scholarly article | Q13442814 |
review article | Q7318358 |
P50 | author | Otilia V Vieira | Q57564744 |
Cristiano Ramos | Q89156384 | ||
P2093 | author name string | André R A Marques | |
Gisela Machado-Oliveira | |||
P2860 | cites work | Endothelial cell senescence in human atherosclerosis: role of telomere in endothelial dysfunction | Q24292963 |
Telomere shortening triggers senescence of human cells through a pathway involving ATM, p53, and p21(CIP1), but not p16(INK4a) | Q24293520 | ||
Autophagy mediates the mitotic senescence transition | Q24320401 | ||
A biomarker that identifies senescent human cells in culture and in aging skin in vivo | Q24562644 | ||
Atherosclerosis | Q24644573 | ||
Fission and selective fusion govern mitochondrial segregation and elimination by autophagy | Q24652230 | ||
Megabase chromatin domains involved in DNA double-strand breaks in vivo | Q24680284 | ||
Extracellular Vesicles as New Players in Cellular Senescence | Q26738427 | ||
The crucial impact of lysosomes in aging and longevity | Q26749118 | ||
From C-Reactive Protein to Interleukin-6 to Interleukin-1: Moving Upstream To Identify Novel Targets for Atheroprotection | Q26770209 | ||
Extracellular vesicles in cancer: exosomes, microvesicles and the emerging role of large oncosomes | Q26825792 | ||
PGC-1 family coactivators and cell fate: roles in cancer, neurodegeneration, cardiovascular disease and retrograde mitochondria-nucleus signalling | Q26864953 | ||
Signal integration in the endoplasmic reticulum unfolded protein response | Q27860577 | ||
TOR signaling in growth and metabolism | Q27860757 | ||
TOR, a central controller of cell growth | Q27933354 | ||
Tor, a phosphatidylinositol kinase homologue, controls autophagy in yeast | Q27939948 | ||
Naturally occurring p16(Ink4a)-positive cells shorten healthy lifespan | Q28005547 | ||
Potential therapeutic effects of the MTOR inhibitors for preventing ageing and progeria-related disorders | Q28067037 | ||
Cellular and molecular biology of aging endothelial cells | Q28081013 | ||
The hallmarks of aging | Q28131641 | ||
Oxidants, oxidative stress and the biology of ageing | Q28131725 | ||
Formation of elongated giant mitochondria in DFO-induced cellular senescence: involvement of enhanced fusion process through modulation of Fis1 | Q28255413 | ||
mTOR regulates the expression of DNA damage response enzymes in long-lived Snell dwarf, GHRKO, and PAPPA-KO mice | Q28278550 | ||
Formation of MacroH2A-containing senescence-associated heterochromatin foci and senescence driven by ASF1a and HIRA | Q28300188 | ||
Atherosclerosis: basic mechanisms. Oxidation, inflammation, and genetics | Q28300402 | ||
Enhanced glycogenesis is involved in cellular senescence via GSK3/GS modulation | Q28564432 | ||
Senescent intimal foam cells are deleterious at all stages of atherosclerosis | Q28595661 | ||
Aging of mice is associated with p16(Ink4a)- and β-galactosidase-positive macrophage accumulation that can be induced in young mice by senescent cells | Q28972229 | ||
The mammalian unfolded protein response | Q29547400 | ||
Senescent cells, tumor suppression, and organismal aging: good citizens, bad neighbors | Q29614306 | ||
DNA damage foci at dysfunctional telomeres | Q29614811 | ||
The role of autophagy in neurodegenerative disease | Q29614834 | ||
Are cross-bridging structures involved in the bundle formation of intermediate filaments and the decrease in locomotion that accompany cell aging? | Q36213362 | ||
Overexpression of the vimentin gene in transgenic mice inhibits normal lens cell differentiation | Q36221672 | ||
Protein degradation in mitochondria: implications for oxidative stress, aging and disease: a novel etiological classification of mitochondrial proteolytic disorders. | Q36239804 | ||
Arginase-I enhances vascular endothelial inflammation and senescence through eNOS-uncoupling | Q36268285 | ||
Morphologic changes accompanying senescence of cultured human diploid cells | Q36270412 | ||
