| scholarly article | Q13442814 |
| P356 | DOI | 10.1152/AJPHEART.00638.2005 |
| P698 | PubMed publication ID | 16085678 |
| P50 | author | Lori Walker | Q57438001 |
| P2093 | author name string | Pieter P de Tombe | |
| Peter M Buttrick | |||
| Alexander S Geha | |||
| Malek G Massad | |||
| Ronald D McKinney | |||
| Eias E Jweied | |||
| Irwin Brodsky | |||
| P2860 | cites work | Sarcomere protein gene mutations in hypertrophic cardiomyopathy of the elderly | Q24292234 |
| Transgenic mouse model of stunned myocardium | Q28142983 | ||
| Identification of a functionally critical protein kinase C phosphorylation residue of cardiac troponin T | Q28183029 | ||
| Increased Ca2+-sensitivity of the contractile apparatus in end-stage human heart failure results from altered phosphorylation of contractile proteins | Q28198289 | ||
| Phosphorylation or glutamic acid substitution at protein kinase C sites on cardiac troponin I differentially depress myofilament tension and shortening velocity | Q28205242 | ||
| Troponin T isoform expression in humans. A comparison among normal and failing adult heart, fetal heart, and adult and fetal skeletal muscle | Q28240311 | ||
| Cardiac troponin I mutants. Phosphorylation by protein kinases C and A and regulation of Ca(2+)-stimulated MgATPase of reconstituted actomyosin S-1 | Q28291787 | ||
| Differential regulation of cardiac actomyosin S-1 MgATPase by protein kinase C isozyme-specific phosphorylation of specific sites in cardiac troponin I and its phosphorylation site mutants | Q28298293 | ||
| Proteolytic N-terminal truncation of cardiac troponin I enhances ventricular diastolic function | Q28299362 | ||
| A non-equilibrium isoelectric focusing method to determine states of phosphorylation of cardiac troponin I: identification of Ser-23 and Ser-24 as significant sites of phosphorylation by protein kinase C | Q28300371 | ||
| Integration of cardiac myofilament activity and regulation with pathways signaling hypertrophy and failure | Q34122478 | ||
| Antiadrenergic therapy of chronic heart failure: surprises and new opportunities | Q35077386 | ||
| Myocardial beta-1 adrenoceptor down-regulation in aging and heart failure: implications for beta-blocker use in older adults with heart failure | Q35607857 | ||
| New insights into diastolic heart failure: role of diabetes mellitus | Q35690722 | ||
| Molecular and integrated biology of thin filament protein phosphorylation in heart muscle | Q35807681 | ||
| Biology of the troponin complex in cardiac myocytes. | Q36057095 | ||
| Molecular targets of diabetic cardiovascular complications | Q36198737 | ||
| Protein kinase A does not alter economy of force maintenance in skinned rat cardiac trabeculae. | Q36701635 | ||
| Myofibrillar calcium sensitivity of isometric tension is increased in human dilated cardiomyopathies: role of altered beta-adrenergically mediated protein phosphorylation | Q37357150 | ||
| Troponin I serines 43/45 and regulation of cardiac myofilament function | Q42524860 | ||
| Angiotensin II promotes glucose-induced activation of cardiac protein kinase C isozymes and phosphorylation of troponin I. | Q43687870 | ||
| Cardiac troponin T variants produced by aberrant splicing of multiple exons in animals with high instances of dilated cardiomyopathy | Q44177990 | ||
| Troponin I in the murine myocardium: influence on length-dependent activation and interfilament spacing | Q44300159 | ||
| Stunned peri-infarct canine myocardium is characterized by degradation of troponin T, not troponin I. | Q44974874 | ||
| Cardiac troponin T isoforms affect the Ca(2+) sensitivity of force development in the presence of slow skeletal troponin I: insights into the role of troponin T isoforms in the fetal heart. | Q45052899 | ||
| Molecular basis of human cardiac troponin T isoforms expressed in the developing, adult, and failing heart. | Q55063334 | ||
| Echocardiographic evidence for the existence of a distinct diabetic cardiomyopathy (The Framingham Heart Study) | Q57604699 | ||
| P433 | issue | 6 | |
| P304 | page(s) | H2478-83 | |
