | Q57187959 | ||
| Transgenic expression of the p16(INK4a) cyclin-dependent kinase inhibitor leads to enhanced apoptosis and differentiation arrest of CD4-CD8- immature thymocytes | Q77671503 | ||
| P433 | issue | 29 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | lymphocyte | Q715347 |
| P304 | page(s) | 4110-4115 | |
| P577 | publication date | 2006-02-20 | |
| P1433 | published in | Oncogene | Q1568657 |
| P1476 | title | The tumor suppressor p16Ink4a regulates T lymphocyte survival. | |
| P478 | volume | 25 |
| Q34634251 | Acute lymphoblastic leukemia cells that survive combination chemotherapy in vivo remain sensitive to allogeneic immune effects |
| Q28731544 | Bone marrow p16INK4a-deficiency does not modulate obesity, glucose homeostasis or atherosclerosis development |
| Q38156169 | EBV finds a polycomb-mediated, epigenetic solution to the problem of oncogenic stress responses triggered by infection |
| Q36092170 | Evidence of accelerated ageing in clinical drug addiction from immune, hepatic and metabolic biomarkers |
| Q34754281 | Expression of p16(INK4a) prevents cancer and promotes aging in lymphocytes |
| Q36057582 | Fibroblast growth factor-7 partially reverses murine thymocyte progenitor aging by repression of Ink4a |
| Q26853420 | Genetic regulation of thymocyte progenitor aging |
| Q36839913 | High-risk acute lymphoblastic leukemia cells with bcr-abl and INK4A/ARF mutations retain susceptibility to alloreactive T cells |
| Q24322656 | Identification and characterization of a novel protein ISOC2 that interacts with p16INK4a |
| Q31111648 | Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines |
| Q39231572 | Menin Plays a Critical Role in the Regulation of the Antigen-Specific CD8+ T Cell Response upon Listeria Infection. |
| Q92977910 | Photosynthesis-inspired H2 generation using a chlorophyll-loaded liposomal nanoplatform to detect and scavenge excess ROS |
| Q36684482 | Promises and limitations of murine models in the development of anticancer T-cell vaccines |
| Q37522773 | Tumor suppressor mechanisms in immune aging |
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