MTOR regulates the pro-tumorigenic senescence-associated secretory phenotype by promoting IL1A translation | Q36405926 | ||
The coordination of nuclear and mitochondrial communication during aging and calorie restriction | Q36433751 | ||
Endothelial dysfunction: a multifaceted disorder (The Wiggers Award Lecture). | Q36457102 | ||
Meis1 regulates the metabolic phenotype and oxidant defense of hematopoietic stem cells | Q36474576 | ||
Restoration of lysosomal pH in RPE cells from cultured human and ABCA4(-/-) mice: pharmacologic approaches and functional recovery | Q36518020 | ||
Vascular smooth muscle cell senescence in atherosclerosis | Q36529123 | ||
The endoplasmic reticulum: folding, calcium homeostasis, signaling, and redox control | Q36598776 | ||
Endothelial Cell Dysfunction and the Pathobiology of Atherosclerosis | Q36602763 | ||
The Ca2+/Mn2+ ion-pump PMR1 links elevation of cytosolic Ca(2+) levels to α-synuclein toxicity in Parkinson's disease models | Q36603128 | ||
Peroxisomes and oxidative stress | Q36620921 | ||
Vascular calcification: pathobiological mechanisms and clinical implications | Q36649449 | ||
CMV seropositivity and T-cell senescence predict increased cardiovascular mortality in octogenarians: results from the Newcastle 85+ study | Q36665428 | ||
Beyond retrograde and anterograde signalling: mitochondrial-nuclear interactions as a means for evolutionary adaptation and contemporary disease susceptibility | Q36677229 | ||
Vascular cell senescence: contribution to atherosclerosis | Q36699740 | ||
Parkin overexpression during aging reduces proteotoxicity, alters mitochondrial dynamics, and extends lifespan | Q36884177 | ||
p16INK4a deficiency promotes IL-4-induced polarization and inhibits proinflammatory signaling in macrophages. | Q36916423 | ||
Lysosomes and oxidative stress in aging and apoptosis | Q37078535 | ||
ATF6alpha induces XBP1-independent expansion of the endoplasmic reticulum | Q37186859 | ||
Inclusion bodies enriched for p62 and polyubiquitinated proteins in macrophages protect against atherosclerosis | Q37255799 | ||
A role for mitochondrial oxidants in stress-induced premature senescence of human vascular smooth muscle cells | Q37272651 | ||
Autophagy and aging: keeping that old broom working | Q37321185 | ||
Evaluating Health Span in Preclinical Models of Aging and Disease: Guidelines, Challenges, and Opportunities for Geroscience | Q37322066 | ||
Calorie restriction protects against experimental abdominal aortic aneurysms in mice. | Q37346647 | ||
Senescence-associated exosome release from human prostate cancer cells. | Q37352797 | ||
Stress-induced premature senescence of endothelial cells: a perilous state between recovery and point of no return | Q37422975 | ||
A comparative analysis of the cell biology of senescence and aging | Q37473186 | ||
Downregulation of peroxiredoxin-3 by hydrophobic bile acid induces mitochondrial dysfunction and cellular senescence in human trophoblasts | Q37498885 | ||
Dietary rapamycin supplementation reverses age-related vascular dysfunction and oxidative stress, while modulating nutrient-sensing, cell cycle, and senescence pathways | Q37592020 | ||
Nicotinamide enhances mitochondria quality through autophagy activation in human cells. | Q45994645 | ||
Sequence-specific DNA cleavage by Fe2+-mediated fenton reactions has possible biological implications | Q46085269 | ||
Cytoplasmic chromatin triggers inflammation in senescence and cancer | Q46165767 | ||
A lysosomal switch triggers proteostasis renewal in the immortal C. elegans germ lineage | Q46255501 | ||
Mechanisms driving the ageing heart | Q46279016 | ||
Lipid mediators of autophagy in stress-induced premature senescence of endothelial cells | Q46802157 | ||
Quantifying dense bodies and lipofuscin during aging: a morphologist's perspective | Q47218484 | ||
