Serum amyloid A (SAA), inflammation, cholesterol and mevalonate kinase deficiency – reply

Serum amyloid A (SAA), inflammation, cholesterol and mevalonate kinase deficiency – reply is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.3109/13506129.2012.702703

P2093author name stringRobert Kisilevsky
P2860cites workAcute-phase serum amyloid A: perspectives on its physiological and pathological rolesQ28259372
Impact of serum amyloid A on high density lipoprotein composition and levelsQ34187954
ATP binding cassette G1-dependent cholesterol efflux during inflammationQ34503409
The farnesyltransferase inhibitors tipifarnib and lonafarnib inhibit cytokines secretion in a cellular model of mevalonate kinase deficiency.Q39571798
Targeting farnesyl-transferase as a novel therapeutic strategy for mevalonate kinase deficiency: in vitro and in vivo approaches.Q43141414
Decreased cholesterol levels reflect a consumption of anti-inflammatory isoprenoids associated with an impaired control of inflammation in a mouse model of mevalonate kinase deficiencyQ43154533
Natural isoprenoids are able to reduce inflammation in a mouse model of mevalonate kinase deficiencyQ46662065
The path of murine serum amyloid a through peritoneal macrophagesQ58257518
P433issue3
P921main subjectinflammationQ101991
P304page(s)165-166
P577publication date2012-06-28
P1433published inAmyloidQ4749545
P1476titleSerum amyloid A (SAA), inflammation, cholesterol and mevalonate kinase deficiency – reply
P478volume19

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