| scholarly article | Q13442814 |
| P50 | author | Pascal Schneider | Q40058302 |
| Jeffrey Miller | Q42613275 | ||
| P2093 | author name string | Richard A Flavell | |
| Hideo Yagita | |||
| Norm Allaire | |||
| Jeffrey A Winkles | |||
| Jennifer S Michaelson | |||
| Linda C Burkly | |||
| Timothy S Zheng | |||
| Monica Wang | |||
| Sharron A N Brown | |||
| Beth Browning | |||
| Martin L Scott | |||
| Shawn Weng | |||
| Mahasweta Girgenrath | |||
| Christine A Kostek | |||
| Yen-ming Hsu | |||
| P2860 | cites work | A novel TNF receptor family member binds TWEAK and is implicated in angiogenesis | Q24291951 |
| The Fn14 immediate-early response gene is induced during liver regeneration and highly expressed in both human and murine hepatocellular carcinomas | Q24681487 | ||
| Expression profiling of cytokines and related genes in regenerating skeletal muscle after cardiotoxin injection: a role for osteopontin | Q24685614 | ||
| Immortalization of mouse myogenic cells can occur without loss of p16INK4a, p19ARF, or p53 and is accelerated by inactivation of Bax | Q24791108 | ||
| Diseased muscles that lack dystrophin or laminin-alpha2 have altered compositions and proliferation of mononuclear cell populations | Q24791491 | ||
| Proinflammatory activity of TWEAK on human dermal fibroblasts and synoviocytes: blocking and enhancing effects of anti-TWEAK monoclonal antibodies | Q24803611 | ||
| TWEAK induces angiogenesis and proliferation of endothelial cells | Q28139175 | ||
| Dual role for TWEAK in angiogenic regulation | Q28200688 | ||
| The TNF and TNF receptor superfamilies: integrating mammalian biology | Q28203717 | ||
| Muscle satellite cells | Q28205273 | ||
| Fibroblast growth factor-inducible 14 mediates multiple pathways of TWEAK-induced cell death | Q28218718 | ||
| The role of tumor necrosis factor-alpha (TNF-alpha) in skeletal muscle regeneration. Studies in TNF-alpha(-/-) and TNF-alpha(-/-)/LT-alpha(-/-) mice | Q74216064 | ||
| TWEAK stimulation of astrocytes and the proinflammatory consequences | Q74280324 | ||
| Tweaking liver progenitor cells | Q81323126 | ||
| TWEAK, a new secreted ligand in the tumor necrosis factor family that weakly induces apoptosis | Q28257134 | ||
| MyoD or Myf-5 is required for the formation of skeletal muscle | Q28510042 | ||
| Regeneration of transgenic skeletal muscles with altered timing of expression of the basic helix-loop-helix muscle regulatory factor MRF4 | Q28573663 | ||
| Pro- and anti-apoptotic members of the Bcl-2 family in skeletal muscle: a distinct role for Bcl-2 in later stages of myogenesis | Q28591162 | ||
| Inflammatory Blockade Restores Adult Hippocampal Neurogenesis | Q29619037 | ||
| Inflammation is detrimental for neurogenesis in adult brain | Q29619040 | ||
| Points of control in inflammation | Q29620319 | ||
| TWEAK induces liver progenitor cell proliferation | Q33919951 | ||
| NF-kappaB controls cell growth and differentiation through transcriptional regulation of cyclin D1. | Q33959017 | ||
| Myogenic satellite cells: physiology to molecular biology | Q34084128 | ||
| Interactions of tumor necrosis factor (TNF) and TNF receptor family members in the mouse and human | Q34503355 | ||
| The inflammatory macrophage: a story of Jekyll and Hyde | Q35041589 | ||
| Biology and clinical applications of mesenchymal stem cells | Q35606415 | ||
| Myodegeneration in EDA-A2 transgenic mice is prevented by XEDAR deficiency | Q36233106 | ||
| Bcl-2 expression identifies an early stage of myogenesis and promotes clonal expansion of muscle cells | Q36256254 | ||