Lysosomal lipid accumulation from oxidized low density lipoprotein is correlated with hypertrophy of the Golgi apparatus and trans-Golgi network. | Q47764891 | ||
Lysosomal sequestration of free and esterified cholesterol from oxidized low density lipoprotein in macrophages of different species. | Q47764904 | ||
Inflammageing and metaflammation: The yin and yang of type 2 diabetes. | Q47870904 | ||
Ionizing radiation induces long-term senescence in endothelial cells through mitochondrial respiratory complex II dysfunction and superoxide generation. | Q47913083 | ||
The ATF6α arm of the Unfolded Protein Response mediates replicative senescence in human fibroblasts through a COX2/prostaglandin E2 intracrine pathway. | Q47958620 | ||
Interaction between mTOR pathway inhibition and autophagy induction attenuates adriamycin-induced vascular smooth muscle cell senescence through decreased expressions of p53/p21/p16. | Q47967166 | ||
Senescence and aging: Causes, consequences, and therapeutic avenues | Q48347290 | ||
Identification and characterization of an increased glycoprotein in aging: age-associated translocation of cathepsin D. | Q48445810 | ||
Glycogen accumulation of the aging human brain | Q48485172 | ||
Smooth Muscle Cell Fate and Plasticity in Atherosclerosis | Q49551883 | ||
Short Leukocyte Telomere Length Precedes Clinical Expression of Atherosclerosis: Blood-and-Muscle Model | Q49837902 | ||
Targeting HSP90 Ameliorates Nephropathy and Atherosclerosis Through Suppression of NF-κB and STAT Signaling Pathways in Diabetic Mice | Q50580624 | ||
Lysosomal enzyme activities in parenchymal and nonparenchymal liver cells isolated from young, adult and old rats. | Q50958351 | ||
Transdifferentiation of vascular smooth muscle cells to macrophage-like cells during atherogenesis. | Q51065835 | ||
Age-Associated Sirtuin 1 Reduction in Vascular Smooth Muscle Links Vascular Senescence and Inflammation to Abdominal Aortic Aneurysm. | Q51482469 | ||
The PI3K/Akt/mTOR pathway regulates the replicative senescence of human VSMCs. | Q51516524 | ||
Vascular Smooth Muscle-Specific Progerin Expression Accelerates Atherosclerosis and Death in a Mouse Model of Hutchinson-Gilford Progeria Syndrome. | Q51766620 | ||
Hallmarks of Cellular Senescence. | Q51767478 | ||
Leukocyte telomere length is associated with high-risk plaques on virtual histology intravascular ultrasound and increased proinflammatory activity. | Q52612656 | ||
Vascular smooth muscle cell death, autophagy and senescence in atherosclerosis. | Q52735227 | ||
Ultrastructural changes accompanying the aging of human diploid cells in culture | Q52992355 | ||
ER Stress Response in Human Cellular Models of Senescence. | Q53025676 | ||
Atorvastatin induces T cell proliferation by a telomerase reverse transcriptase (TERT) mediated mechanism. | Q53027311 | ||
Disturbed flow promotes endothelial senescence via a p53-dependent pathway. | Q53061350 | ||
Long noncoding RNA H19 mediates melatonin inhibition of premature senescence of c-kit(+) cardiac progenitor cells by promoting miR-675. | Q53125918 | ||
Quantitative assessment of markers for cell senescence. | Q53345092 | ||
Anti-oncogenic role of the endoplasmic reticulum differentially activated by mutations in the MAPK pathway. | Q53600237 | ||
Vascular smooth muscle cells undergo telomere-based senescence in human atherosclerosis: effects of telomerase and oxidative stress. | Q53616098 | ||
Golgin-84-associated Golgi fragmentation triggers tau hyperphosphorylation by activation of cyclin-dependent kinase-5 and extracellular signal-regulated kinase. | Q54388404 | ||
A systems approach for decoding mitochondrial retrograde signaling pathways. | Q54447301 | ||
Protein Carbamylation: A Marker Reflecting Increased Age-Related Cell Oxidation. | Q55128365 | ||
Anti-senescence compounds: A potential nutraceutical approach to healthy aging | Q56335111 | ||
Significant Role for p16INK4a in p53-Independent Telomere-Directed Senescence | Q56969662 | ||
