Hyporesponsiveness of donor cells to lipopolysaccharide stimulation reduces the severity of experimental idiopathic pneumonia syndrome: potential role for a gut-lung axis of inflammation.

scientific article published in December 2000

Hyporesponsiveness of donor cells to lipopolysaccharide stimulation reduces the severity of experimental idiopathic pneumonia syndrome: potential role for a gut-lung axis of inflammation. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.4049/JIMMUNOL.165.11.6612
P698PubMed publication ID11086106
P5875ResearchGate publication ID12241921

P2093author name stringCrawford JM
Martin TR
Kobzik L
Ferrara JL
Brewer JP
Hill GR
Cooke KR
Gerbitz A
P2860cites workDefective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 GeneQ22299417
P433issue11
P407language of work or nameEnglishQ1860
P921main subjectinflammationQ101991
pneumoniaQ12192
donor cellQ107637879
lipopolysaccharideQ421804
Idiopathic pneumonia syndromeQ5988895
P304page(s)6612-6619
P577publication date2000-12-01
P1433published inJournal of ImmunologyQ3521441
P1476titleHyporesponsiveness of donor cells to lipopolysaccharide stimulation reduces the severity of experimental idiopathic pneumonia syndrome: potential role for a gut-lung axis of inflammation.
P478volume165

Reverse relations

cites work (P2860)
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