scholarly article | Q13442814 |
P356 | DOI | 10.1139/CJPP-2015-0146 |
P698 | PubMed publication ID | 26524499 |
P2093 | author name string | Yuan Li | |
Madhu B Anand-Srivastava | |||
Grace Bou Daou | |||
P2860 | cites work | Modulation of protein kinase activity and gene expression by reactive oxygen species and their role in vascular physiology and pathophysiology | Q34059645 |
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The reaction of glucagon with its receptor: evidence for discrete regions of activity and binding in the glucagon molecule | Q37474698 | ||
The cyclic AMP-mediated stimulation of cell proliferation | Q38268154 | ||
Role of epidermal growth factor receptor transactivation in endothelin-1-induced enhanced expression of Gi protein and proliferation in A10 vascular smooth muscle cells | Q39174251 | ||
Implication of multiple signaling pathways in the regulation of angiotensin II induced enhanced expression of Giα proteins in vascular smooth muscle cells | Q39316460 | ||
Small cytoplasmic domain peptides of natriuretic peptide receptor-C attenuate cell proliferation through Gialpha protein/MAP kinase/PI3-kinase/AKT pathways | Q40241686 | ||
Hypertensive sodium-proton exchanger phenotype persists in immortalized lymphoblasts from essential hypertensive patients. A cell culture model for human hypertension | Q40305844 | ||
Enhanced G protein activation in immortalized lymphoblasts from patients with essential hypertension. | Q41318399 | ||
Induction of G alpha i2-specific antisense RNA in vivo inhibits neonatal growth | Q42511887 | ||
Enhanced expression of inhibitory guanine nucleotide regulatory protein in spontaneously hypertensive rats. Relationship to adenylate cyclase inhibition | Q42796280 | ||
Lymphocytes from spontaneously hypertensive rats exhibit enhanced adenylyl cyclase-Gi protein signaling | Q43512555 | ||
Inactivation of enhanced expression of G(i) proteins by pertussis toxin attenuates the development of high blood pressure in spontaneously hypertensive rats | Q44095813 | ||
The G protein-gated atrial K+ channel is stimulated by three distinct Gi alpha-subunits | Q44104764 | ||
Altered expression of G-protein mRNA in spontaneously hypertensive rats | Q44707739 | ||
Losartan-induced attenuation of blood pressure in L-NAME hypertensive rats is associated with reversal of the enhanced expression of Gi alpha proteins | Q44858371 | ||
Epidermal growth factor receptor transactivation by endogenous vasoactive peptides contributes to hyperproliferation of vascular smooth muscle cells of SHR | Q45577207 | ||
H2O2-induced phosphorylation of ERK1/2 and PKB requires tyrosine kinase activity of insulin receptor and c-Src | Q46584373 | ||
Oxidative stress contributes to the enhanced expression of Gialpha proteins and adenylyl cyclase signaling in vascular smooth muscle cells from spontaneously hypertensive rats | Q46790688 | ||
Refinement of the Coomassie brilliant blue G assay for quantitative protein determination | Q50890041 | ||
cAMP attenuates the enhanced expression of Gi proteins and hyperproliferation of vascular smooth muscle cells from SHR: role of ROS and ROS-mediated signaling | Q51075050 | ||
Evidence for hyperplasia in mesenteric resistance vessels of spontaneously hypertensive rats using a three-dimensional disector | Q54441587 | ||
Transactivation of epidermal growth factor receptor by enhanced levels of endogenous angiotensin II contributes to the overexpression of Giα proteins in vascular smooth muscle cells from SHR | Q54578386 | ||
Enhanced levels of endogenous endothelin-1 contribute to the over expression of Giα protein in vascular smooth muscle cells from SHR: Role of growth factor receptor activation | Q54641259 | ||
P433 | issue | 1 | |
P304 | page(s) | 49-58 | |
P577 | publication date | 2015-06-18 | |
P1433 | published in | Canadian Journal of Physiology and Pharmacology | Q5030248 |
P1476 | title | Enhanced expression of Giα proteins contributes to the hyperproliferation of vascular smooth muscle cells from spontaneously hypertensive rats via MAP kinase- and PI3 kinase-independent pathways | |
P478 | volume | 94 |
Q48173752 | Contribution of oxidative stress and growth factor receptor transactivation in natriuretic peptide receptor C-mediated attenuation of hyperproliferation of vascular smooth muscle cells from SHR. |
Q53823179 | Inhibition of overexpression of Giα proteins and nitroxidative stress contribute to sodium nitroprusside-induced attenuation of high blood pressure in SHR. |
Q37556115 | Knockdown of Inhibitory Guanine Nucleotide Binding Protein Giα-2 by Antisense Oligodeoxynucleotides Attenuates the Development of Hypertension and Tachycardia in Spontaneously Hypertensive Rats |
Q50126148 | Natriuretic peptide receptor-C activation attenuates angiotensin II-induced enhanced oxidative stress and hyperproliferation of aortic vascular smooth muscle cells |
Q33888027 | Nitric oxide attenuates overexpression of Giα proteins in vascular smooth muscle cells from SHR: Role of ROS and ROS-mediated signaling |
Q47255917 | Resveratrol attenuates hyperproliferation of vascular smooth muscle cells from spontaneously hypertensive rats: Role of ROS and ROS-mediated cell signaling |
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