scholarly article | Q13442814 |
P2093 | author name string | Shigeki Miyamoto | |
Joan Heller Brown | |||
Hal M Hoffman | |||
Andrew Willeford | |||
Audrey Nickle | |||
Takeshi Suetomi | |||
P2860 | cites work | CaMKII is a RIP3 substrate mediating ischemia- and oxidative stress-induced myocardial necroptosis. | Q46615618 |
NADPH oxidase 2 mediates angiotensin II-dependent cellular arrhythmias via PKA and CaMKII. | Q46859563 | ||
The NLRP3 inflammasome is up-regulated in cardiac fibroblasts and mediates myocardial ischaemia-reperfusion injury. | Q51072106 | ||
S100a8/a9 released by CD11b+Gr1+ neutrophils activates cardiac fibroblasts to initiate angiotensin II-Induced cardiac inflammation and injury. | Q54214133 | ||
CD4 + T Cells Promote the Transition From Hypertrophy to Heart Failure During Chronic Pressure Overload | Q58003417 | ||
Isolation and culture of adult mouse cardiac myocytes | Q64377366 | ||
Angiotensin II stimulated expression of transforming growth factor-beta1 in cardiac fibroblasts and myofibroblasts | Q73542345 | ||
Calmodulin kinase II inhibition protects against myocardial cell apoptosis in vivo | Q79967267 | ||
Inflammasome activation of cardiac fibroblasts is essential for myocardial ischemia/reperfusion injury | Q83333586 | ||
Inflammasomes: mechanism of action, role in disease, and therapeutics | Q27001186 | ||
Cardiokines: recent progress in elucidating the cardiac secretome | Q27022911 | ||
Left Ventricular T-Cell Recruitment Contributes to the Pathogenesis of Heart Failure. | Q27340146 | ||
A role for mitochondria in NLRP3 inflammasome activation | Q28131794 | ||
CaMKII determines mitochondrial stress responses in heart | Q28591986 | ||
Bacterial RNA and small antiviral compounds activate caspase-1 through cryopyrin/Nalp3 | Q29615598 | ||
Activation and regulation of the inflammasomes | Q29615664 | ||
A dynamic pathway for calcium-independent activation of CaMKII by methionine oxidation | Q29620430 | ||
Nrf2 deficiency exaggerates doxorubicin-induced cardiotoxicity and cardiac dysfunction. | Q33660939 | ||
Macrophage-stimulated cardiac fibroblast production of IL-6 is essential for TGF β/Smad activation and cardiac fibrosis induced by angiotensin II | Q34264496 | ||
Oxidized Ca(2+)/calmodulin-dependent protein kinase II triggers atrial fibrillation. | Q34370565 | ||
The cardiokine story unfolds: ischemic stress-induced protein secretion in the heart | Q34809985 | ||
Linkage of beta1-adrenergic stimulation to apoptotic heart cell death through protein kinase A-independent activation of Ca2+/calmodulin kinase II. | Q34833218 | ||
CaM Kinase II mediates maladaptive post-infarct remodeling and pro-inflammatory chemoattractant signaling but not acute myocardial ischemia/reperfusion injury | Q34880308 | ||
Mitochondrial reprogramming induced by CaMKIIδ mediates hypertrophy decompensation | Q35132584 | ||
CCR2 mediates the uptake of bone marrow-derived fibroblast precursors in angiotensin II-induced cardiac fibrosis | Q35159918 | ||
Tumor necrosis factor: a mechanistic link between angiotensin-II-induced cardiac inflammation and fibrosis. | Q35196311 | ||
Cardiac fibroblasts contribute to myocardial dysfunction in mice with sepsis: the role of NLRP3 inflammasome activation | Q35250425 | ||
A small-molecule inhibitor of the NLRP3 inflammasome for the treatment of inflammatory diseases. | Q35350543 | ||
The inflammasome promotes adverse cardiac remodeling following acute myocardial infarction in the mouse. | Q35621283 | ||
Up-regulated TLR4 in cardiomyocytes exacerbates heart failure after long-term myocardial infarction | Q35750924 | ||
Pharmacologic Inhibition of the NLRP3 Inflammasome Preserves Cardiac Function After Ischemic and Nonischemic Injury in the Mouse | Q35847003 | ||
Oxidation of CaMKII determines the cardiotoxic effects of aldosterone. | Q35903753 | ||
Angiotensin II revisited: new roles in inflammation, immunology and aging | Q36039299 | ||
Necrotic myocardial cells release damage-associated molecular patterns that provoke fibroblast activation in vitro and trigger myocardial inflammation and fibrosis in vivo. | Q36142000 | ||
Mononuclear Phagocytes Are Dispensable for Cardiac Remodeling in Established Pressure-Overload Heart Failure | Q36261243 | ||
Temporal neutrophil polarization following myocardial infarction | Q36702435 | ||
Macrophage activation by endogenous danger signals | Q37045743 | ||
Inflammation as a therapeutic target in heart failure? A scientific statement from the Translational Research Committee of the Heart Failure Association of the European Society of Cardiology | Q37090971 | ||