| Satellite cells attract monocytes and use macrophages as a support to escape apoptosis and enhance muscle growth. | Q36324730 | ||
| Macrophages contribute to the maintenance of stable regenerating neurites following peripheral nerve injury | Q37471979 | ||
| Modes of FGF release in vivo and in vitro | Q38037841 | ||
| Highly coordinated gene regulation in mouse skeletal muscle regeneration | Q38359867 | ||
| Tumor necrosis factor-like weak inducer of apoptosis inhibits skeletal myogenesis through sustained activation of nuclear factor-kappaB and degradation of MyoD protein. | Q40320063 | ||
| Tumor necrosis factor inhibits human myogenesis in vitro | Q40649958 | ||
| The Fn14 cytoplasmic tail binds tumour-necrosis-factor-receptor-associated factors 1, 2, 3 and 5 and mediates nuclear factor-kappaB activation | Q40677513 | ||
| Macrophages enhance muscle satellite cell proliferation and delay their differentiation | Q40809908 | ||
| Blood borne macrophages are essential for the triggering of muscle regeneration following muscle transplant | Q40815680 | ||
| NF-kappaB-induced loss of MyoD messenger RNA: possible role in muscle decay and cachexia | Q40852201 | ||
| TNF inhibits myogenesis and downregulates the expression of myogenic regulatory factors myoD and myogenin | Q41070673 | ||
| Cell cycle exit upon myogenic differentiation | Q41655693 | ||
| Regeneration of muscles after cardiotoxin injury I. Cytological aspects | Q42519124 | ||
| Stimulation of mesothelial cell proliferation by exudate macrophages enhances serosal wound healing in a murine model | Q42855960 | ||
| Macrophage depletion impairs oligodendrocyte remyelination following lysolecithin-induced demyelination | Q43700744 | ||
| Fiber regeneration is not persistent in dystrophic (MDX) mouse skeletal muscle | Q44596500 | ||
| Functional Genomic Analysis of Remyelination Reveals Importance of Inflammation in Oligodendrocyte Regeneration | Q47415104 | ||
| TNF alpha promotes proliferation of oligodendrocyte progenitors and remyelination | Q48744723 | ||
| Mechanical loading and injury induce human myotubes to release neutrophil chemoattractants. | Q51580116 | ||
| Flow cytometric identification of murine neutrophils and monocytes. | Q52199383 | ||
| Multifaceted roles of TNF-? in myoblast destruction: A multitude of signal transduction pathways | Q60275504 | ||
| P433 | issue | 24 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | mesenchymal progenitor cell | Q106510074 |
| P304 | page(s) | 5826-5839 | |
| P577 | publication date | 2006-11-23 | |
| P1433 | published in | The EMBO Journal | Q1278554 |
| P1476 | title | TWEAK, via its receptor Fn14, is a novel regulator of mesenchymal progenitor cells and skeletal muscle regeneration | |
| P478 | volume | 25 |
| Q50871994 | 5-Azacytidine-mediated hMSC behavior on electrospun scaffolds for skeletal muscle regeneration. |
| Q28729829 | A Bioinformatics Resource for TWEAK-Fn14 Signaling Pathway |
| Q38895292 | A Paracrine Mechanism Accelerating Expansion of Human Induced Pluripotent Stem Cell-Derived Hepatic Progenitor-Like Cells |
| Q38123074 | A further TWEAK to multiple sclerosis pathophysiology |
| Q92506834 | A major role of TWEAK/Fn14 axis as a therapeutic target for post-angioplasty restenosis |
| Q30496163 | A novel role for tumor necrosis factor-like weak inducer of apoptosis (TWEAK) in the development of cardiac dysfunction and failure |
| Q34554113 | Activation of stem cells in hepatic diseases. |
