Perinatal BPA exposure and reproductive axis function in CD-1 mice

scientific article published on 9 May 2018

Perinatal BPA exposure and reproductive axis function in CD-1 mice is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1016/J.REPROTOX.2018.05.002
P932PMC publication ID6169306
P698PubMed publication ID29752986

P2093author name stringAna M Soto
Beverly S Rubin
Cheryl M Schaeberle
Nicole Acevedo
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Modification of the oestrous cycle of the mouse by external stimuli associated with the male; changes in the oestrous cycle determined by vaginal smearsQ78384570
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Investigation of the effects of subchronic low dose oral exposure to bisphenol A (BPA) and ethinyl estradiol (EE) on estrogen receptor expression in the juvenile and adult female rat hypothalamusQ28392411
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Neonatal exposure to bisphenol a and reproductive and endocrine alterations resembling the polycystic ovarian syndrome in adult ratsQ34022159
Developmental programming: impact of fetal exposure to endocrine-disrupting chemicals on gonadotropin-releasing hormone and estrogen receptor mRNA in sheep hypothalamusQ34040092
Biomonitoring studies should be used by regulatory agencies to assess human exposure levels and safety of bisphenol A.Q34060021
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Perinatal exposure to environmentally relevant levels of bisphenol A decreases fertility and fecundity in CD-1 miceQ34834773
Effects of early prepubertal exposure to bisphenol A on the onset of puberty, ovarian weights, and estrous cycle in female miceQ35767778
The anti mullerian hormone- a novel marker for assessing the ovarian reserve in women with regular menstrual cyclesQ36554078
BPA Directly Decreases GnRH Neuronal Activity via Noncanonical PathwayQ36911568
In utero bisphenol A exposure disrupts germ cell nest breakdown and reduces fertility with age in the mouseQ37705004
Evidence of altered brain sexual differentiation in mice exposed perinatally to low, environmentally relevant levels of bisphenol A.Q42685050
Delayed Neuroendocrine Sexual Maturation in Female Rats After a Very Low Dose of Bisphenol A Through Altered GABAergic Neurotransmission and Opposing Effects of a High DoseQ42699154
Prepubertal bisphenol A exposure interferes with ovarian follicle development and its relevant gene expressionQ42709351
Fetal exposure to bisphenol A affects the primordial follicle formation by inhibiting the meiotic progression of oocytesQ42722749
Neonatal exposure to bisphenol A reduces the pool of primordial follicles in the rat ovaryQ42943686
Transfer of bisphenol A across the human placenta.Q43115691
P304page(s)39-46
P577publication date2018-05-09
P1433published inReproductive ToxicologyQ15633823
P1476titlePerinatal BPA exposure and reproductive axis function in CD-1 mice
P478volume79

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Q61798660The Case for BPA as an Obesogen: Contributors to the Controversycites workP2860

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