Ca2+/calmodulin-dependent kinase II triggers cell membrane injury by inducing complement factor B gene expression in the mouse heart | Q37143342 | ||
Requirement for Ca2+/calmodulin-dependent kinase II in the transition from pressure overload-induced cardiac hypertrophy to heart failure in mice. | Q37170829 | ||
Calcium/calmodulin-dependent protein kinase II links ER stress with Fas and mitochondrial apoptosis pathways. | Q37363053 | ||
Characterization of the inflammatory and fibrotic response in a mouse model of cardiac pressure overload. | Q37434655 | ||
Necrotic cells trigger a sterile inflammatory response through the Nlrp3 inflammasome | Q37453447 | ||
Endoplasmic reticulum stress in bone marrow-derived cells prevents acute cardiac inflammation and injury in response to angiotensin II. | Q37482793 | ||
PEDF Inhibits the Activation of NLRP3 Inflammasome in Hypoxia Cardiomyocytes through PEDF Receptor/Phospholipase A2. | Q37534751 | ||
Embryonic and adult-derived resident cardiac macrophages are maintained through distinct mechanisms at steady state and during inflammation | Q37581780 | ||
Mechanisms of cardiac fibrosis in inflammatory heart disease. | Q37743971 | ||
Emerging role of damage-associated molecular patterns derived from mitochondria in inflammation | Q37844960 | ||
Innate immune signaling in cardiac ischemia | Q37859249 | ||
Fibrosis and heart failure | Q38057701 | ||
Danger signals in the initiation of the inflammatory response after myocardial infarction | Q38173348 | ||
Myocyte signalling in leucocyte recruitment to the heart | Q38185647 | ||
The inflammasome in myocardial injury and cardiac remodeling | Q38261433 | ||
Complexity of danger: the diverse nature of damage-associated molecular patterns. | Q38267715 | ||
Inflammation in cardiac injury, repair and regeneration. | Q38390454 | ||
Role of inflammatory cells in fibroblast activation | Q38643076 | ||
The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis. | Q38875774 | ||
Chronic Heart Failure and Inflammation: What Do We Really Know? | Q38875783 | ||
Mechanism and Regulation of NLRP3 Inflammasome Activation | Q38964695 | ||
CaMKII is a nodal signal for multiple programmed cell death pathways in heart | Q39056528 | ||
ASC speck formation as a readout for inflammasome activation | Q39126034 | ||
CaMKIIδ subtypes differentially regulate infarct formation following ex vivo myocardial ischemia/reperfusion through NF-κB and TNF-α. | Q40385305 | ||
The Cardiomyocyte as a Source of Cytokines in Cardiac Injury | Q41256651 | ||
TNF receptor 1 signaling is critically involved in mediating angiotensin-II-induced cardiac fibrosis | Q41324289 | ||
Ca2+/Calmodulin-dependent protein kinase II δ mediates myocardial ischemia/reperfusion injury through nuclear factor-κB. | Q41584910 | ||
Angiotensin II induces afterdepolarizations via reactive oxygen species and calmodulin kinase II signaling. | Q41873666 | ||
Mitochondrial reactive oxygen species induces NLRP3-dependent lysosomal damage and inflammasome activation | Q41914695 | ||
Monocytic fibroblast precursors mediate fibrosis in angiotensin-II-induced cardiac hypertrophy | Q42097897 | ||
CaMKIIδ mediates β-adrenergic effects on RyR2 phosphorylation and SR Ca(2+) leak and the pathophysiological response to chronic β-adrenergic stimulation. | Q42114357 | ||
Optimized dosing of a CCR2 antagonist for amplification of vaccine immunity | Q42771123 | ||
BMS-345541 is a highly selective inhibitor of I kappa B kinase that binds at an allosteric site of the enzyme and blocks NF-kappa B-dependent transcription in mice | Q44196757 | ||
Inhibition of the NLRP3 inflammasome limits the inflammatory injury following myocardial ischemia-reperfusion in the mouse | Q45027851 | ||
Distinct effects of leukocyte and cardiac phosphoinositide 3-kinase γ activity in pressure overload-induced cardiac failure | Q45103986 | ||
Angiotensin II increases collagen I expression via transforming growth factor-beta1 and extracellular signal-regulated kinase in cardiac fibroblasts | Q46046832 | ||
Complement-induced activation of the cardiac NLRP3 inflammasome in sepsis | Q46499624 | ||
P4510 | describes a project that uses | ImageJ | Q1659584 |
P433 | issue | 12 | |
P577 | publication date | 2018-06-21 | |
P1433 | published in | JCI insight | Q27727187 |
P1476 | title | CaMKIIδ-mediated inflammatory gene expression and inflammasome activation in cardiomyocytes initiate inflammation and induce fibrosis | |
P478 | volume | 3 |
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Q91722872 | Genetic deletion of calcium/calmodulin-dependent protein kinase type II delta does not mitigate adverse myocardial remodeling in volume-overloaded hearts |
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