scholarly article | Q13442814 |
P2093 | author name string | Eva Martin | |
Michael Schuler | |||
Sabine Weigle | |||
Benjamin Wahl | |||
Andrea Voegtle | |||
P2860 | cites work | Fucoxanthin Inhibits Myofibroblast Differentiation and Extracellular Matrix Production in Nasal Polyp-Derived Fibroblasts via Modulation of Smad-Dependent and Smad-Independent Signaling Pathways | Q58753101 |
Oxymatrine Inhibits Transforming Growth Factor β1 (TGF-β1)-Induced Cardiac Fibroblast-to-Myofibroblast Transformation (FMT) by Mediating the Notch Signaling Pathway In Vitro | Q58757229 | ||
Organ and tissue fibrosis: Molecular signals, cellular mechanisms and translational implications | Q62734535 | ||
Enhanced Genome Editing with Cas9 Ribonucleoprotein in Diverse Cells and Organisms | Q64389471 | ||
TGF-beta 1, but not TGF-beta 2 or TGF-beta 3, is differentially present in epithelial cells of advanced pulmonary fibrosis: an immunohistochemical study | Q71078177 | ||
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Epithelial mesenchymal transition (EMT): a universal process in lung diseases with implications for cystic fibrosis pathophysiology | Q90319217 | ||
Establishing a Cell-Based High-Content Screening Assay for TCM Compounds with Anti-Renal Fibrosis Effects | Q90536836 | ||
RNAi/CRISPR Screens: from a Pool to a Valid Hit | Q91275315 | ||
Signaling Mechanisms of Myofibroblastic Activation: Outside-in and Inside-Out | Q91299535 | ||
When Quality Beats Quantity: Decision Theory, Drug Discovery, and the Reproducibility Crisis | Q24288687 | ||
Epithelial-mesenchymal transition and its implications for fibrosis | Q24302042 | ||
Beyond editing: repurposing CRISPR-Cas9 for precision genome regulation and interrogation | Q26775746 | ||
RNAi screening comes of age: improved techniques and complementary approaches | Q28654521 | ||
A high-content EMT screen identifies multiple receptor tyrosine kinase inhibitors with activity on TGFβ receptor | Q28828478 | ||
Chemical regulators of epithelial plasticity reveal a nuclear receptor pathway controlling myofibroblast differentiation | Q28829818 | ||
How were new medicines discovered? | Q29616641 | ||
Highly efficient RNA-guided genome editing in human cells via delivery of purified Cas9 ribonucleoproteins | Q33657682 | ||
Systems biology approaches to understanding Epithelial Mesenchymal Transition (EMT) in mucosal remodeling and signaling in asthma | Q33796731 | ||
IL-22 contributes to TGF-β1-mediated epithelial-mesenchymal transition in asthmatic bronchial epithelial cells | Q34248468 | ||
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Eosinophils promote epithelial to mesenchymal transition of bronchial epithelial cells. | Q34735424 | ||
Transforming growth factors-beta 1, -beta 2, and -beta 3 stimulate fibroblast procollagen production in vitro but are differentially expressed during bleomycin-induced lung fibrosis | Q35763549 | ||
Kupffer cells-dependent inflammation in the injured liver increases recruitment of mesenchymal stem cells in aging mice | Q36738749 | ||
The Scar-in-a-Jar: studying potential antifibrotic compounds from the epigenetic to extracellular level in a single well. | Q37434404 | ||
PARP3 controls TGFβ and ROS driven epithelial-to-mesenchymal transition and stemness by stimulating a TG2-Snail-E-cadherin axis. | Q37665592 | ||
The pathogenesis of pulmonary fibrosis: a moving target | Q38055722 | ||
Developing predictive assays: the phenotypic screening "rule of 3". | Q38537329 | ||
Highly efficient gene inactivation by adenoviral CRISPR/Cas9 in human primary cells. | Q38628654 | ||
miR-93 promotes TGF-β-induced epithelial-to-mesenchymal transition through downregulation of NEDD4L in lung cancer cells | Q38818616 | ||
Basal cells of the human airways acquire mesenchymal traits in idiopathic pulmonary fibrosis and in culture | Q38833878 | ||
CFTR inactivation by lentiviral vector-mediated RNA interference and CRISPR-Cas9 genome editing in human airway epithelial cells | Q38844377 | ||
Opportunities and challenges in phenotypic drug discovery: an industry perspective | Q39420418 | ||
Collagen matrix deposition is dramatically enhanced in vitro when crowded with charged macromolecules: the biological relevance of the excluded volume effect. | Q40127292 | ||
In vitro enhancement of collagen matrix formation and crosslinking for applications in tissue engineering: a preliminary study. | Q40129393 | ||
Efficient delivery of nuclease proteins for genome editing in human stem cells and primary cells | Q40403124 | ||
Up-regulation of Syndecan-4 contributes to TGF-β1-induced epithelial to mesenchymal transition in lung adenocarcinoma A549 cells | Q41683435 | ||
Comparative analysis of lysyl oxidase (like) family members in pulmonary fibrosis | Q41977476 | ||
Expression of α-Smooth Muscle Actin Determines the Fate of Mesenchymal Stromal Cells. | Q42003431 | ||
Epithelial-mesenchymal transition in primary human bronchial epithelial cells is Smad-dependent and enhanced by fibronectin and TNF-alpha | Q42917434 | ||
Small molecule-mediated inhibition of myofibroblast transdifferentiation for the treatment of fibrosis | Q43083609 | ||
Optimized RNP transfection for highly efficient CRISPR/Cas9-mediated gene knockout in primary T cells. | Q50024744 | ||
The big five in fibrosis: Macrophages, myofibroblasts, matrix, mechanics, and miscommunication. | Q50170928 | ||
Revealing the pathogenic and aging-related mechanisms of the enigmatic idiopathic pulmonary fibrosis. an integral model. | Q50676736 | ||
Rapid and highly efficient mammalian cell engineering via Cas9 protein transfection. | Q52423691 | ||
RNAi screening identifies a mechanosensitive ROCK-JAK2-STAT3 network central to myofibroblast activation. | Q52573450 | ||
Transition of asthmatic bronchial fibroblasts to myofibroblasts is inhibited by cell-cell contacts. | Q53229056 | ||
Idiopathic pulmonary fibrosis | Q56764887 | ||
P433 | issue | 2 | |
P304 | page(s) | e115 | |
P577 | publication date | 2019-06-03 | |
P1433 | published in | Journal of biological methods | Q27725861 |
P1476 | title | Primary cell-based phenotypic assays to pharmacologically and genetically study fibrotic diseases in vitro | |
P478 | volume | 6 |
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