| scholarly article | Q13442814 |
| P6179 | Dimensions Publication ID | 1109798825 |
| P356 | DOI | 10.1186/S12989-018-0280-2 |
| P2888 | exact match | https://scigraph.springernature.com/pub.10.1186/s12989-018-0280-2 |
| P2880 | NIOSHTIC-2 ID | 20053798 |
| P932 | PMC publication ID | 6230229 |
| P698 | PubMed publication ID | 30413212 |
| P50 | author | W. Travis Goldsmith | Q81798231 |
| Sherri A. Friend | Q106263443 | ||
| P2093 | author name string | Alaeddin Bashir Abukabda | |
| Carroll Rolland McBride | |||
| Elizabeth Compton Bowdridge | |||
| Krista Lee Garner | |||
| Thomas Paul Batchelor | |||
| Timothy Robert Nurkiewicz | |||
| P2860 | cites work | Nanoparticle inhalation impairs endothelium-dependent vasodilation in subepicardial arterioles | Q23909457 |
| Impairment of coronary arteriolar endothelium-dependent dilation after multi-walled carbon nanotube inhalation: a time-course study | Q23919580 | ||
| Pulmonary nanoparticle exposure disrupts systemic microvascular nitric oxide signaling | Q23922830 | ||
| Nanoparticle inhalation augments particle-dependent systemic microvascular dysfunction | Q23922832 | ||
| IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines | Q24294720 | ||
| Lymphoid tissue inducer-like cells are an innate source of IL-17 and IL-22 | Q24648620 | ||
| The origin and role of innate lymphoid cells in the lung | Q26740079 | ||
| Inflammasome, IL-1 and inflammation in ozone-induced lung injury | Q26750611 | ||
| Group 2 innate lymphoid cells in the lung | Q27021302 | ||
| Group 2 innate lymphoid cells and asthma | Q28080605 | ||
| Endogenous IL-33 is highly expressed in mouse epithelial barrier tissues, lymphoid organs, brain, embryos, and inflamed tissues: in situ analysis using a novel Il-33-LacZ gene trap reporter strain | Q28260796 | ||
| Maternal engineered nanomaterial exposure and fetal microvascular function: does the Barker hypothesis apply? | Q28387965 | ||
| A carbon nanotube toxicity paradigm driven by mast cells and the IL-₃₃/ST₂ axis | Q28388782 | ||
| Multi-walled carbon nanotube instillation impairs pulmonary function in C57BL/6 mice | Q28391783 | ||
| Uterine microvascular sensitivity to nanomaterial inhalation: An in vivo assessment | Q28392163 | ||
| Atomic layer deposition coating of carbon nanotubes with aluminum oxide alters pro-fibrogenic cytokine expression by human mononuclear phagocytes in vitro and reduces lung fibrosis in mice in vivo | Q28394247 | ||
| Innate Immune Responses to Nanoparticle Exposure in the Lung | Q28394494 | ||
| Equivalent titanium dioxide nanoparticle deposition by intratracheal instillation and whole body inhalation: the effect of dose rate on acute respiratory tract inflammation | Q28395053 | ||
| Impacts of prenatal nanomaterial exposure on male adult Sprague-Dawley rat behavior and cognition | Q28396408 | ||
| Intravenously delivered graphene nanosheets and multiwalled carbon nanotubes induce site-specific Th2 inflammatory responses via the IL-33/ST2 axis | Q28397819 | ||
| Innate lymphoid cells--a proposal for uniform nomenclature | Q29617431 | ||
| Heterogeneous Vascular Bed Responses to Pulmonary Titanium Dioxide Nanoparticle Exposure. | Q33720365 | ||
| IL-33-responsive innate lymphoid cells are an important source of IL-13 in chronic rhinosinusitis with nasal polyps | Q33738493 | ||
| Airway exposure to multi-walled carbon nanotubes disrupts the female reproductive cycle without affecting pregnancy outcomes in mice. | Q33742492 | ||
| IL-33 mediates multi-walled carbon nanotube (MWCNT)-induced airway hyper-reactivity via the mobilization of innate helper cells in the lung. | Q33841609 | ||
| Innate signals compensate for the absence of PKC-{theta} during in vivo CD8(+) T cell effector and memory responses | Q34063599 | ||
| The expanding family of innate lymphoid cells: regulators and effectors of immunity and tissue remodeling. | Q34152180 | ||
| The nanomaterial-dependent modulation of dendritic cells and its potential influence on therapeutic immunosuppression in lupus | Q34180779 | ||
| Induction of an inflammatory loop by interleukin-1β and tumor necrosis factor-α involves NF-kB and STAT-1 in differentiated human neuroprogenitor cells | Q34903095 | ||
| Contribution of IL-33-activated type II innate lymphoid cells to pulmonary eosinophilia in intestinal nematode-infected mice | Q35807590 | ||
| Innate lymphoid cells promote lung-tissue homeostasis after infection with influenza virus | Q35872309 | ||
| Group 2 innate lymphoid cells mediate ozone-induced airway inflammation and hyperresponsiveness in mice | Q36563818 | ||
