Haluk Lacin

researcher

Haluk Lacin is …
instance of (P31):
humanQ5

External links are
P2381Academic Tree ID69887
P496ORCID iD0000-0003-2468-9618
P1153Scopus author ID6503859417
P4012Semantic Scholar author ID6088837

P1416affiliationJanelia Research CampusQ1319362
P69educated atWashington University School of MedicineQ7972509
P108employerUniversity of Missouri–Kansas CityQ940690
Janelia Research CampusQ1319362
Howard Hughes Medical InstituteQ1512226
Washington University School of MedicineQ7972509
P106occupationresearcherQ1650915

Reverse relations

author (P50)
Q53973755A new dynamic model system for the study of capture reactions for diffusable compounds in cytochemistry. II. Effect of the composition of the incubation medium on the trapping of phosphate ions in acid phosphatase cytochemistry
Q30577256Genome-wide identification of Drosophila Hb9 targets reveals a pivotal role in directing the transcriptome within eight neuronal lineages, including activation of nitric oxide synthase and Fd59a/Fox-D.
Q39425120Lineage mapping identifies molecular and architectural similarities between the larval and adult Drosophila central nervous system
Q41963522Neuromancer1 and Neuromancer2 regulate cell fate specification in the developing embryonic CNS of Drosophila melanogaster
Q64117504Neurotransmitter identity is acquired in a lineage-restricted manner in the CNS
Q33938577Senseless acts as a binary switch during sensory organ precursor selection.
Q39786052Synaptic transmission parallels neuromodulation in a central food-intake circuit
Q48007221The extracellular metalloprotease AdamTS-A anchors neural lineages in place within and preserves the architecture of the central nervous system
Q37593634Transcription factor expression uniquely identifies most postembryonic neuronal lineages in the Drosophila thoracic central nervous system.
Q90683980Unc-4 acts to promote neuronal identity and development of the take-off circuit in the Drosophila CNS
Q42229822dbx mediates neuronal specification and differentiation through cross-repressive, lineage-specific interactions with eve and hb9.

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