Factors Promoting Conduction Slowing as Substrates for Block and Reentry in Infarcted Hearts

scientific article published on 12 August 2019

Factors Promoting Conduction Slowing as Substrates for Block and Reentry in Infarcted Hearts is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1016/J.BPJ.2019.08.008
P932PMC publication ID6990374
P698PubMed publication ID31521328

P2093author name stringSébastien Roujol
Gernot Plank
Martin J Bishop
John Whitaker
Radhouene Neji
Mark O'Neill
Fernando O Campos
P2860cites workBasic Mechanisms of Cardiac Impulse Propagation and Associated ArrhythmiasQ22337019
Arrhythmia risk stratification of patients after myocardial infarction using personalized heart modelsQ27318488
Sodium current reduction unmasks a structure-dependent substrate for arrhythmogenesis in the normal ventriclesQ27328980
Local Gradients in Electrotonic Loading Modulate the Local Effective Refractory Period: Implications for Arrhythmogenesis in the Infarct Border ZoneQ33574084
Automatically generated, anatomically accurate meshes for cardiac electrophysiology problems.Q33642224
Remodeling of ventricular conduction pathways in healed canine infarct border zonesQ34187384
Overview of Basic Mechanisms of Cardiac ArrhythmiaQ34632042
Susceptibility to arrhythmia in the infarcted heart depends on myofibroblast density.Q35224007
Investigating a Novel Activation-Repolarisation Time Metric to Predict Localised Vulnerability to Reentry Using Computational ModellingQ35943221
Is heart size a factor in ventricular fibrillation? Or how close are rabbit and human hearts?Q36526285
Infarct tissue heterogeneity by magnetic resonance imaging identifies enhanced cardiac arrhythmia susceptibility in patients with left ventricular dysfunctionQ36741315
Arrhythmogenic ion-channel remodeling in the heart: heart failure, myocardial infarction, and atrial fibrillation.Q36788875
The functional role of electrophysiological heterogeneity in the rabbit ventricle during rapid pacing and arrhythmiasQ36837988
Accelerating cardiac bidomain simulations using graphics processing unitsQ36966728
Ventricular scars and ventricular tachycardiaQ37600649
An efficient finite element approach for modeling fibrotic clefts in the heartQ37677070
Cardiac fibrosis and arrhythmogenesis: the road to repair is paved with perilsQ37716873
Fibrosis and cardiac arrhythmiasQ37819889
Arrhythmogenic implications of fibroblast-myocyte interactionsQ38003371
Image-Based Personalization of Cardiac Anatomy for Coupled Electromechanical Modeling.Q38596284
Massive Accumulation of Myofibroblasts in the Critical Isthmus Is Associated With Ventricular Tachycardia Inducibility in Post-Infarct Swine Heart.Q38645319
Imaging-Based Simulations for Predicting Sudden Death and Guiding Ventricular Tachycardia AblationQ39428205
Virtual electrophysiological study in a 3-dimensional cardiac magnetic resonance imaging model of porcine myocardial infarction.Q39612848
Accuracy of prediction of infarct-related arrhythmic circuits from image-based models reconstructed from low and high resolution MRI.Q40362730
Increased afterload following myocardial infarction promotes conduction-dependent arrhythmias that are unmasked by hypokalemiaQ41296175
Tachycardia in post-infarction hearts: insights from 3D image-based ventricular models.Q41299379
A novel rule-based algorithm for assigning myocardial fiber orientation to computational heart models.Q42127914
Representing cardiac bidomain bath-loading effects by an augmented monodomain approach: application to complex ventricular models.Q42715688
Amiodarone: an effective antiarrhythmic drug with unusual side effectsQ42908838
Three-dimensional mechanisms of increased vulnerability to electric shocks in myocardial infarction: altered virtual electrode polarizations and conduction delay in the peri-infarct zone.Q44095153
Delayed rectifier K currents have reduced amplitudes and altered kinetics in myocytes from infarcted canine ventricleQ44877000