Aging, Cell Senescence, and Chronic Disease: Emerging Therapeutic Strategies | Q57047926 | ||
Animal models of atherosclerosis | Q57761322 | ||
Cardiopoietic Stem Cell Therapy in Heart Failure | Q57779306 | ||
Lysosomal oxidation of LDL alters lysosomal pH, induces senescence and increases secretion of pro-inflammatory cytokines in human macrophages | Q58610946 | ||
Vascular Smooth Muscle Cell Senescence Promotes Atherosclerosis and Features of Plaque Vulnerability | Q63915322 | ||
Length-independent telomere damage drives post-mitotic cardiomyocyte senescence | Q64233922 | ||
Metabolic features and regulation in cell senescence | Q64272024 | ||
Extension of human cell lifespan by nicotinamide phosphoribosyltransferase | Q64377238 | ||
Peroxisomes: a nexus for lipid metabolism and cellular signaling. | Q37633445 | ||
Cellular senescence mediates fibrotic pulmonary disease | Q37672706 | ||
Rapamycin: one drug, many effects | Q37679469 | ||
ATF6α regulates morphological changes associated with senescence in human fibroblasts | Q37708694 | ||
Pathophysiology of vascular calcification: Pivotal role of cellular senescence in vascular smooth muscle cells | Q37774469 | ||
Architecture of the mammalian Golgi | Q37866547 | ||
Mitochondria and autophagy: critical interplay between the two homeostats | Q37918611 | ||
Emerging role of NF-κB signaling in the induction of senescence-associated secretory phenotype (SASP). | Q37969844 | ||
Fusion and fission: interlinked processes critical for mitochondrial health | Q38039146 | ||
The many routes of Golgi-dependent trafficking | Q38121022 | ||
Cellular senescence and its effector programs | Q38180648 | ||
Mechanisms of plaque formation and rupture. | Q38218000 | ||
The unfolded protein response and cellular senescence. A review in the theme: cellular mechanisms of endoplasmic reticulum stress signaling in health and disease | Q38300472 | ||
GSK3 inactivation is involved in mitochondrial complex IV defect in transforming growth factor (TGF) β1-induced senescence | Q38324818 | ||
Pioglitazone activates aortic telomerase and prevents stress-induced endothelial apoptosis | Q38336771 | ||
Longevity Pathways (mTOR, SIRT, Insulin/IGF-1) as Key Modulatory Targets on Aging and Neurodegeneration | Q38522675 | ||
DNA Damage and Repair in Vascular Disease | Q38600660 | ||
Exenatide mitigated diet-induced vascular aging and atherosclerotic plaque growth in ApoE-deficient mice under chronic stress | Q38653357 | ||
Galantamine alleviates senescence of U87 cells induced by beta-amyloid through decreasing ROS production | Q38701915 | ||
Translating the Science of Aging into Therapeutic Interventions | Q38756902 | ||
p16(Ink4a)-induced senescence of pancreatic beta cells enhances insulin secretion. | Q38788371 | ||
Impaired ATP6V0A2 expression contributes to Golgi dispersion and glycosylation changes in senescent cells | Q38816331 | ||
Chemical screening identifies ATM as a target for alleviating senescence | Q38873970 | ||
Reversal of phenotypes of cellular senescence by pan-mTOR inhibition. | Q38964715 | ||
PKCδ phosphorylation is an upstream event of GSK3 inactivation-mediated ROS generation in TGF-β1-induced senescence | Q38985955 | ||
Mitochondria are targets for peroxisome-derived oxidative stress in cultured mammalian cells | Q39103215 | ||
Prelamin A accumulation in endothelial cells induces premature senescence and functional impairment | Q39132672 | ||
Robust multiparametric assessment of cellular senescence | Q39214864 | ||
Cellular Senescence: A Translational Perspective | Q39246705 | ||
The Clinical Potential of Senolytic Drugs | Q39312260 | ||
Vascular ageing: Underlying mechanisms and clinical implications. | Q39380015 | ||
Studies on giant mitochondria | Q39767169 | ||
Innate immune sensing of cytosolic chromatin fragments through cGAS promotes senescence. | Q40036388 | ||
Restoration of peroxisomal catalase import in a model of human cellular aging | Q40084094 | ||