| P577 | publication date | 2005-08-05 | |
| P1433 | published in | American Journal of Physiology Heart and Circulatory Physiology | Q3193662 |
| P1476 | title | Depressed cardiac myofilament function in human diabetes mellitus | |
| P478 | volume | 289 |
| Q34815218 | ACE-inhibition, but not weight reduction restores cardiomyocyte response to β-adrenergic stimulation in the metabolic syndrome. |
| Q47221649 | Altered myofilament structure and function in dogs with Duchenne muscular dystrophy cardiomyopathy |
| Q35315109 | Biochemical and myofilament responses of the right ventricle to severe pulmonary hypertension |
| Q37378507 | Current techniques for the study of troponin I phosphorylation in human heart |
| Q51919754 | Development of contractile dysfunction in rat heart failure: hierarchy of cellular events. |
| Q58599424 | Diabetes with heart failure increases methylglyoxal modifications in the sarcomere, which inhibit function |
| Q26799318 | Diabetic Cardiomyopathy; Summary of 41 Years |
| Q57186141 | Diabetic cardiomyopathy |
| Q38271210 | Diabetic cardiomyopathy and its mechanisms: Role of oxidative stress and damage |
| Q30457504 | Diabetic cardiomyopathy: pathophysiology and clinical features |
| Q28395852 | Diabetic cardiomyopathy: signaling defects and therapeutic approaches |
| Q30362092 | Diagnostic approaches for diabetic cardiomyopathy. |
| Q37663364 | Effect of isoflurane on myocardial energetic and oxidative stress in cardiac muscle from Zucker diabetic fatty rat. |
| Q57455677 | Frequency-dependent myofilament Ca2+ desensitization in failing rat myocardium |
| Q38596700 | Glucose Homeostasis and Cardiovascular Alterations in Diabetes |
| Q49384661 | Hyperglycemia-induced cardiac contractile dysfunction in the diabetic heart. |
| Q37719325 | Impaired relaxation despite upregulated calcium-handling protein atrial myocardium from type 2 diabetic patients with preserved ejection fraction. |
| Q36125213 | Incomplete recovery of myocyte contractile function despite improvement of myocardial architecture with left ventricular assist device support |
| Q51502401 | Increased myocardial stiffness with maintenance of length-dependent calcium activation by female sex hormones in diabetic rats. |
| Q37278004 | Interplay between impaired calcium regulation and insulin signaling abnormalities in diabetic cardiomyopathy |
| Q35636210 | Interventricular differences in myofilament function in experimental congestive heart failure |
| Q57455682 | Left ventricular myofilament dysfunction in rat experimental hypertrophy and congestive heart failure |
| Q37583506 | Mammalian target of rapamycin (mTOR) inhibition with rapamycin improves cardiac function in type 2 diabetic mice: potential role of attenuated oxidative stress and altered contractile protein expression. |
| Q38196172 | Nitric oxide synthase regulation of cardiac excitation-contraction coupling in health and disease |
| Q36762969 | Nonischemic heart failure in diabetes mellitus |
| Q37040657 | O-linked GlcNAc modification of cardiac myofilament proteins: a novel regulator of myocardial contractile function |
| Q59800367 | Of mice and men: models and mechanisms of diabetic cardiomyopathy |
| Q39195672 | Pathophysiological Fundamentals of Diabetic Cardiomyopathy |
| Q36104643 | Removal of Abnormal Myofilament O-GlcNAcylation Restores Ca2+ Sensitivity in Diabetic Cardiac Muscle. |
| Q37405997 | Synchronous in situ ATPase activity, mechanics, and Ca2+ sensitivity of human and porcine myocardium |
| Q38815425 | The Type 2 Diabetic Heart: Its Role in Exercise Intolerance and the Challenge to Find Effective Exercise Interventions |
| Q37701737 | The diabetic cardiomyopathy |
| Q37239549 | The role of Akt/GSK-3beta signaling in familial hypertrophic cardiomyopathy |
| Q35968502 | The use of human cardiac tissue in biophysical research: the risks of translation |
| Q57455665 | Tissue procurement strategies affect the protein biochemistry of human heart samples |
| Q37029676 | Troponin phosphorylation and myofilament Ca2+-sensitivity in heart failure: increased or decreased? |
Search more.