| Q24336188 | Adipocytes as immune cells: differential expression of TWEAK, BAFF, and APRIL and their receptors (Fn14, BAFF-R, TACI, and BCMA) at different stages of normal and pathological adipose tissue development |
| Q35954775 | Blocking TWEAK-Fn14 interaction inhibits hematopoietic stem cell transplantation-induced intestinal cell death and reduces GVHD. |
| Q24296044 | C3orf58, a novel paracrine protein, stimulates cardiomyocyte cell-cycle progression through the PI3K-AKT-CDK7 pathway |
| Q37031812 | CD163 interacts with TWEAK to regulate tissue regeneration after ischaemic injury |
| Q42768605 | Cell differentiation modeled via a coupled two-switch regulatory network |
| Q42006323 | Characterization of primary human skeletal muscle cells from multiple commercial sources |
| Q42776466 | Connective Tissue Growth Factor Promotes Efficient Generation of Human Induced Pluripotent Stem Cell-Derived Choroidal Endothelium |
| Q27678102 | Crystal Structure of Human TWEAK in Complex with the Fab Fragment of a Neutralizing Antibody Reveals Insights into Receptor Binding |
| Q33931130 | DNA methyltransferase 3a and mitogen-activated protein kinase signaling regulate the expression of fibroblast growth factor-inducible 14 (Fn14) during denervation-induced skeletal muscle atrophy |
| Q35556794 | Development of an Fn14 agonistic antibody as an anti-tumor agent |
| Q85167906 | Direct targeting of fibroblast growth factor-inducible 14 protein protects against renal ischemia reperfusion injury |
| Q89964419 | Divergent Roles of Inflammation in Skeletal Muscle Recovery From Injury |
| Q44193895 | Effects of intermittent media replacement on the gene expression of differentiating neural progenitor cells |
| Q48822291 | Expression of TWEAK and its receptor Fn14 in the multiple sclerosis brain: implications for inflammatory tissue injury |
| Q42492245 | Expression of TWEAK in normal human skin, dermatitis and epidermal neoplasms: association with proliferation and differentiation of keratinocytes. |
| Q37690660 | Expression of tumor necrosis factor-like weak inducer of apoptosis and fibroblast growth factor-inducible 14 in patients with polymyositis and dermatomyositis |
| Q44266626 | Expression profiling of skeletal muscle in young bulls treated with steroidal growth promoters. |
| Q28294844 | Fibroblast growth factor inducible 14 (Fn14) is required for the expression of myogenic regulatory factors and differentiation of myoblasts into myotubes. Evidence for TWEAK-independent functions of Fn14 during myogenesis |
| Q33624326 | Fibroblast growth factor-inducible 14 (Fn14) is expressed in the lower genital tract and may play a role in amplifying inflammation during infection. |
| Q34162346 | Genetic Ablation of TWEAK Augments Regeneration and Post-Injury Growth of Skeletal Muscle in Mice |
| Q64096636 | Genome-Wide Microarray Analysis Suggests Transcriptomic Response May Not Play a Major Role in High- to Low-Altitude Acclimation in Harvest Mouse () |
| Q41199861 | HPV Type 16 Infection Switches Keratinocytes from Apoptotic to Proliferative Fate under TWEAK/Fn14 Interaction |
| Q34060199 | Hedgehog signaling is critical for normal liver regeneration after partial hepatectomy in mice |
| Q26991848 | Historical perspectives on tumor necrosis factor and its superfamily: 25 years later, a golden journey |
| Q39853470 | Human cardiac explant-conditioned medium: soluble factors and cardiomyogenic effect on mesenchymal stem cells. |
| Q61442950 | Human skeletal muscle macrophages increase following cycle training and are associated with adaptations that may facilitate growth |
