scholarly article | Q13442814 |
P50 | author | Jamal Hallajzadeh | Q94566734 |
P2093 | author name string | Zatollah Asemi | |
Mohammad Ali Mansournia | |||
Bahman Yousefi | |||
Mansooreh Momen-Heravi | |||
Fatemeh Sadoughi | |||
Parisa Maleki Dana | |||
P2860 | cites work | Testosterone Therapy in Men With Hypogonadism: An Endocrine Society Clinical Practice Guideline. | Q51244776 |
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TMPRSS2 and ADAM17 cleave ACE2 differentially and only proteolysis by TMPRSS2 augments entry driven by the severe acute respiratory syndrome coronavirus spike protein | Q24339582 | ||
The pattern of Middle East respiratory syndrome coronavirus in Saudi Arabia: a descriptive epidemiological analysis of data from the Saudi Ministry of Health | Q28393304 | ||
Efficient activation of the severe acute respiratory syndrome coronavirus spike protein by the transmembrane protease TMPRSS2. | Q30432156 | ||
Structure of SARS coronavirus spike receptor-binding domain complexed with receptor | Q34451350 | ||
Evidence that TMPRSS2 activates the severe acute respiratory syndrome coronavirus spike protein for membrane fusion and reduces viral control by the humoral immune response | Q35076795 | ||
The androgen-regulated protease TMPRSS2 activates a proteolytic cascade involving components of the tumor microenvironment and promotes prostate cancer metastasis. | Q35224818 | ||
Androgen receptor influences on body defense system via modulation of innate and adaptive immune systems: lessons from conditional AR knockout mice | Q36355602 | ||
Modulation of TNF-alpha-converting enzyme by the spike protein of SARS-CoV and ACE2 induces TNF-alpha production and facilitates viral entry | Q36693984 | ||
Sex-Based Differences in Susceptibility to Severe Acute Respiratory Syndrome Coronavirus Infection. | Q38857463 | ||
Angiotensin II induced proteolytic cleavage of myocardial ACE2 is mediated by TACE/ADAM-17: a positive feedback mechanism in the RAS. | Q39044195 | ||
Receptor and viral determinants of SARS-coronavirus adaptation to human ACE2. | Q42844863 | ||
Do men have a higher case fatality rate of severe acute respiratory syndrome than women do? | Q47244472 | ||
Angiotensin-converting enzyme 2 (ACE2) as a SARS-CoV-2 receptor: molecular mechanisms and potential therapeutic target | Q88291889 | ||
SARS-CoV-2 Cell Entry Depends on ACE2 and TMPRSS2 and Is Blocked by a Clinically Proven Protease Inhibitor | Q88292103 | ||
The relationship between circulating testosterone and inflammatory cytokines in men | Q89168700 | ||
Clinical Advances in Sex- and Gender-Informed Medicine to Improve the Health of All: A Review | Q89627718 | ||
Immediate Psychological Responses and Associated Factors during the Initial Stage of the 2019 Coronavirus Disease (COVID-19) Epidemic among the General Population in China | Q90189664 | ||
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The use of anti-inflammatory drugs in the treatment of people with severe coronavirus disease 2019 (COVID-19): The Perspectives of clinical immunologists from China | Q90720693 | ||
Disparities in Age-Specific Morbidity and Mortality from SARS-CoV-2 in China and the Republic of Korea | Q90783903 | ||
Prevalence and predictors of PTSS during COVID-19 outbreak in China hardest-hit areas: Gender differences matter | Q91513013 | ||
SARS-CoV-2 receptor ACE2 and TMPRSS2 are primarily expressed in bronchial transient secretory cells | Q91561671 | ||
TMPRSS2 and COVID-19: Serendipity or opportunity for intervention? | Q91815388 | ||
Sex-Specific SARS-CoV-2 Mortality: Among Hormone-Modulated ACE2 Expression, Risk of Venous Thromboembolism and Hypovitaminosis D | Q93211345 | ||
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Clinical and Epidemiological Characteristics of 1,420 European Patients with mild-to-moderate Coronavirus Disease 2019 | Q94466261 | ||
Testosterone, a key hormone in the context of COVID-19 pandemic | Q94469424 | ||
Radiographic severity index in COVID-19 pneumonia: relationship to age and sex in 783 Italian patients | Q94484526 | ||
Sex and Gender Differences in Health: What the COVID-19 Pandemic Can Teach Us | Q94563776 | ||
Transcriptional landscape of SARS-CoV-2 infection dismantles pathogenic pathways activated by the virus, proposes unique sex-specific differences and predicts tailored therapeutic strategies | Q94588809 | ||
Age and sex differences in soluble ACE2 may give insights for COVID-19 | Q94918222 | ||
Gender Differences in Patients With COVID-19: Focus on Severity and Mortality | Q94926165 | ||
Low testosterone levels predict clinical adverse outcomes in SARS-CoV-2 pneumonia patients | Q95278344 | ||
Prevalence of depression, anxiety, and insomnia among healthcare workers during the COVID-19 pandemic: A systematic review and meta-analysis | Q95298015 | ||
COVID-19 and Individual Genetic Susceptibility/Receptivity: Role of ACE1/ACE2 Genes, Immunity, Inflammation and Coagulation. Might the Double X-chromosome in Females Be Protective against SARS-CoV-2 Compared to the Single X-Chromosome in Males? | Q95307667 | ||
Estrogen regulates the expression of SARS-CoV-2 receptor ACE2 in differentiated airway epithelial cells | Q95311584 | ||
P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
P6216 | copyright status | copyrighted | Q50423863 |
P921 | main subject | COVID-19 | Q84263196 |
P304 | page(s) | 1-4 | |
P577 | publication date | 2020-06-18 | |
P1433 | published in | Prehospital and Disaster Medicine | Q15758348 |
P1476 | title | An Insight into the Sex Differences in COVID-19 Patients: What are the Possible Causes? |
Q101409822 | Age-severity matched cytokine profiling reveals specific signatures in Covid-19 patients |
Q110013316 | SARS-CoV-2 Spike Protein S1-Mediated Endothelial Injury and Pro-Inflammatory State Is Amplified by Dihydrotestosterone and Prevented by Mineralocorticoid Antagonism |
Q99612915 | SARS-CoV-2 and the possible connection to ERs, ACE2, and RAGE: Focus on susceptibility factors |
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