Techniques to Induce and Quantify Cellular Senescence | Q40197071 | ||
TGF beta1 induces prolonged mitochondrial ROS generation through decreased complex IV activity with senescent arrest in Mv1Lu cells. | Q40462552 | ||
Cellular aging of mitochondrial DNA-depleted cells | Q40489808 | ||
Senescing human cells and ageing mice accumulate DNA lesions with unrepairable double-strand breaks | Q40593721 | ||
Mitogen-activated protein kinase p38 defines the common senescence-signalling pathway | Q40671353 | ||
Iron chelation-induced senescence-like growth arrest in hepatocyte cell lines: association of transforming growth factor beta1 (TGF-beta1)-mediated p27Kip1 expression. | Q40738994 | ||
The unfolded protein response: controlling cell fate decisions under ER stress and beyond | Q29615499 | ||
Association between telomere length in blood and mortality in people aged 60 years or older | Q29615532 | ||
Senescence-associated secretory phenotypes reveal cell-nonautonomous functions of oncogenic RAS and the p53 tumor suppressor | Q29615559 | ||
Cellular senescence: when bad things happen to good cells | Q29615561 | ||
A DNA damage checkpoint response in telomere-initiated senescence | Q29615919 | ||
Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas | Q29616243 | ||
Beyond cholesterol. Modifications of low-density lipoprotein that increase its atherogenicity | Q29617216 | ||
The senescence-associated secretory phenotype: the dark side of tumor suppression | Q29620106 | ||
Rb-mediated heterochromatin formation and silencing of E2F target genes during cellular senescence | Q29620428 | ||
Lysosome-mediated processing of chromatin in senescence. | Q30541249 | ||
Higher-order unfolding of satellite heterochromatin is a consistent and early event in cell senescence | Q30560506 | ||
Mitochondrial dysfunction accounts for the stochastic heterogeneity in telomere-dependent senescence | Q33283342 | ||
Atherosclerosis profile and incidence of cardiovascular events: a population-based survey | Q33503404 | ||
Microvesicles from the plasma of elderly subjects and from senescent endothelial cells promote vascular calcification | Q33563602 | ||
Linking ER Stress to Autophagy: Potential Implications for Cancer Therapy. | Q33633401 | ||
Rapamycin extends life and health in C57BL/6 mice | Q33684583 | ||
Inflammation in atherosclerosis: from pathophysiology to practice | Q33709505 | ||
Senescent cells: a novel therapeutic target for aging and age-related diseases | Q33732761 | ||
Cellular senescence drives age-dependent hepatic steatosis. | Q33810220 | ||
Leucocyte telomere length and risk of cardiovascular disease: systematic review and meta-analysis | Q33860488 | ||
Persistent mTORC1 signaling in cell senescence results from defects in amino acid and growth factor sensing. | Q33870045 | ||
Moderate Autophagy Inhibits Vascular Smooth Muscle Cell Senescence to Stabilize Progressed Atherosclerotic Plaque via the mTORC1/ULK1/ATG13 Signal Pathway. | Q33872781 | ||
Inflammatory networks during cellular senescence: causes and consequences | Q33889156 | ||
Mitochondria, oxygen free radicals, disease and ageing | Q33922972 | ||
NAD+ and sirtuins in aging and disease | Q33955066 | ||
Senescence-associated beta-galactosidase is lysosomal beta-galactosidase | Q33995193 | ||
The role of senescent cells in ageing | Q34041036 | ||
Cellular senescence: from physiology to pathology | Q34041305 | ||
The Achilles' heel of senescent cells: from transcriptome to senolytic drugs. | Q34043405 | ||
Mitochondria are required for pro-ageing features of the senescent phenotype. | Q34046100 | ||
Chronic senolytic treatment alleviates established vasomotor dysfunction in aged or atherosclerotic mice. | Q34046144 | ||
Sterol regulatory element-binding protein (SREBP)-1-mediated lipogenesis is involved in cell senescence | Q34121467 | ||
Aging, cellular senescence, and cancer | Q34199776 | ||
Rapamycin slows aging in mice | Q34275079 | ||