Relationship between voltage map "channels" and the location of critical isthmus sites in patients with post-infarction cardiomyopathy and ventricular tachycardia.Q45918991
Dispersion of repolarization and arrhythmogenesisQ46073149
Pulmonary vein reentry--properties and size matter: insights from a computational analysisQ47257801
Optical mapping of Langendorff-perfused human hearts: establishing a model for the study of ventricular fibrillation in humans.Q51043950
Computational tools for modeling electrical activity in cardiac tissue.Q51723812
Complex electrophysiological remodeling in postinfarction ischemic heart failure.Q52358398
Effect of rate on changes in conduction velocity and extracellular potassium concentration during acute ischemia in the in situ pig heart.Q54038738
Electrotonic influences on action potentials from isolated ventricular cells.Q54312599
Modeling the Electrophysiological Properties of the Infarct Border Zone.Q54941313
Ectopic beats arise from micro-reentries near infarct regions in simulations of a patient-specific heart modelQ58608787
Fibrosis Microstructure Modulates Reentry in Non-ischemic Dilated Cardiomyopathy: Insights From Imaged Guided 2D Computational ModelingQ60947382
Real-time interactive simulations of large-scale systems on personal computers and cell phones: Toward patient-specific heart modeling and other applicationsQ64115634
Reentry as a cause of ventricular tachycardia in patients with chronic ischemic heart disease: electrophysiologic and anatomic correlationQ68266650
Block of impulse propagation at an abrupt tissue expansion: evaluation of the critical strand diameter in 2- and 3-dimensional computer modelsQ71806051
Slow conduction in the infarcted human heart. 'Zigzag' course of activationQ72885921
Alterations of Na+ currents in myocytes from epicardial border zone of the infarcted heart. A possible ionic mechanism for reduced excitability and postrepolarization refractorinessQ73454279
M cells and transmural heterogeneity of action potential configuration in myocytes from the left ventricular wall of the pig heartQ73573714
Inhomogeneous transmural conduction during early ischaemia in patients with coronary artery diseaseQ73650748
Ionic mechanisms of propagation in cardiac tissue. Roles of the sodium and L-type calcium currents during reduced excitability and decreased gap junction couplingQ73833055
Slow conduction in cardiac tissue, II: effects of branching tissue geometryQ77426338
Dynamic changes in conduction velocity and gap junction properties during development of pacing-induced heart failureQ80160002
Laminar arrangement of ventricular myocytes influences electrical behavior of the heartQ81464508
Structural heterogeneity alone is a sufficient substrate for dynamic instability and altered restitutionQ82791523
Alternans and spiral breakup in a human ventricular tissue modelQ82973183
Ventricular tachycardia and sudden cardiac deathQ83406806
High-resolution 3-dimensional reconstruction of the infarct border zone: impact of structural remodeling on electrical activationQ84417351
Localizing the critical isthmus of postinfarct ventricular tachycardia: the value of pace-mapping during sinus rhythmQ87265042
Role of 3-Dimensional Architecture of Scar and Surviving Tissue in Ventricular Tachycardia: Insights From High-Resolution Ex Vivo Porcine ModelsQ89030298
Characterizing the clinical implementation of a novel activation-repolarization metric to identify targets for catheter ablation of ventricular tachycardias using computational modelsQ90171594
Personalized virtual-heart technology for guiding the ablation of infarct-related ventricular tachycardiaQ92211968
P433issue12
P1104number of pages14
P304page(s)2361-2374
P577publication date2019-08-12
P1433published inBiophysical JournalQ2032955
P1476titleFactors Promoting Conduction Slowing as Substrates for Block and Reentry in Infarcted Hearts
P478volume117

Reverse relations

Q98781360The study of border zone formation in ischemic heart using electro-chemical coupled computational modelcites workP2860

Search more.