| Q46893464 | Identification of adult hepatic progenitor cells capable of repopulating injured rat liver |
| Q33753870 | Immune and myodegenerative pathomechanisms in inclusion body myositis |
| Q51835509 | Influence of narrowband ultraviolet B phototherapy on serum tumour necrosis factor-like weak inducer of apoptosis (TWEAK) in patients with psoriasis. |
| Q28273742 | Interaction of TWEAK with Fn14 leads to the progression of fibrotic liver disease by directly modulating hepatic stellate cell proliferation |
| Q33584761 | Modulation of ozone-sensitive genes in alpha-tocopherol transfer protein null mice |
| Q42598607 | Molecular characterization of cytokine TWEAK and its receptor Fn14 in pig (Sus scrofa). |
| Q54346149 | Molecular structure and characterization of the cytokine TWEAK and its receptor Fn14 in bovine. |
| Q57092433 | Muscle Fn14 gene expression is associated with fat-free mass retention during energy deficit at high altitude |
| Q35436852 | Muscle inflammation susceptibility: a prognostic index of recovery potential after hip arthroplasty? |
| Q37075935 | NF-kappaB signaling in skeletal muscle: prospects for intervention in muscle diseases |
| Q37446811 | Nuclear Factor κB is Required for Tumor Growth Inhibition Mediated by Enavatuzumab (PDL192), a Humanized Monoclonal Antibody to TweakR. |
| Q34559082 | PGC1α plays a critical role in TWEAK-induced cardiac dysfunction |
| Q37091285 | Pathology is alleviated by doxycycline in a laminin-alpha2-null model of congenital muscular dystrophy |
| Q26824503 | Pharmacology of manipulating lean body mass |
| Q37368645 | Proinflammatory cytokine tumor necrosis factor (TNF)-like weak inducer of apoptosis (TWEAK) suppresses satellite cell self-renewal through inversely modulating Notch and NF-κB signaling pathways |
| Q33676528 | Regulation of fibroblast growth factor-inducible 14 (Fn14) expression levels via ligand-independent lysosomal degradation |
| Q39120708 | Regulation of muscle growth and regeneration by the immune system |
| Q28658848 | Regulatory circuitry of TWEAK-Fn14 system and PGC-1α in skeletal muscle atrophy program. |
| Q28066930 | Repair Injured Heart by Regulating Cardiac Regenerative Signals |
| Q41574249 | Role of Omentin, Vaspin, Cardiotrophin-1, TWEAK and NOV/CCN3 in Obesity and Diabetes Development |
| Q38021183 | Role of TWEAK in lupus nephritis: a bench-to-bedside review |
| Q38189358 | Role of the TWEAK-Fn14-cIAP1-NF-κB Signaling Axis in the Regulation of Myogenesis and Muscle Homeostasis |
| Q38101240 | Signaling mechanisms in mammalian myoblast fusion |
| Q34277356 | Single muscle fiber gene expression with run taper |
| Q37422514 | Skeletal muscle as a paradigm for regenerative biology and medicine |
| Q57801740 | Soluble TNF-like weak inducer of apoptosis (TWEAK) enhances poly(I:C)-induced RIPK1-mediated necroptosis |
| Q51371786 | Soluble tumor necrosis factor-like weak inducer of apoptosis plasma levels as a novel biomarker of endothelial function in prevalent orthotopic heart transplant recipients. |
| Q39443461 | Studies of binding of tumor necrosis factor (TNF)-like weak inducer of apoptosis (TWEAK) to fibroblast growth factor inducible 14 (Fn14). |
| Q31132382 | TNF-like weak inducer of apoptosis (TWEAK) promotes beta cell neogenesis from pancreatic ductal epithelium in adult mice |
| Q39832953 | TNF-like weak inducer of apoptosis inhibits proinflammatory TNF receptor-1 signaling |
| Q30277293 | TWEAK Regulates Muscle Functions in a Mouse Model of RNA Toxicity |
| Q37697186 | TWEAK activation of the non-canonical NF-κB signaling pathway differentially regulates melanoma and prostate cancer cell invasion |