| IL-9-mediated survival of type 2 innate lymphoid cells promotes damage control in helminth-induced lung inflammation | Q37397810 | ||
| IL-25 and type 2 innate lymphoid cells induce pulmonary fibrosis. | Q37474764 | ||
| Negative regulation of NF-κB p65 activity by serine 536 phosphorylation | Q37668699 | ||
| Innate type 2 cells and asthma | Q38022971 | ||
| Innate immune cells in asthma | Q38137544 | ||
| New kids on the block: group 2 innate lymphoid cells and type 2 inflammation in the lung | Q38159924 | ||
| Innate lymphoid cells in asthma: when innate immunity comes in a Th2 flavor | Q38169043 | ||
| Isolation and characterization of mouse innate lymphoid cells. | Q38235846 | ||
| Type-2 innate lymphoid cells in asthma and allergy | Q38294532 | ||
| The biology of innate lymphoid cells | Q38319907 | ||
| Innate lymphoid cells: the role in respiratory infections and lung tissue damage | Q38622555 | ||
| IL-33 Receptor (ST2) Signalling is Important for Regulation of Th2-Mediated Airway Inflammation in a Murine Model of Acute Respiratory Syncytial Virus Infection | Q38905619 | ||
| The second touch hypothesis: T cell activation, homing and polarization | Q40055228 | ||
| Isolation of Group 2 Innate Lymphoid Cells from Mouse Lungs | Q40082836 | ||
| IL33-mediated ILC2 activation and neutrophil IL5 production in the lung response after severe trauma: A reverse translation study from a human cohort to a mouse trauma model. | Q40120480 | ||
| Alpha-macroglobulin secreted by alveolar macrophages serves as a binding protein for a macrophage-derived homologue of platelet-derived growth factor. | Q41258565 | ||
| SnapShot: innate lymphoid cells | Q44846377 | ||
| Reactive oxygen species damage drives cardiac and mitochondrial dysfunction following acute nano-titanium dioxide inhalation exposure | Q46240480 | ||
| IL-33 promotes the egress of group 2 innate lymphoid cells from the bone marrow. | Q47340483 | ||
| Nanoparticle effect on neutrophil produced myeloperoxidase. | Q49330389 | ||
| Maternal engineered nanomaterial inhalation during gestation alters the fetal transcriptome. | Q49421963 | ||
| Distribution of lung cell numbers and volumes between alveolar and nonalveolar tissue. | Q50791172 | ||
| Prenatal exposure to carbon black (printex 90): effects on sexual development and neurofunction. | Q51446734 | ||
| Estrous cycle-dependent modulation of in vivo microvascular dysfunction after nanomaterial inhalation. | Q55436946 | ||
| Anti-IL-33 antibody treatment inhibits airway inflammation in a murine model of allergic asthma | Q84030123 | ||
| Human IL-25- and IL-33-responsive type 2 innate lymphoid cells are defined by expression of CRTH2 and CD161 | Q84933191 | ||
| The interleukin-33 receptor ST2 is important for the development of peripheral airway hyperresponsiveness and inflammation in a house dust mite mouse model of asthma | Q86813000 | ||
| P6195 | funding scheme | extramural research | Q124503498 |
| P433 | issue | 1 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | titanium | Q716 |
| lung | Q7886 | ||
| nanotechnology | Q11468 | ||
| nanoparticle | Q61231 | ||
| toxin | Q184651 | ||
| titanium dioxide | Q193521 | ||
| toxicity | Q274160 | ||
| cardiovascular disease | Q389735 | ||
| reproductive health | Q1981388 | ||
| health effect | Q2574087 | ||
| respiratory disease | Q3286546 | ||
| lung disease | Q3392853 | ||
| phlebotomy | Q3595842 | ||
| P5008 | on focus list of Wikimedia project | Wikimedia–NIOSH collaboration | Q104416361 |
| P304 | page(s) | 43 | |
| P577 | publication date | 2018-11-09 | |
| P1433 | published in | Particle and Fibre Toxicology | Q15724404 |
| P859 | sponsor | NIOSH Health Effects Laboratory Division | Q123344455 |
| P1476 | title | Group II innate lymphoid cells and microvascular dysfunction from pulmonary titanium dioxide nanoparticle exposure | |
| Group II innate lymphoid cells and microvascular dysfunction from pulmonary titanium dioxide nanoparticle exposure. | |||
| P478 | volume | 15 |
| Q77792113 | Maternal engineered nanomaterial inhalation during gestation disrupts vascular kisspeptin reactivity |
| Q64097954 | Particle toxicology and health - where are we? |
| Q64882984 | ROS promote epigenetic remodeling and cardiac dysfunction in offspring following maternal engineered nanomaterial (ENM) exposure. |
| Q97546356 | Recent insights on indirect mechanisms in developmental toxicity of nanomaterials |
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