Origins of Alterations to Rankl Null Mutant Mouse Dental Root Development

scientific article published on 23 March 2020

Origins of Alterations to Rankl Null Mutant Mouse Dental Root Development is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.3390/IJMS21062201
P932PMC publication ID7139335
P698PubMed publication ID32209985

P50authorDominique HeymannQ43131742
Andrea GamaQ90631422
Ana Carolina AcevedoQ90631425
Frédéric LézotQ90631428
P2093author name stringSylvie Babajko
Ariane Berdal
Beatriz Castaneda
Jérome Amiaud
Jorge William Vargas-Franco
Diana Carolina Sánchez Mesa
Elizabeth Restrepo Bedoya
P2860cites workRANK is essential for osteoclast and lymph node developmentQ24598872
Bone resorption: an actor of dental and periodontal development?Q28081762
Opg, Rank, and Rankl in tooth development: co-ordination of odontogenesis and osteogenesisQ28504606
Regulation of bone formation and remodeling by G-protein-coupled receptor 48Q28507625
R-spondins/Lgrs expression in tooth developmentQ28513901
OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesisQ28589430
Receptor activator of NF-kappaB (RANK) stimulates the proliferation of epithelial cells of the epidermo-pilosebaceous unitQ34750367
Role of RANKL (TNFSF11)-dependent osteopetrosis in the dental phenotype of Msx2 null mutant miceQ35053424
From the ranks of mammary progesterone mediators, RANKL takes the spotlightQ35659426
Osteoblast Lineage Cells Play an Essential Role in Periodontal Bone Loss Through Activation of Nuclear Factor-Kappa BQ36370991
RANK is the intrinsic hematopoietic cell surface receptor that controls osteoclastogenesis and regulation of bone mass and calcium metabolismQ37108966
Diverse roles of the tumor necrosis factor family member TRANCE in skeletal physiology revealed by TRANCE deficiency and partial rescue by a lymphocyte-expressed TRANCE transgeneQ37265676
High- and low-dose OPG-Fc cause osteopetrosis-like changes in infant mice.Q37463716
Formation of the tooth-bone interface.Q37665094
Msx2 -/- transgenic mice develop compound amelogenesis imperfecta, dentinogenesis imperfecta and periodental osteopetrosis.Q38298208
LGR4 acts as a key receptor for R-spondin 2 to promote osteogenesis through Wnt signaling pathwayQ38773507
LGR4 is required for sequential molar developmentQ41682698
Treatment of OPG-deficient mice with WP9QY, a RANKL-binding peptide, recovers alveolar bone loss by suppressing osteoclastogenesis and enhancing osteoblastogenesisQ41995909
The role of RANKL (TRANCE/TNFSF11), a tumor necrosis factor family member, in skeletal development: effects of gene knockout and transgenic rescueQ46286533
OPG knockout mouse teeth display reduced alveolar bone mass and hypermineralization in enamel and dentinQ46296901
Bone resorption control of tooth eruption and root morphogenesis: Involvement of the receptor activator of NF-κB (RANK).Q46356299
Osteoclastogenesis during mouse tooth germ development is mediated by receptor activator of NFKappa-B ligand (RANKL).Q47702660
Blockade of LGR4 inhibits proliferation and odonto/osteogenic differentiation of stem cells from apical papillae.Q48229864
Tooth-bone morphogenesis during postnatal stages of mouse first molar development.Q51878904
Bone resorption deficiency affects tooth root development in RANKL mutant mice due to attenuated IGF-1 signaling in radicular odontoblasts.Q52687454
LGR4 is a receptor for RANKL and negatively regulates osteoclast differentiation and bone resorption.Q54200830
Interactions of the tooth and bone during development.Q54704161
The Intrinsic and Extrinsic Implications of RANKL/RANK Signaling in Osteosarcoma: From Tumor Initiation to Lung MetastasesQ57790452
Maternal RANKL Reduces the Osteopetrotic Phenotype of Null Mutant Mouse PupsQ58582775
Skeletal consequences of RANKL-blocking antibody (IK22-5) injections during growth: Mouse strain disparities and synergic effect with zoledronic acidQ61051801
Osteoclasts in the Dental Microenvironment: A Delicate Balance Controls Dental HistogenesisQ61051809
The RANKL-RANK Axis: A Bone to Thymus Round TripQ64102143
Loss of Lgr4 inhibits differentiation, migration and apoptosis, and promotes proliferation in bone mesenchymal stem cellsQ90398444
Genetically-achieved disturbances to the expression levels of TNFSF11 receptors modulate the effects of zoledronic acid on growing mouse skeletonsQ91510619
P275copyright licenseCreative Commons Attribution 4.0 InternationalQ20007257
P6216copyright statuscopyrightedQ50423863
P433issue6
P577publication date2020-03-23
P1433published inInternational Journal of Molecular SciencesQ3153277
P1476titleOrigins of Alterations to Rankl Null Mutant Mouse Dental Root Development
P478volume21

Search more.