Pancreatic insulinomas in multiple endocrine neoplasia, type I knockout mice can develop in the absence of chromosome instability or microsatellite instability.

scientific article published in October 2004

Pancreatic insulinomas in multiple endocrine neoplasia, type I knockout mice can develop in the absence of chromosome instability or microsatellite instability. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1158/0008-5472.CAN-04-1648
P8608Fatcat IDrelease_gtxh557yizbqfk5ntcg7vhxnmi
P698PubMed publication ID15466197
P5875ResearchGate publication ID8249851

P50authorJoe W. GrayQ85648340
Koei ChinQ114345970
Francis CollinsQ336658
P2093author name stringMeghan T Miller
Allen M Spiegel
Stephen J Marx
Peter C Scacheri
J Graeme Hodgson
Alyssa L Kennedy
P433issue19
P407language of work or nameEnglishQ1860
P921main subjectknockout mouseQ1364740
multiple endocrine neoplasiaQ1553018
microsatelliteQ265193
P304page(s)7039-7044
P577publication date2004-10-01
P1433published inCancer ResearchQ326097
P1476titlePancreatic insulinomas in multiple endocrine neoplasia, type I knockout mice can develop in the absence of chromosome instability or microsatellite instability.
P478volume64

Reverse relations

cites work (P2860)
Q35082433Exploring the tumors of multiple endocrine neoplasia type 1 in mouse models for basic and preclinical studies
Q21145267Genome-wide analysis of menin binding provides insights into MEN1 tumorigenesis
Q36707655In search of tumor suppressing functions of menin
Q36514229MEN1 and FANCD2 mediate distinct mechanisms of DNA crosslink repair
Q24337658Menin and MLL cooperatively regulate expression of cyclin-dependent kinase inhibitors
Q36111935Molecular genetics of multiple endocrine neoplasia types 1 and 2.
Q64996730Ribozyme-mediated compensatory induction of menin-oncosuppressor function in primary fibroblasts from MEN1 patients.