Aging and atherosclerosis: mechanisms, functional consequences, and potential therapeutics for cellular senescence | Q34286746 | ||
The target of rapamycin (TOR) proteins | Q34287448 | ||
Integrating the DNA damage and protein stress responses during cancer development and treatment. | Q64883630 | ||
Aged-senescent cells contribute to impaired heart regeneration. | Q64986339 | ||
The participation of lysosomes in the transformation of smooth muscle cells to foamy cells in the aorta of cholesterol-fed rabbits | Q69147783 | ||
Mitochondrial alterations in aged rat adrenal cortical cells | Q69799789 | ||
Lysosomal activity in the aging rat liver: II. Morphometry of acid phosphatase positive dense bodies | Q70625698 | ||
Increased organization of cytoskeleton accompanying the aging of human fibroblasts in vitro | Q70795796 | ||
Glycogen accumulation in tibial nerves of experimentally diabetic and aging control rats | Q71008147 | ||
Electron microscopy of human fibroblasts in tissue culture during logarithmic and confluent stages of growth | Q71543506 | ||
Effects of the type of dietary fat at two levels of vitamin E in Wistar male rats during development and aging. IV. Biochemical and morphometric parameters of the heart | Q72660398 | ||
Hereditary catalase deficiencies and increased risk of diabetes | Q73299266 | ||
Differential expression of thymosin beta-10 by early passage and senescent vascular endothelium is modulated by VPF/VEGF: evidence for senescent endothelial cells in vivo at sites of atherosclerosis | Q73395517 | ||
Evaluation of the activities of eight lysosomal hydrolases in sera of humans, rats and pigs of different ages | Q74246113 | ||
Cooperative interactions between RB and p53 regulate cell proliferation, cell senescence, and apoptosis in human vascular smooth muscle cells from atherosclerotic plaques | Q74452665 | ||
T-cell-mediated lysis of endothelial cells in acute coronary syndromes | Q77595337 | ||
Enhanced apoptosis in prolonged cultures of senescent porcine pulmonary artery endothelial cells | Q77648336 | ||
Aging- and smoking-associated alteration in the relative content of mitochondrial DNA in human lung | Q77779562 | ||
Age-related changes in focal adhesions lead to altered cell behavior in tendon fibroblasts | Q79742016 | ||
Senescence-associated alterations of cytoskeleton: extraordinary production of vimentin that anchors cytoplasmic p53 in senescent human fibroblasts | Q81462678 | ||
Measurement of telomere length by the Southern blot analysis of terminal restriction fragment lengths | Q82491708 | ||
Polymorphic variations in manganese superoxide dismutase (MnSOD), glutathione peroxidase-1 (GPX1), and catalase (CAT) contribute to elevated plasma triglyceride levels in Chinese patients with type 2 diabetes or diabetic cardiovascular disease | Q82945501 | ||
Blockade of the nuclear factor-κB pathway in the endothelium prevents insulin resistance and prolongs life spans | Q83389474 | ||
Senescent cells: a therapeutic target for cardiovascular disease | Q88243444 | ||
Cellular Senescence in Postmitotic Cells: Beyond Growth Arrest | Q88502149 | ||
Mitochondria-to-nucleus retrograde signaling drives formation of cytoplasmic chromatin and inflammation in senescence | Q89453242 | ||
Senolytics improve physical function and increase lifespan in old age | Q89486364 | ||
Monoamine oxidase-A is a novel driver of stress-induced premature senescence through inhibition of parkin-mediated mitophagy | Q90021806 | ||
Identification and characterization of Cardiac Glycosides as senolytic compounds | Q90857560 | ||
ER Stress Activates the NLRP3 Inflammasome: A Novel Mechanism of Atherosclerosis | Q91112980 | ||
Replicative senescence of human dermal fibroblasts affects structural and functional aspects of the Golgi apparatus | Q91119708 | ||
Forkhead box O3 protects the heart against paraquat-induced aging-associated phenotypes by upregulating the expression of antioxidant enzymes | Q91540700 | ||
