scholarly article | Q13442814 |
P50 | author | Hao Jiang | Q89351537 |
P2093 | author name string | Changyi Li | |
P2860 | cites work | Nicotinamide enhances repair of ultraviolet radiation-induced DNA damage in primary melanocytes | Q87816653 |
Toll-like receptor 2 and P. acnes: do they trigger initial acne vulgaris lesions? | Q95403240 | ||
Mammalian wax biosynthesis. II. Expression cloning of wax synthase cDNAs encoding a member of the acyltransferase enzyme family | Q24655639 | ||
Toll-like receptor signalling in macrophages links the autophagy pathway to phagocytosis | Q29614481 | ||
The role of facial sebum secretion in acne pathogenesis: facts and controversies | Q33524947 | ||
Oxidant/antioxidant status in obese adolescent females with acne vulgaris | Q33567875 | ||
The immune system in atherosclerosis | Q34164644 | ||
The complete genome sequence of Propionibacterium acnes, a commensal of human skin | Q34337656 | ||
New insights into the regulation of HDL metabolism and reverse cholesterol transport | Q34428702 | ||
Inflammatory events are involved in acne lesion initiation | Q34535060 | ||
The macrophage foam cell as a target for therapeutic intervention | Q34986351 | ||
Myeloperoxidase in human neutrophil host defence | Q34994685 | ||
New perspectives on the use of niacin in the treatment of lipid disorders | Q35743885 | ||
Differential lipid metabolism in monocytes and macrophages: influence of cholesterol loading | Q36731741 | ||
Update on strategies to increase HDL quantity and function | Q37504704 | ||
New developments in our understanding of acne pathogenesis and treatment | Q37530982 | ||
NADPH oxidase promotes neutrophil extracellular trap formation in pulmonary aspergillosis | Q37713391 | ||
The versatility of HDL: a crucial anti-inflammatory regulator. | Q37778848 | ||
The tolls and dangers of atherosclerotic disease | Q37862325 | ||
Multiple roles for neutrophils in atherosclerosis | Q37994653 | ||
Lipid homeostasis and the formation of macrophage-derived foam cells in atherosclerosis | Q37996954 | ||
Acne is an inflammatory disease and alterations of sebum composition initiate acne lesions | Q38153454 | ||
Sebum, inflammasomes and the skin: current concepts and future perspective | Q39002277 | ||
Sebaceous gland lipids. | Q39533668 | ||
Sebum analysis of individuals with and without acne | Q39534170 | ||
Therapy for acne vulgaris | Q41429600 | ||
Toll-like receptors stimulate human neutrophil function | Q44493366 | ||
Accumulation of lipid peroxide in the content of comedones may be involved in the progression of comedogenesis and inflammatory changes in comedones. | Q45968731 | ||
Serum homocysteine levels in acne patients. | Q47574652 | ||
Study of Facial Sebum Levels and Follicular Red Fluorescence in Patients with Acne Vulgaris in Nigeria. | Q50277331 | ||
Acne patients frequently associated with abnormal plasma lipid profile. | Q51060812 | ||
Mechanical stress and the development of pseudo-comedones and tunnels in Hidradenitis suppurativa/Acne inversa. | Q54689860 | ||
Phagocytic killing of microorganisms by radical processes: consequences of the reaction of hydroxyl radicals with chloride yielding chlorine atoms | Q77814524 | ||
Pathogenesis of atherosclerosis | Q81520313 | ||
Pathogenesis of atherosclerosis | Q83187658 | ||
P433 | issue | 1 | |
P304 | page(s) | 1-5 | |
P577 | publication date | 2019-02-01 | |
P1433 | published in | Inflammation | Q2560341 |
P1476 | title | Common Pathogenesis of Acne Vulgaris and Atherosclerosis | |
P478 | volume | 42 |
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