| Q36097846 | TWEAK and TRAF6 regulate skeletal muscle atrophy. |
| Q39257705 | TWEAK and cIAP1 regulate myoblast fusion through the noncanonical NF-κB signaling pathway |
| Q26996775 | TWEAK and the progression of renal disease: clinical translation |
| Q82261059 | TWEAK regulates proliferation and differentiation of adult neural progenitor cells |
| Q37893443 | TWEAK, a multifunctional cytokine in kidney injury |
| Q38194552 | TWEAK-Fn14 Cytokine-Receptor Axis: A New Player of Myocardial Remodeling and Cardiac Failure |
| Q45652001 | TWEAK-Fn14 Signaling Activates Myofibroblasts to Drive Progression of Fibrotic Kidney Disease. |
| Q35138483 | TWEAK-Fn14 pathway activation after exercise in human skeletal muscle: insights from two exercise modes and a time course investigation |
| Q37516544 | TWEAK/Fn14 Signaling Axis Mediates Skeletal Muscle Atrophy and Metabolic Dysfunction. |
| Q38171480 | TWEAK/Fn14 and Non-Canonical NF-kappaB Signaling in Kidney Disease |
| Q84067885 | TWEAK/Fn14 promotes the proliferation and collagen synthesis of rat cardiac fibroblasts via the NF-кB pathway |
| Q30301119 | TWEAK/Fn14, a pathway and novel therapeutic target in myotonic dystrophy |
| Q38177788 | TWEAK: A New Player in Obesity and Diabetes. |
| Q26795601 | The TWEAK receptor Fn14 is a potential cell surface portal for targeted delivery of glioblastoma therapeutics |
| Q37135363 | The TWEAK-Fn14 cytokine-receptor axis: discovery, biology and therapeutic targeting |
| Q33724956 | The TWEAK-Fn14 dyad is involved in age-associated pathological changes in skeletal muscle |
| Q26999255 | The TWEAK-Fn14 pathway: a potent regulator of skeletal muscle biology in health and disease |
| Q38129961 | The TWEAK-Fn14 system as a potential drug target |
| Q33751732 | The TWEAK-Fn14 system is a critical regulator of denervation-induced skeletal muscle atrophy in mice. |
| Q35671234 | The TWEAK-Fn14 system: breaking the silence of cytokine-induced skeletal muscle wasting |
| Q39275635 | The obestatin/GPR39 system is up-regulated by muscle injury and functions as an autocrine regenerative system |
| Q36630399 | The role of TWEAK/Fn14 signaling in the MPTP-model of Parkinson's disease |
| Q36230448 | Therapeutic potential of the TWEAK/Fn14 pathway in intractable gastrointestinal cancer |
| Q84476548 | To TWEAK, or not to TWEAK: that is the question |
| Q36001939 | Transcriptome signature of resistance exercise adaptations: mixed muscle and fiber type specific profiles in young and old adults |
| Q37383983 | Transient TWEAK overexpression leads to a general salivary epithelial cell proliferation |
| Q35922471 | Trpc1 ion channel modulates phosphatidylinositol 3-kinase/Akt pathway during myoblast differentiation and muscle regeneration. |
| Q43005719 | Tumor necrosis factor-like weak inducer of apoptosis is a mitogen for liver progenitor cells |
| Q35026182 | Tumor necrosis factor-like weak inducer of apoptosis or Fn14 deficiency reduce elastase perfusion-induced aortic abdominal aneurysm in mice. |
| Q47148974 | Tumor necrosis factor-like weak inducer of apoptosis regulates quadriceps muscle atrophy and fiber-type alteration in a rat model of chronic obstructive pulmonary disease |
| Q35893298 | Tweak induces proliferation in renal tubular epithelium: a role in uninephrectomy induced renal hyperplasia |
| Q48283526 | Tweak regulates astrogliosis, microgliosis and skeletal muscle atrophy in a mouse model of amyotrophic lateral sclerosis. |
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