Glucose intolerance in familial combined hyperlipidaemia. EUFAM study group

scientific article published on 01 January 1998

Glucose intolerance in familial combined hyperlipidaemia. EUFAM study group is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1046/J.1365-2362.1998.00243.X
P698PubMed publication ID9502184

P50authorMarja-Riitta TaskinenQ18246437
P2093author name stringC Ehnholm
J S Viikari
K Ylitalo
K V Porkka
I Nuotio
L Suurinkeroinen
P Pajukanta
J Vakkilainen
P433issue1
P921main subjectglucose intoleranceQ2661464
P304page(s)24-32
P577publication date1998-01-01
P1433published inEuropean Journal of Clinical InvestigationQ15745208
P1476titleGlucose intolerance in familial combined hyperlipidaemia. EUFAM study group
P478volume28

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cites work (P2860)
Q34389958A genome scan for familial combined hyperlipidemia reveals evidence of linkage with a locus on chromosome 11.
Q44590583Confirmed Locus on Chromosome 11p and Candidate Loci on 6q and 8p for the Triglyceride and Cholesterol Traits of Combined Hyperlipidemia
Q44240874Effects of nateglinide and glibenclamide on postprandial lipid and glucose metabolism in type 2 diabetes.
Q44098360Genetic influences contributing to LDL particle size in familial combined hyperlipidaemia
Q34107190Genetics of familial combined hyperlipidemia
Q34389639Genomewide scan for familial combined hyperlipidemia genes in finnish families, suggesting multiple susceptibility loci influencing triglyceride, cholesterol, and apolipoprotein B levels
Q43631829Insulin resistance in the St. Thomas' mixed hyperlipidaemic (SMHL) rabbit, a model for familial combined hyperlipidaemia
Q43813854Lipoprotein and apolipoprotein abnormalities in familial combined hyperlipidemia: a 20-year prospective study
Q53959743Metabolic pathogenesis of familial combined hyperlipidaemia with emphasis on insulin resistance, adipose tissue metabolism and free fatty acids.
Q28363810Monogenic dyslipidemias: window on determinants of plasma lipoprotein metabolism
Q73409229Reduced hormone-sensitive lipase activity is not a major metabolic defect in Finnish FCHL families
Q45975588TCF7L2 is associated with high serum triacylglycerol and differentially expressed in adipose tissue in families with familial combined hyperlipidaemia.

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