Cardiac glycosides are broad-spectrum senolytics | Q91677826 | ||
Pharmacological inhibition of mTOR attenuates replicative cell senescence and improves cellular function via regulating the STAT3-PIM1 axis in human cardiac progenitor cells | Q91787891 | ||
Inhibition of Senescence-Associated Genes Rb1 and Meis2 in Adult Cardiomyocytes Results in Cell Cycle Reentry and Cardiac Repair Post-Myocardial Infarction | Q91963816 | ||
Senescence-induced inflammation: an important player and key therapeutic target in atherosclerosis | Q92394184 | ||
Pharmacological clearance of senescent cells improves survival and recovery in aged mice following acute myocardial infarction | Q92669344 | ||
Mitochondrial dysfunction and cell senescence: deciphering a complex relationship | Q92847042 | ||
Vascular smooth muscle cells in atherosclerosis | Q93053742 | ||
Allogeneic mesenchymal stem cell transplantation in severe and refractory systemic lupus erythematosus: 4 years of experience | Q95505284 | ||
Oxidative damage, mitochondrial oxidant generation and antioxidant defenses during aging and in response to food restriction in the mouse | Q34323890 | ||
Autophagy and ageing: implications for age-related neurodegenerative diseases. | Q34373575 | ||
Chaperone-mediated autophagy: roles in disease and aging | Q34388237 | ||
Astrocyte senescence as a component of Alzheimer's disease | Q34416365 | ||
Peroxisome senescence in human fibroblasts | Q34416985 | ||
From cell senescence to age-related diseases: differential mechanisms of action of senescence-associated secretory phenotypes | Q34482996 | ||
The human amniotic fluid stem cell secretome effectively counteracts doxorubicin-induced cardiotoxicity. | Q34534895 | ||
Mitochondrial alterations, cellular response to oxidative stress and defective degradation of proteins in aging. | Q34546103 | ||
Autophagy and human disease: emerging themes | Q34620377 | ||
Resveratrol prevents high fat/sucrose diet-induced central arterial wall inflammation and stiffening in nonhuman primates. | Q34624641 | ||
Mechanisms of cardiovascular disease in accelerated aging syndromes | Q34646841 | ||
Cellular senescence: putting the paradoxes in perspective | Q34777425 | ||
Peroxisome Metabolism and Cellular Aging | Q34796539 | ||
Telomere length: a review of methods for measurement. | Q34938123 | ||
Lysosome biogenesis and lysosomal membrane proteins: trafficking meets function | Q34996827 | ||
Mathematical modeling of the role of mitochondrial fusion and fission in mitochondrial DNA maintenance | Q35022679 | ||
Risk of developing multimorbidity across all ages in an historical cohort study: differences by sex and ethnicity | Q35065283 | ||
Premature aging induced by radiation exhibits pro-atherosclerotic effects mediated by epigenetic activation of CD44 expression | Q35093041 | ||
New concepts in nutraceuticals as alternative for pharmaceuticals | Q35109316 | ||
Intracellular sorting and transport of proteins | Q35131675 | ||
Control of the senescence-associated secretory phenotype by NF-κB promotes senescence and enhances chemosensitivity | Q35493953 | ||
Reversal of human cellular senescence: roles of the p53 and p16 pathways | Q35561999 | ||
Mitochondrial signaling: the retrograde response | Q35739853 | ||
Human papillomavirus E7 repression in cervical carcinoma cells initiates a transcriptional cascade driven by the retinoblastoma family, resulting in senescence | Q35784727 | ||
Oncogene-induced senescence results in marked metabolic and bioenergetic alterations | Q35954897 | ||
The yeast retrograde response as a model of intracellular signaling of mitochondrial dysfunction | Q35966892 | ||
Lamin B1 loss is a senescence-associated biomarker. | Q35998585 | ||
Senescence and cytoskeleton: overproduction of vimentin induces senescent-like morphology in human fibroblasts | Q40769499 | ||
