Nitric oxide synthase I (NOS-I) is deficient in the sarcolemma of striated muscle fibers in patients with Duchenne muscular dystrophy, suggesting an association with dystrophin

scientific article published on 01 January 1996

Nitric oxide synthase I (NOS-I) is deficient in the sarcolemma of striated muscle fibers in patients with Duchenne muscular dystrophy, suggesting an association with dystrophin is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1016/S0065-1281(96)80051-1
P698PubMed publication ID9054190

P2093author name stringGossrau R
Gosztonyi G
Grozdanovic Z
P2860cites workDuchenne muscular dystrophy: deficiency of dystrophin-associated proteins in the sarcolemmaQ70651010
Demonstration of nitric oxide synthase (NOS) in marmosets by NADPH diaphorase (NADPH-d) histochemistry and NOS immunoreactivityQ70797515
When NADPH diaphorase (NADPHd) works in the presence of formaldehyde, the enzyme appears to visualize selectively cells with constitutive nitric oxide synthase (NOS)Q71673738
Endothelial type nitric oxide synthase in skeletal muscle fibers: mitochondrial relationshipsQ71698647
The Duchenne muscular dystrophy gene product is localized in sarcolemma of human skeletal muscleQ24297845
The complete sequence of dystrophin predicts a rod-shaped cytoskeletal proteinQ28118471
Glycoprotein complex anchoring dystrophin to sarcolemmaQ28292439
Primary structure of dystrophin-associated glycoproteins linking dystrophin to the extracellular matrixQ28296676
Nitric oxide in skeletal muscleQ28583418
Membrane organization of the dystrophin-glycoprotein complexQ29615149
Dystrophin: the protein product of the Duchenne muscular dystrophy locusQ29618077
Dystrophin-glycoprotein complex: its role in the molecular pathogenesis of muscular dystrophies.Q40792342
Deficiency of dystrophin-associated proteins: a common mechanism leading to muscle cell necrosis in severe childhood muscular dystrophiesQ40838945
Immunostaining of skeletal and cardiac muscle surface membrane with antibody against Duchenne muscular dystrophy peptideQ41429227
Cloned human brain nitric oxide synthase is highly expressed in skeletal muscleQ41578075
Duchenne muscular dystrophy: deficiency of dystrophin at the muscle cell surfaceQ44350613
Nitric oxide release is present from incubated skeletal muscle preparationsQ46492515
Demonstration of a unique population of neurons with NADPH-diaphorase histochemistryQ48720909
Association of dystrophin and an integral membrane glycoprotein.Q52485843
Deficiency of a glycoprotein component of the dystrophin complex in dystrophic muscleQ59089242
Immunoelectron microscopic localization of dystrophin in myofibresQ59096146
Ultrastructural localization of dystrophin in human muscle by using gold immunolabellingQ68091120
Characterization of dystrophin in muscle-biopsy specimens from patients with Duchenne's or Becker's muscular dystrophyQ68137662
P433issue1
P921main subjectDuchenne muscular dystrophyQ1648484
P304page(s)61-69
P577publication date1996-01-01
P1433published inActa HistochemicaQ2690684
P1476titleNitric oxide synthase I (NOS-I) is deficient in the sarcolemma of striated muscle fibers in patients with Duchenne muscular dystrophy, suggesting an association with dystrophin
P478volume98

Reverse relations

cites work (P2860)
Q38482999A role for nitric oxide in muscle repair: nitric oxide-mediated activation of muscle satellite cells
Q73330668Absence of nitric oxide synthase I despite the presence of the dystrophin complex in human striated muscle
Q71668561Adhalin (alpha-sarcoglycan) is not required for anchoring of nitric oxide synthase I (NOS I) to the sarcolemma in non-mammalian skeletal (striated) muscle fibers
Q53475327Administration of chinese herbal medicines facilitates the locomotor activity in dystrophin-deficient mice.
Q74444837Caveolin-3 and nitric oxide synthase I in healthy and diseased skeletal muscle
Q33644573Characteristics of skeletal muscle in mdx mutant mice
Q73338676Differences in the localization of the postsynaptic nitric oxide synthase I and acetylcholinesterase suggest a heterogeneity of neuromuscular junctions in rat and mouse skeletal muscles
Q31133021Early onset of lipofuscin accumulation in dystrophin-deficient skeletal muscles of DMD patients and mdx mice
Q37088443Epigenetic drugs in the treatment of skeletal muscle atrophy.
Q24538577Gene expression comparison of biopsies from Duchenne muscular dystrophy (DMD) and normal skeletal muscle
Q71875157Histochemistry of therapeutically relevant enzymes in human tumours transplanted into severe combined immunodeficient (SCID) mice: nitric oxide synthase-associated diaphorase, beta-D-glucuronidase and non-specific alkaline phosphatase
Q33950422Identification of a neuronal nitric oxide synthase in isolated cardiac mitochondria using electrochemical detection
Q77715320Inhibition of skeletal muscle sarcoplasmic reticulum Ca2+-ATPase by nitric oxide
Q42514805Muscular nitric oxide synthase (muNOS) and utrophin
Q34465013NO message from muscle
Q36560597New insights in the regulation of calcium transfers by muscle dystrophin-based cytoskeleton: implications in DMD.
Q73906319Nitric oxide synthase (NOS) I during postnatal development in rat and mouse skeletal muscle
Q82394991Nitric oxide synthase activity in muscle foods
Q36402599Sarcolemmal targeting of nNOSμ improves contractile function of mdx muscle
Q43122388Skeletal muscle contraction-induced vasodilation in the microcirculation
Q36820423Skeletal muscle function during exercise-fine-tuning of diverse subsystems by nitric oxide
Q46303843Sparing of mdx extraocular muscles from dystrophic pathology is not attributable to normalized concentration or distribution of neuronal nitric oxide synthase
Q73042306The NO way to increase muscular utrophin expression?
Q78475375The expression of neuronal nitric oxide synthase and dystrophin in rat regenerating muscles
Q35834674The muscular dystrophies: from genes to therapies
Q44455459The nitric oxide synthase-1 and nitric oxide synthase-3/nitric oxide signalling systems in the heart of wild type mice and mouse mutants
Q42411441Two tales concerning skeletal muscle
Q64073142nNOS/GSNOR interaction contributes to skeletal muscle differentiation and homeostasis

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