| scholarly article | Q13442814 |
| P2093 | author name string | Feng He | |
| Zhenjun Tian | |||
| Jianshe Zhang | |||
| Wuying Chu | |||
| Mengxin Cai | |||
| Shaojun Du | |||
| Lusha Liu | |||
| Lichen Han | |||
| P2860 | cites work | Functional analysis of slow myosin heavy chain 1 and myomesin-3 in sarcomere organization in zebrafish embryonic slow muscles. | Q38578182 |
| Sarcomere Dysfunction in Nemaline Myopathy | Q39258305 | ||
| Genetic compensation induced by deleterious mutations but not gene knockdowns. | Q40744194 | ||
| Myofibrillogenesis in skeletal muscle cells in zebrafish | Q42027499 | ||
| Proteomic analysis of organ-specific post-translational lysine-acetylation and -methylation in mice by use of anti-acetyllysine and -methyllysine mouse monoclonal antibodies | Q42485737 | ||
| The chromatin-binding protein Smyd1 restricts adult mammalian heart growth | Q42784088 | ||
| Correction: Still Heart Encodes a Structural HMT, SMYD1b, with Chaperone-Like Function during Fast Muscle Sarcomere Assembly | Q43073585 | ||
| Methylation, a new epigenetic mark for protein stability | Q43261361 | ||
| The UCS factor Steif/Unc-45b interacts with the heat shock protein Hsp90a during myofibrillogenesis | Q47073713 | ||
| The myosin co-chaperone UNC-45 is required for skeletal and cardiac muscle function in zebrafish | Q47073763 | ||
| The myosin-interacting protein SMYD1 is essential for sarcomere organization | Q47073895 | ||
| mRNA processing in mutant zebrafish lines generated by chemical and CRISPR-mediated mutagenesis produces unexpected transcripts that escape nonsense-mediated decay | Q47150815 | ||
| Zebrafish Embryonic Slow Muscle Is a Rapid System for Genetic Analysis of Sarcomere Organization by CRISPR/Cas9, but Not NgAgo | Q49642318 | ||
| Loss of zebrafish Smyd1a interferes with myofibrillar integrity without triggering the misfolded myosin response | Q50055639 | ||
| Muscle-specific expression of the smyd1 gene is controlled by its 5.3-kb promoter and 5'-flanking sequence in zebrafish embryos. | Q50711281 | ||
| Defective myogenesis in the absence of the muscle-specific lysine methyltransferase SMYD1. | Q51598998 | ||
| siRNA-mediated inhibition of skNAC and Smyd1 expression disrupts myofibril organization: Immunofluorescence and electron microscopy study in C2C12 cells. | Q52369214 | ||
| The sarcomere and sarcomerogenesis. | Q52591693 | ||
| The sequence of the NH2-terminal 204-residue fragment of the heavy chain of rabbit skeletal muscle myosin | Q71070903 | ||
| Histone methyltransferase Smyd1 regulates mitochondrial energetics in the heart | Q90646232 | ||
| Genome-wide survey and developmental expression mapping of zebrafish SET domain-containing genes | Q21562301 | ||
| skNAC, a Smyd1-interacting transcription factor, is involved in cardiac development and skeletal muscle growth and regeneration | Q24306754 | ||
| SMYD1, the myogenic activator, is a direct target of serum response factor and myogenin | Q24318909 | ||
| Efficient genome editing in zebrafish using a CRISPR-Cas system | Q24610828 | ||
| SMYD proteins: key regulators in skeletal and cardiac muscle development and function | Q26865167 | ||
| m-Bop, a repressor protein essential for cardiogenesis, interacts with skNAC, a heart- and muscle-specific transcription factor | Q28217589 | ||
| Targeting the sarcomere to correct muscle function | Q28260629 | ||
| How to build a myofibril | Q28296222 | ||
| BOP, a regulator of right ventricular heart development, is a direct transcriptional target of MEF2C in the developing heart | Q28505957 | ||
| Bop encodes a muscle-restricted protein containing MYND and SET domains and is essential for cardiac differentiation and morphogenesis | Q28587459 | ||
| The ATPase-dependent chaperoning activity of Hsp90a regulates thick filament formation and integration during skeletal muscle myofibrillogenesis | Q28754755 | ||
| High-resolution in situ hybridization to whole-mount zebrafish embryos | Q29617529 | ||
| Heat-shock protein 90alpha1 is required for organized myofibril assembly in skeletal muscles of zebrafish embryos. | Q30481082 | ||
| Knockdown and overexpression of Unc-45b result in defective myofibril organization in skeletal muscles of zebrafish embryos. | Q33694916 | ||
| Frameshift indels introduced by genome editing can lead to in-frame exon skipping. | Q33751947 | ||
| SmyD1, a histone methyltransferase, is required for myofibril organization and muscle contraction in zebrafish embryos. | Q34928212 | ||
| Reverse genetic screening reveals poor correlation between morpholino-induced and mutant phenotypes in zebrafish. | Q35806361 | ||
| In vivo protein trapping produces a functional expression codex of the vertebrate proteome | Q35835730 | ||
| SMYD1 and G6PD modulation are critical events for miR-206-mediated differentiation of rhabdomyosarcoma | Q36186504 | ||
| Mouse myofibers lacking the SMYD1 methyltransferase are susceptible to atrophy, internalization of nuclei and myofibrillar disarray | Q36802641 | ||
| Efficient multiplex biallelic zebrafish genome editing using a CRISPR nuclease system | Q37117695 | ||
| Smyd1b is required for skeletal and cardiac muscle function in zebrafish | Q37306191 | ||
| Expression and functional characterization of Smyd1a in myofibril organization of skeletal muscles | Q37510551 | ||
| Lysine methylation of nonhistone proteins is a way to regulate their stability and function. | Q37772914 | ||
| Lysine methylation: beyond histones | Q37970950 | ||
| skNAC and Smyd1 in transcriptional control | Q38297716 | ||
| P433 | issue | 5 | |
| P304 | page(s) | 6209-6225 | |
| P577 | publication date | 2019-02-28 | |
| P1433 | published in | FASEB Journal | Q520194 |
| P1476 | title | Defective sarcomere assembly in smyd1a and smyd1b zebrafish mutants | |
| P478 | volume | 33 |
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