Widening of capillary neck and alteration of extracellular matrix ultrastructure in diabetic rat glomerulus as revealed by computer morphometry and improved tissue processing

scientific article published on 01 January 1993

Widening of capillary neck and alteration of extracellular matrix ultrastructure in diabetic rat glomerulus as revealed by computer morphometry and improved tissue processing is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1007/BF01606586
P698PubMed publication ID8212540

P2093author name stringT Sakai
M Fukui
Y Tomino
H Koide
I Shirato
P2860cites workStructural-functional relationships in diabetic nephropathyQ34616152
Differential expression of basement membrane collagen chains in diabetic nephropathyQ35830767
Immunohistochemical localization of extracellular matrix components in human diabetic glomerular lesionsQ35831145
Predominance of hemodynamic rather than metabolic factors in the pathogenesis of diabetic glomerulopathyQ37531744
The glomerular mesangiumQ40284542
The kidney in diabetesQ40302193
Modulation of basement membrane component gene expression in glomeruli of aminonucleoside nephrosisQ44361019
Glomerular structure in type-1 (insulin-dependent) diabetic patients with normo- and microalbuminuriaQ45062498
Effects of tissue preparation on glomerular volume and capillary structure in the rat.Q45999355
Glomerular abnormalities in long-term experimental diabetes. Role of hemodynamic and nonhemodynamic factors and effects of antihypertensive therapyQ54274561
Adaptation of the rat kidney to altered water intake and urine concentration.Q54388605
Glomerular Structure and Function in Diabetic Nephropathy: Early to Advanced StagesQ60218025
A strong correlation between glomerular filtration rate and filtration surface in diabetic nephropathyQ60218055
Changes in glomerular structure following acute mesangial failure in the isolated perfused kidneyQ68107687
Continuous stretch-relaxation in culture alters rat mesangial cell morphology, growth characteristics, and metabolic activityQ68107696
Mesangial cell-glomerular basement membrane connections counteract glomerular capillary and mesangium expansionQ68628592
Role of mesangial cell contraction in adaptation of the glomerular tuft to changes in extracellular volumeQ68754394
The structural relationship between mesangial cells and basement membrane of the renal glomerulusQ69261878
Impaired contractile responsiveness of diabetic glomeruli to angiotensin II: a possible indication of mesangial dysfunction in diabetes mellitusQ69539064
Amelioration of mesangial volume and surface alterations following islet transplantation in diabetic ratsQ71504168
Variability of intercellular spaces between macula densa cells: a transmission electron microscopic study in rabbits and ratsQ72052403
Development of glomerular lesions in experimental long-term diabetes in the ratQ72112884
Specific determination of blood glucose with o-toluidineQ76490966
Effects of sorbinil on glomerular structure and function in long-term-diabetic ratsQ93512129
P433issue2
P304page(s)121-129
P577publication date1993-01-01
P1433published inVirchows Archiv. A, Pathological Anatomy and HistopathologyQ27713801
P1476titleWidening of capillary neck and alteration of extracellular matrix ultrastructure in diabetic rat glomerulus as revealed by computer morphometry and improved tissue processing
P478volume423

Reverse relations

cites work (P2860)
Q42049689Cytoskeletal changes in podocytes associated with foot process effacement in Masugi nephritis.
Q74480189Effects of the antihypertensive drug efonidipine hydrochloride on albuminuria and renal histopathology in young spontaneously hypertensive rats with diabetes
Q28376092Effects of the antihypertensive drug nifedipine on albuminuria and renal histopathology in young spontaneously hypertensive rats with diabetes
Q72785136Podocyte damage is a critical step in the development of glomerulosclerosis in the uninephrectomised-desoxycorticosterone hypertensive rat
Q71914751The development of focal segmental glomerulosclerosis in masugi nephritis is based on progressive podocyte damage

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