scholarly article | Q13442814 |
P2093 | author name string | Michael P Czubryt | |
Krista L Filomeno | |||
Ian M C Dixon | |||
Morvarid S Kavosh | |||
Sunil G Rattan | |||
Natalie Landry | |||
P2860 | cites work | Ski is a component of the histone deacetylase complex required for transcriptional repression by Mad and thyroid hormone receptor | Q22008813 |
NIH Image to ImageJ: 25 years of image analysis | Q23319322 | ||
Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method | Q25938999 | ||
Endothelial to Mesenchymal Transition (EndoMT) in the Pathogenesis of Human Fibrotic Diseases | Q26748338 | ||
c-Ski acts as a transcriptional co-repressor in transforming growth factor-beta signaling through interaction with smads | Q28138749 | ||
Direct interaction of Ski with either Smad3 or Smad4 is necessary and sufficient for Ski-mediated repression of transforming growth factor-beta signaling | Q28186807 | ||
c-Jun associates with the oncoprotein Ski and suppresses Smad2 transcriptional activity | Q28201147 | ||
Decreased Smad 7 expression contributes to cardiac fibrosis in the infarcted rat heart | Q28581449 | ||
Focal adhesion kinase: the first ten years | Q28609173 | ||
Focal adhesion kinase: in command and control of cell motility | Q29615192 | ||
How matrix metalloproteinases regulate cell behavior | Q29620360 | ||
Matrix metalloproteinase-2 proteolysis of calponin-1 contributes to vascular hypocontractility in endotoxemic rats | Q34243045 | ||
Neutral metalloproteinases produced by human mononuclear phagocytes. Enzyme profile, regulation, and expression during cellular development | Q34263093 | ||
Myocardial matrix metalloproteinase-2: inside out and upside down. | Q35032465 | ||
Cardiac remodeling and physical training post myocardial infarction | Q35075553 | ||
Cardiac fibroblasts: function, regulation of gene expression, and phenotypic modulation | Q35220774 | ||
The transcription factor scleraxis is a critical regulator of cardiac fibroblast phenotype | Q35961854 | ||
Intracellular targets of matrix metalloproteinase-2 in cardiac disease: rationale and therapeutic approaches | Q36665320 | ||
Cellular mechanisms of tissue fibrosis. 2. Contributory pathways leading to myocardial fibrosis: moving beyond collagen expression | Q36699619 | ||
Cardiac Fibrosis: The Fibroblast Awakens | Q36708340 | ||
Transforming Sloan-Kettering viruses generated from the cloned v-ski oncogene by in vitro and in vivo recombinations | Q36859994 | ||
Increased secretion and expression of myostatin in skeletal muscle from extremely obese women. | Q37023094 | ||
MMP-9 signaling in the left ventricle following myocardial infarction | Q37139613 | ||
Targeting matrix metalloproteinases in inflammatory conditions. | Q37631616 | ||
Emerging concepts in cardiac matrix biology | Q37661389 | ||
Potential therapeutic targets for cardiac fibrosis: TGFbeta, angiotensin, endothelin, CCN2, and PDGF, partners in fibroblast activation | Q37764239 | ||
Tissue inhibitor of metalloproteinases (TIMPs) in heart failure | Q37895982 | ||
TGF-β signaling in fibrosis | Q37899212 | ||
Cardiac fibroblasts, fibrosis and extracellular matrix remodeling in heart disease | Q38040163 | ||
Cardiac intercellular communication: are myocytes and fibroblasts fair-weather friends? | Q38046798 | ||
Myofibroblast-mediated mechanisms of pathological remodelling of the heart | Q38064729 | ||
Activation of intracellular matrix metalloproteinase-2 by reactive oxygen-nitrogen species: Consequences and therapeutic strategies in the heart | Q38150577 | ||
DNA methylation in cardiac fibrosis: new advances and perspectives | Q38228803 | ||
MMP-2 is localized to the mitochondria-associated membrane of the heart | Q39038689 | ||
