Lysis of plasma clots by urokinase-soluble urokinase receptor complexes

scientific article published on 01 September 1998

Lysis of plasma clots by urokinase-soluble urokinase receptor complexes is …
instance of (P31):
scholarly articleQ13442814

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P698PubMed publication ID9731065

P2093author name stringCines DB
Kuo A
Higazi AA
Hiss E
Bdeir K
Arad S
Barghouti I
P433issue6
P304page(s)2075-2083
P577publication date1998-09-01
P1433published inBloodQ885070
P1476titleLysis of plasma clots by urokinase-soluble urokinase receptor complexes
P478volume92

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cites work (P2860)
Q35580943Blood-brain barrier permeability and tPA-mediated neurotoxicity
Q33420330Endogenous plasminogen activators mediate progressive intracerebral hemorrhage after traumatic brain injury in mice
Q36158746Increased soluble urokinase plasminogen activator receptor (suPAR) is associated with thrombosis and inhibition of plasmin generation in paroxysmal nocturnal hemoglobinuria (PNH) patients
Q37138982Inhibition of integrin alphavbeta3 prevents urokinase plasminogen activator-mediated impairment of cerebrovasodilation after cerebral hypoxia/ischemia
Q28088764Mesothelial cells in tissue repair and fibrosis
Q42028534Soluble urokinase receptor conjugated to carrier red blood cells binds latent pro-urokinase and alters its functional profile
Q34167946Urokinase plasminogen activator impairs SNP and PGE2 cerebrovasodilation after brain injury through activation of LRP and ERK MAPK.
Q34570654tPA contributes to impairment of ATP and Ca sensitive K channel mediated cerebrovasodilation after hypoxia/ischemia through upregulation of ERK MAPK.
Q36948533uPA impairs cerebrovasodilation after hypoxia/ischemia through LRP and ERK MAPK
Q36512703uPA modulates the age-dependent effect of brain injury on cerebral hemodynamics through LRP and ERK MAPK.

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