N-cadherin mediates angiogenesis by regulating monocyte chemoattractant protein-1 expression via PI3K/Akt signaling in prostate cancer cells.

scientific article

N-cadherin mediates angiogenesis by regulating monocyte chemoattractant protein-1 expression via PI3K/Akt signaling in prostate cancer cells. is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1016/J.YEXCR.2011.07.024
P698PubMed publication ID21855541

P50authorJasti S. RaoQ108443825
P2093author name stringArun Kumar Nalla
Jitendra Patel
Norman Estes
P2860cites workDifferential localization of VE- and N-cadherins in human endothelial cells: VE-cadherin competes with N-cadherin for junctional localizationQ24683420
Monocyte chemotactic protein (MCP)-1 promotes angiogenesis via a novel transcription factor, MCP-1-induced protein (MCPIP)Q28115465
N-cadherin acts upstream of VE-cadherin in controlling vascular morphogenesisQ28505270
Role of cadherins in the transendothelial migration of melanoma cells in cultureQ32162592
Metastatic patterns of prostate cancer: an autopsy study of 1,589 patientsQ33904020
Disparate E-cadherin mutations in LCIS and associated invasive breast carcinomasQ33918637
AKT plays a central role in tumorigenesisQ33944165
The role of endothelial cells in tumor invasion and metastasisQ34294331
Purified N-cadherin is a potent substrate for the rapid induction of neurite outgrowthQ36222783
Epithelial-mesenchymal transition in development and cancer: role of phosphatidylinositol 3' kinase/AKT pathwaysQ36313178
Akt-regulated pathways in prostate cancerQ36313185
Exogenous expression of N-cadherin in breast cancer cells induces cell migration, invasion, and metastasisQ36316373
The role of tumor microenvironment in prostate cancer bone metastasisQ36771551
The role of epithelial-mesenchymal transition in cancer pathologyQ36845018
HAb18G/CD147 promotes epithelial-mesenchymal transition through TGF-β signaling and is transcriptionally regulated by SlugQ39550377
CD151 amplifies signaling by integrin α6β1 to PI3K and induces the epithelial-mesenchymal transition in HCC cellsQ39592399
Downregulation of N-cadherin expression inhibits invasiveness, arrests cell cycle and induces cell apoptosis in esophageal squamous cell carcinomaQ39762637
Phospho-Akt pathway activation and inhibition depends on N-cadherin or phospho-EGFR expression in invasive human bladder cancer cell lines.Q39907005
Activation of MCP-1/CCR2 axis promotes prostate cancer growth in boneQ39918594
Soluble N-cadherin fragment promotes angiogenesisQ40222376
Twist overexpression correlates with hepatocellular carcinoma metastasis through induction of epithelial-mesenchymal transition.Q40227825
Prostate cancer progression into androgen independency is associated with alterations in cell adhesion and invasivityQ40240573
Novel expression of N-cadherin elicits in vitro bladder cell invasion via the Akt signaling pathway.Q40560244
N-Cadherin expression in human prostate carcinoma cell lines. An epithelial-mesenchymal transformation mediating adhesion withStromal cells.Q40928947
Phosphatidylinositol 3-kinase function is required for transforming growth factor beta-mediated epithelial to mesenchymal transition and cell migration.Q42492769
A switch from E-cadherin to N-cadherin expression indicates epithelial to mesenchymal transition and is of strong and independent importance for the progress of prostate cancerQ42522113
Twist regulates cytokine gene expression through a negative feedback loop that represses NF-kappaB activityQ47614351
N-cadherin-mediated adhesion and aberrant catenin expression in anaplastic thyroid-carcinoma cell lines.Q52536650
Expression of N-cadherin adhesion molecules associated with early morphogenetic events in chick developmentQ59053468
Complex cadherin expression in human prostate cancer cellsQ73396798
Cell-cell interactions during transendothelial migration of tumor cellsQ77640053
Changes in cadherin-catenin complexes in the progression of human bladder carcinomaQ77849608
Monocyte chemoattractant protein-1-induced angiogenesis is mediated by vascular endothelial growth factor-AQ80894668
N-cadherin switching occurs in high Gleason grade prostate cancerQ81237591
P433issue17
P407language of work or nameEnglishQ1860
P921main subjectprostate cancerQ181257
P304page(s)2512-2521
P577publication date2011-08-09
P1433published inExperimental Cell ResearchQ1524289
P1476titleN-cadherin mediates angiogenesis by regulating monocyte chemoattractant protein-1 expression via PI3K/Akt signaling in prostate cancer cells.
P478volume317

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cites work (P2860)
Q58607358Activation of hepatocyte growth factor/MET signaling initiates oncogenic transformation and enhances tumoraggressiveness in the murine prostate
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Q34713218Constitutively active androgen receptor variants upregulate expression of mesenchymal markers in prostate cancer cells
Q36105331E-cadherin-dependent stimulation of traction force at focal adhesions via the Src and PI3K signaling pathways.
Q42031689ED-B fibronectin expression is a marker of epithelial-mesenchymal transition in translational oncology.
Q36561957Effect of SIRT1 Gene on Epithelial-Mesenchymal Transition of Human Prostate Cancer PC-3 Cells
Q28481696Extract of Pleurotus pulmonarius suppresses liver cancer development and progression through inhibition of VEGF-induced PI3K/AKT signaling pathway
Q48211076GP73 promotes invasion and metastasis of bladder cancer by regulating the epithelial-mesenchymal transition through the TGF-β1/Smad2 signalling pathway.
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Q55227135MicroRNA-126 inhibits proliferation and metastasis in prostate cancer via regulation of ADAM9.
Q34631703N-cadherin expression is associated with acquisition of EMT phenotype and with enhanced invasion in erlotinib-resistant lung cancer cell lines
Q58067565Perturbations of BMP/TGF-β and VEGF/VEGFR signalling pathways in non-syndromic sporadic brain arteriovenous malformations (BAVM)
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Q64098698Suppressive Role of Androgen/Androgen Receptor Signaling via Chemokines on Prostate Cancer Cells
Q38697619Tumor matrix stiffness promotes metastatic cancer cell interaction with the endothelium.
Q51765575Tumor microenvironment promotes prostate cancer cell dissemination via the Akt/mTOR pathway.

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