Angiotensin type 2 receptor stimulation ameliorates left ventricular fibrosis and dysfunction via regulation of tissue inhibitor of matrix metalloproteinase 1/matrix metalloproteinase 9 axis and transforming growth factor β1 in the rat heart | Q39282244 | ||
Basic mechanisms in heart failure: the cytokine hypothesis | Q41188647 | ||
Novel extracellular matrix biomarkers as predictors of adverse outcome in chronic heart failure: association between biglycan and response to statin therapy in the CORONA trial | Q41632689 | ||
Antifibrotic properties of c-Ski and its regulation of cardiac myofibroblast phenotype and contractility. | Q42478274 | ||
Airway mesenchymal cell death by mevalonate cascade inhibition: integration of autophagy, unfolded protein response and apoptosis focusing on Bcl2 family proteins | Q44609613 | ||
Fibroblast-specific TGF-β-Smad2/3 signaling underlies cardiac fibrosis | Q47094281 | ||
Transgenic overexpression of macrophage matrix metalloproteinase-9 exacerbates age-related cardiac hypertrophy, vessel rarefaction, inflammation, and fibrosis. | Q47909549 | ||
The ED-A domain enhances the capacity of fibronectin to store latent TGF-β binding protein-1 in the fibroblast matrix. | Q48183721 | ||
Generating favorable growth factor and protease release profiles to enable extracellular matrix accumulation within an in vitro tissue engineering environment. | Q50987512 | ||
Differential impact of mechanical unloading on structural and nonstructural components of the extracellular matrix in advanced human heart failure. | Q51453829 | ||
The Oncoprotein c-Ski Functions as a Direct Antagonist of the Transforming Growth Factor-β Type I Receptor | Q54641166 | ||
The Ski-Zeb2-Meox2 pathway provides a novel mechanism for regulation of the cardiac myofibroblast phenotype. | Q54703943 | ||
Cardiac fibroblast to myofibroblast differentiation in vivo and in vitro: Expression of focal adhesion components in neonatal and adult rat ventricular myofibroblasts | Q58047168 | ||
Regulation of collagen synthesis by inhibitory Smad7 in cardiac myofibroblasts | Q58047195 | ||
Elevation of Expression of Smads 2, 3, and 4, Decorin and TGF-βin the Chronic Phase of Myocardial Infarct Scar Healing | Q58047304 | ||
Antiproliferative and antifibrotic effects of mimosine on adult cardiac fibroblasts1Previously published in abstract form: Circulation 94(8) (1996) I 355.1 | Q58047318 | ||
Assigning matrix metalloproteinase roles in ischaemic cardiac remodelling | Q61420974 | ||
Transcriptional cofactors Ski and SnoN are major regulators of the TGF-β/Smad signaling pathway in health and disease | Q62060038 | ||
The Oncoprotein Ski Acts as an Antagonist of Transforming Growth Factor-β Signaling by Suppressing Smad2 Phosphorylation | Q73356276 | ||
Transcriptional repression by v-Ski and c-Ski mediated by a specific DNA binding site | Q74129025 | ||
Selective spatiotemporal induction of matrix metalloproteinase-2 and matrix metalloproteinase-9 transcription after myocardial infarction | Q83967433 | ||
Matrix metalloproteinase-2 stimulates collagen-I expression through phosphorylation of focal adhesion kinase in rat cardiac fibroblasts | Q84852767 | ||
Differential role of TIMP2 and TIMP3 in cardiac hypertrophy, fibrosis, and diastolic dysfunction | Q87586419 | ||
Regulation of cardiac fibroblast MMP2 gene expression by scleraxis | Q88641624 | ||
P433 | issue | 22 | |
P304 | page(s) | e13897 | |
P577 | publication date | 2018-11-01 | |
P1433 | published in | Physiological Reports | Q15716763 |
P1476 | title | Ski drives an acute increase in MMP-9 gene expression and release in primary cardiac myofibroblasts | |
P478 | volume | 6 |
Q102381776 | Crosstalk between the activated Slit2-Robo1 pathway and TGF-β1 signalling promotes cardiac fibrosis | cites work | P2860 |
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