How RSV Proteins Join Forces to Overcome the Host Innate Immune Response

scientific article published on 17 February 2022

How RSV Proteins Join Forces to Overcome the Host Innate Immune Response is …
instance of (P31):
scholarly articleQ13442814
review articleQ7318358

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P356DOI10.3390/V14020419
P932PMC publication ID8874859
P698PubMed publication ID35216012

P2093author name stringXavier Saelens
Bert Schepens
Iebe Rossey
Koen Sedeyn
Tessa Van Royen
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Respiratory Syncytial Virus Uses CX3CR1 as a Receptor on Primary Human Airway Epithelial CulturesQ28551477
MAVS and MyD88 are essential for innate immunity but not cytotoxic T lymphocyte response against respiratory syncytial virusQ28593715
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Antiviral innate immunity pathways.Q36394971
Recombinant respiratory syncytial virus from which the entire SH gene has been deleted grows efficiently in cell culture and exhibits site-specific attenuation in the respiratory tract of the mouseQ36548529
Central role of the respiratory syncytial virus matrix protein in infection.Q36566590
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Respiratory syncytial virus activates innate immunity through Toll-like receptor 2.Q37051620
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The respiratory syncytial virus matrix protein possesses a Crm1-mediated nuclear export mechanismQ37191929
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PKR-dependent inflammatory signalsQ38054995
Toll-like receptor signaling pathwaysQ38259564
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P275copyright licenseCreative Commons Attribution 4.0 InternationalQ20007257
P6216copyright statuscopyrightedQ50423863
P433issue2
P407language of work or nameEnglishQ1860
P921main subjectmodulation by virus of host processQ14818042
respiratory syncytial virusQ1052913
P304page(s)419
P577publication date2022-02-17
P1433published inVirusesQ7935305
P1476titleHow RSV Proteins Join Forces to Overcome the Host Innate Immune Response
P478volume14

Reverse relations

Q116170570Respiratory Syncytial Virus NS1 Protein Targets the Transactivator Binding Domain of MED25cites workP2860

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