Senescent Vascular Smooth Muscle Cells Drive Inflammation Through an Interleukin-1α-Dependent Senescence-Associated Secretory Phenotype | Q40771854 | ||
Correlation between senescence-associated beta-galactosidase expression in articular cartilage and disease severity of patients with knee osteoarthritis | Q40795677 | ||
Site-specific DNA damage at the GGG sequence by UVA involves acceleration of telomere shortening | Q40813942 | ||
Quantitative and integrated proteome and microRNA analysis of endothelial replicative senescence | Q40899938 | ||
Retrograde Ca2+ signaling in C2C12 skeletal myocytes in response to mitochondrial genetic and metabolic stress: a novel mode of inter-organelle crosstalk | Q40976490 | ||
Quantitative identification of senescent cells in aging and disease. | Q40988773 | ||
Crosstalk between nuclear and mitochondrial genomes | Q41114828 | ||
Identification of HSP90 inhibitors as a novel class of senolytics. | Q41606906 | ||
Microvesicles Derived from Indoxyl Sulfate Treated Endothelial Cells Induce Endothelial Progenitor Cells Dysfunction. | Q41688602 | ||
Senescence-associated (beta)-galactosidase reflects an increase in lysosomal mass during replicative ageing of human endothelial cells | Q41921020 | ||
NF-κB inhibition delays DNA damage-induced senescence and aging in mice | Q42220488 | ||
Uncoupling Oncogene-Induced Senescence (OIS) and DNA Damage Response (DDR) triggered by DNA hyper-replication: lessons from primary mouse embryo astrocytes (MEA). | Q42374837 | ||
Endothelial cell senescence is associated with disrupted cell-cell junctions and increased monolayer permeability | Q42423656 | ||
Chronic endoplasmic reticulum stress activates unfolded protein response in arterial endothelium in regions of susceptibility to atherosclerosis. | Q42559927 | ||
Activation of SIRT1 Attenuates Klotho Deficiency-Induced Arterial Stiffness and Hypertension by Enhancing AMP-Activated Protein Kinase Activity | Q42702475 | ||
Alteration of Golgi structure in senescent cells and its regulation by a G protein γ subunit. | Q42725466 | ||
Coordinate regulation of phospholipid biosynthesis and secretory pathway gene expression in XBP-1(S)-induced endoplasmic reticulum biogenesis. | Q42832143 | ||
Replicative senescence of vascular smooth muscle cells enhances the calcification through initiating the osteoblastic transition | Q43277438 | ||
Increases of mitochondrial mass and mitochondrial genome in association with enhanced oxidative stress in human cells harboring 4,977 BP-deleted mitochondrial DNA. | Q43857312 | ||
Lipid accumulation in smooth muscle cells under LDL loading is independent of LDL receptor pathway and enhanced by hypoxic conditions. | Q44177947 | ||
Protein profiling of the human epidermis from the elderly reveals up-regulation of a signature of interferon-gamma-induced polypeptides that includes manganese-superoxide dismutase and the p85beta subunit of phosphatidylinositol 3-kinase | Q44367191 | ||
Mitochondrial recycling and aging of cardiac myocytes: the role of autophagocytosis | Q44547129 | ||
Complex II Defect via Down-regulation of Iron-Sulfur Subunit Induces Mitochondrial Dysfunction and Cell Cycle Delay in Iron Chelation-induced Senescence-associated Growth Arrest | Q44598323 | ||
Evolutionary biology: essence of mitochondria. | Q44653693 | ||
Retardation of atherosclerosis by overexpression of catalase or both Cu/Zn-superoxide dismutase and catalase in mice lacking apolipoprotein E. | Q45140733 | ||
Cytokeratin-related loss of cellular integrity is not a major driving force of human intrinsic skin aging | Q45260861 | ||
P275 | copyright license | Creative Commons Attribution | Q6905323 |
P433 | issue | 10 | |
P921 | main subject | cellular senescence | Q9075999 |
P577 | publication date | 2020-09-23 | |
P1433 | published in | Cells | Q27724621 |
P1476 | title | Cell Senescence, Multiple Organelle Dysfunction and Atherosclerosis | |
P478 | volume | 9 |
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