Looking to the future of zebrafish as a model to understand the genetic basis of eye disease

scientific article published on 17 August 2019

Looking to the future of zebrafish as a model to understand the genetic basis of eye disease is …
instance of (P31):
scholarly articleQ13442814
review articleQ7318358

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P356DOI10.1007/S00439-019-02055-Z
P932PMC publication ID6710215
P698PubMed publication ID31422478

P50authorStephen W WilsonQ38320460
Florencia CavodeassiQ56606118
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Concerted action of neuroepithelial basal shrinkage and active epithelial migration ensures efficient optic cup morphogenesis.Q42292835
Early stages of zebrafish eye formation require the coordinated activity of Wnt11, Fz5, and the Wnt/beta-catenin pathwayQ42958376
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TGFβ-facilitated optic fissure fusion and the role of bone morphogenetic protein antagonism.Q53834248
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Compensatory growth renders Tcf7l1a dispensable for eye formation despite its requirement in eye field specificationQ61795789
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Detailed analysis of chick optic fissure closure reveals Netrin-1 as an essential mediator of epithelial fusionQ83232103
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PTC-bearing mRNA elicits a genetic compensation response via Upf3a and COMPASS componentsQ92830452
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P433issue8-9
P921main subjecteye diseaseQ3041498
P304page(s)993-1000
P577publication date2019-08-17
P1433published inHuman GeneticsQ5937167
P1476titleLooking to the future of zebrafish as a model to understand the genetic basis of eye disease
P478volume138

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