Edaravone for the treatment of acute cerebral infarction: role of endothelium-derived nitric oxide and oxidative stress

scientific article published on 01 February 2009

Edaravone for the treatment of acute cerebral infarction: role of endothelium-derived nitric oxide and oxidative stress is …
instance of (P31):
scholarly articleQ13442814
review articleQ7318358

External links are
P356DOI10.1517/14656560802636888
P698PubMed publication ID19236202

P50authorYukihito HigashiQ90166979
P433issue2
P921main subjectedaravoneQ335099
cerebral infarctionQ1209150
P304page(s)323-331
P577publication date2009-02-01
P1433published inExpert Opinion on PharmacotherapyQ5421212
P1476titleEdaravone for the treatment of acute cerebral infarction: role of endothelium-derived nitric oxide and oxidative stress
P478volume10

Reverse relations

cites work (P2860)
Q418922752-decenoic acid ethyl ester, a compound that elicits neurotrophin-like intracellular signals, facilitating functional recovery from cerebral infarction in mice
Q33938450A critical assessment of edaravone acute ischemic stroke efficacy trials: is edaravone an effective neuroprotective therapy?
Q46724168Alda-1 reduces cerebral ischemia/reperfusion injury in rat through clearance of reactive aldehydes
Q34256876Changes in cerebrospinal fluid biomarkers in human herpesvirus-6-associated acute encephalopathy/febrile seizures.
Q50741473Effects of microRNA-21 on Nerve Cell Regeneration and Neural Function Recovery in Diabetes Mellitus Combined with Cerebral Infarction Rats by Targeting PDCD4.
Q26852796Oxidative Stress and the Use of Antioxidants in Stroke
Q30968894Prophylactic Edaravone Prevents Transient Hypoxic-Ischemic Brain Injury: Implications for Perioperative Neuroprotection
Q36049201Reactive oxygen and nitrogen species in pathogenesis of vascular complications of diabetes
Q27335193Restoration of dioxin-induced damage to fetal steroidogenesis and gonadotropin formation by maternal co-treatment with α-lipoic acid
Q38629284The Dietary Components Carnosic Acid and Carnosol as Neuroprotective Agents: a Mechanistic View

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