| P50 | author | Robert Maile | Q51777038 |
| P2093 | author name string | Matthew C Wolfgang | |
| | Cindy J Gode | |
| | Bruce A Cairns | |
| | Laurel B Kartchner | |
| | Danté N Duncan | |
| | Julia L M Dunn | |
| | Lindsey I Glenn | |
| P2860 | cites work | Staphylococcus aureus α-hemolysin mediates virulence in a murine model of severe pneumonia through activation of the NLRP3 inflammasome | Q24594277 |
| | The diagnostic and prognostic value of systems biology research in major traumatic and thermal injury: a review | Q28079517 |
| | Reactive oxygen species in inflammation and tissue injury | Q28396273 |
| | The influence of metabolic imbalances and oxidative stress on the outcome of critically ill polytrauma patients: a review. | Q30235314 |
| | Divergent adaptive and innate immunological responses are observed in humans following blunt trauma. | Q33526479 |
| | Long-term persistance of the pathophysiologic response to severe burn injury | Q33971060 |
| | Animal models in burn research | Q34051592 |
| | Nurses' uniforms: How many bacteria do they carry after one shift? | Q34268677 |
| | Bacterial colonization and succession in a newly opened hospital. | Q34557144 |
| | Surgical burn wound infections and their clinical implications | Q34664068 |
| | Bronchoscopy-derived correlates of lung injury following inhalational injuries: a prospective observational study | Q34731383 |
| | Interferon-γ production by neutrophils during bacterial pneumonia in mice | Q35042522 |
| | Role of cytokines as a double-edged sword in sepsis | Q35230688 |
| | Innate immune responses to Pseudomonas aeruginosa infection. | Q35566398 |
| | Association between early airway damage-associated molecular patterns and subsequent bacterial infection in patients with inhalational and burn injury | Q35574722 |
| | A genomic storm in critically injured humans | Q35627656 |
| | Local Application of Probiotic Bacteria Prophylaxes against Sepsis and Death Resulting from Burn Wound Infection | Q36174172 |
| | Circulating Microvesicles Are Elevated Acutely following Major Burns Injury and Associated with Clinical Severity | Q36218569 |
| | ROS-Mediated NLRP3 Inflammasome Activity Is Essential for Burn-Induced Acute Lung Injury | Q36241489 |
| | Heterogeneity of mast cells and expression of Annexin A1 protein in a second degree burn model with silver sulfadiazine treatment | Q36302570 |
| | Inflammatory/cardiovascular-metabolic responses in a rat model of burn injury with superimposed infection | Q36351636 |
| | Impact of burn priming on immune and metabolic functions of whole Liver in a rat cecal ligation and puncture model | Q36576531 |
| | Temporal cytokine profiles in severely burned patients: a comparison of adults and children | Q36714067 |
| | Radiation combined with thermal injury induces immature myeloid cells | Q36714653 |
| | Stimulation of lung innate immunity protects against lethal pneumococcal pneumonia in mice | Q36718650 |
| | Oxidative stress and anti-oxidative mobilization in burn injury | Q36955628 |
| | Airway and lung pathology due to mucosal surface dehydration in {beta}-epithelial Na+ channel-overexpressing mice: role of TNF-{alpha} and IL-4R{alpha} signaling, influence of neonatal development, and limited efficacy of glucocorticoid treatment | Q37137320 |
| | The hypermetabolic response to burn injury and interventions to modify this response | Q37181299 |
| | Activation of Adenosine 2A receptor inhibits neutrophil apoptosis in an autophagy-dependent manner in mice with systemic inflammatory response syndrome | Q37267492 |
| | The compensatory anti-inflammatory response syndrome (CARS) in critically ill patients | Q37309561 |
| | TNF-α/IL-10 Ratio Correlates with Burn Severity and May Serve as a Risk Predictor of Increased Susceptibility to Infections | Q37310054 |
| | Burn injury induces high levels of phosphorylated insulin-like growth factor binding protein-1 | Q37310264 |
| | Flagellin treatment prevents increased susceptibility to systemic bacterial infection after injury by inhibiting anti-inflammatory IL-10+ IL-12- neutrophil polarization | Q37484450 |
| | Burns as a model of SIRS. | Q37501775 |
| | G-CSF drives a posttraumatic immune program that protects the host from infection | Q37620301 |
| | Interactions between Neutrophils and Pseudomonas aeruginosa in Cystic Fibrosis | Q37727995 |
| | Epidemiology of bloodstream infections in burn-injured patients: a review of the national burn repository | Q37771201 |
| | The role of TLRs in neutrophil activation | Q37899376 |
| | Mechanisms of phagocytosis and host clearance of Pseudomonas aeruginosa. | Q38182026 |
| | Ethyl pyruvate reverses development of Pseudomonas aeruginosa pneumonia during sepsis-induced immunosuppression. | Q38597749 |
| | Systemic inflammatory response syndrome following burns is mediated by brain natriuretic peptide/natriuretic peptide A receptor-induced shock factor 1 signaling pathway | Q38759448 |
| | Innate danger signals in acute injury: From bench to bedside | Q38778654 |
| | The Physiologic Basis of Burn Shock and the Need for Aggressive Fluid Resuscitation. | Q38947554 |
| | Innate Immune Cell Recovery Is Positively Regulated by NLRP12 during Emergency Hematopoiesis | Q38979899 |
| | Differential regulation of innate immune cytokine production through pharmacological activation of Nuclear Factor-Erythroid-2-Related Factor 2 (NRF2) in burn patient immune cells and monocytes. | Q40060254 |
| | Timeline of health care-associated infections and pathogens after burn injuries | Q40506586 |
| | Direct detection of blood nitric oxide reveals a burn-dependent decrease of nitric oxide in response to Pseudomonas aeruginosa infection. | Q40643348 |
| | Immunogenicity and protective efficacy of Pseudomonas aeruginosa type a and b flagellin vaccines in a burned mouse model | Q40684952 |
| | Burn-Evoked Reactive Oxygen Species Immediately After Injury are Crucial to Restore the Neutrophil Function Against Postburn Infection in Mice | Q41203466 |
| | Immune responses in relation to the type and time of thermal injury: an experimental study. | Q41622696 |
| | ROS generation, lipid peroxidation and antioxidant enzyme activities in the aging brain | Q42513093 |
| | A ribonucleotide reductase inhibitor reverses burn-induced inflammatory defects | Q43102126 |
| | Prior thermal injury accelerates endotoxin-induced inflammatory cytokine production and intestinal nuclear factor-κB activation in mice | Q43199024 |
| | Proinflammatory clearance of apoptotic neutrophils induces an IL-12(low)IL-10(high) regulatory phenotype in macrophages | Q43820826 |
| | Injury primes the innate immune system for enhanced Toll-like receptor reactivity | Q44002949 |
| | Multiple contributing roles for NOS2 in LPS-induced acute airway inflammation in mice | Q44583874 |
| | Experimental studies of the pathogenesis of infections due to Pseudomonas aeruginosa: description of a burned mouse model | Q44717986 |
| | Toll-Like Receptor Signaling in Burn Wound Healing and Scarring | Q45415293 |
| | Blocking CXCL1-dependent neutrophil recruitment prevents immune damage and reduces pulmonary bacterial infection after inhalation injury. | Q47673945 |
| | Effect from multiple episodes of inadequate empiric antibiotic therapy for ventilator-associated pneumonia on morbidity and mortality among critically ill trauma patients | Q47859194 |
| | Nucleosomes and neutrophil extracellular traps in septic and burn patients | Q47869045 |
| | Microbiology in burns patients with blood stream infections: trends over time and during the course of hospitalization. | Q48361500 |
| | Damage-associated molecular patterns (DAMPs) released after burn are associated with inflammation and monocyte activation. | Q50774937 |
| | Impact of Isolated Burns on Major Organs: A Large Animal Model Characterized. | Q51496516 |
| | Toll-like receptor 2 and 4 ligation results in complex altered cytokine profiles early and late after burn injury. | Q51706893 |
| | Increased Toll-like receptor 4 expression on T cells may be a mechanism for enhanced T cell response late after burn injury. | Q51980102 |
| | Effect of thermal injury upon the early resistance to infection. | Q54696129 |
| | Linking the “two-hit” response following injury to enhanced TLR4 reactivity | Q64447742 |
| | Thermal injury-induced non-specific resistance to fatal Pseudomonas aeruginosa burn-infection in mice | Q69363446 |
| | Injury enhances resistance to Escherichia coli infection by boosting innate immune system function | Q80633629 |
| | Neutrophils, not monocyte/macrophages, are the major splenic source of postburn IL-10 | Q84073325 |
| P433 | issue | 3 | |
| P304 | page(s) | 627-640 | |
| P577 | publication date | 2019-03-02 | |
| P1433 | published in | Burns | Q25392382 |
| P1476 | title | One-hit wonder: Late after burn injury, granulocytes can clear one bacterial infection but cannot control a subsequent infection | |
| P478 | volume | 45 | |