| scholarly article | Q13442814 |
| P50 | author | Ana B Sanz | Q63413139 |
| Maria Dolores Sanchez-niño | Q42881464 | ||
| Luis M Blanco-Colio | Q55542660 | ||
| Marta Ruiz-Ortega | Q56616783 | ||
| P2093 | author name string | Alberto Ortiz | |
| Jesús Egido | |||
| Maria Concepcion Izquierdo | |||
| Pilar Justo | |||
| Aniela Jakubowski | |||
| P2860 | cites work | Tumor necrosis factor primes hepatocytes for DNA replication in the rat | Q58022178 |
| Expression of apoptosis regulatory proteins in tubular epithelium stressed in culture or following acute renal failure | Q63684402 | ||
| Expression of cytokine genes during liver regeneration after partial hepatectomy in rats | Q72618125 | ||
| TWEAK is an endothelial cell growth and chemotactic factor that also potentiates FGF-2 and VEGF-A mitogenic activity | Q73075512 | ||
| Analysis of liver regeneration in mice lacking type 1 or type 2 tumor necrosis factor receptor: requirement for type 1 but not type 2 receptor | Q77355546 | ||
| TWEAK is expressed by glial cells, induces astrocyte proliferation and increases EAE severity | Q78555839 | ||
| TWEAK/Fn14 interactions are instrumental in the pathogenesis of nephritis in the chronic graft-versus-host model of systemic lupus erythematosus | Q79912772 | ||
| Immunosuppressive effects of surgery assessed by flow cytometry in nonhuman primates after nephrectomy | Q81214251 | ||
| TWEAK, via its receptor Fn14, is a novel regulator of mesenchymal progenitor cells and skeletal muscle regeneration | Q24670970 | ||
| TWEAK induces angiogenesis and proliferation of endothelial cells | Q28139175 | ||
| The mitogen-inducible Fn14 gene encodes a type I transmembrane protein that modulates fibroblast adhesion and migration | Q28146243 | ||
| TWEAK, a member of the TNF superfamily, is a multifunctional cytokine that binds the TweakR/Fn14 receptor | Q28176724 | ||
| Pro-inflammatory effect of TWEAK/Fn14 interaction on human umbilical vein endothelial cells | Q28215696 | ||
| Phosphatidylinositol 3-kinase in interleukin 1 signaling. Physical interaction with the interleukin 1 receptor and requirement in NFkappaB and AP-1 activation | Q28253820 | ||
| TWEAK, a new secreted ligand in the tumor necrosis factor family that weakly induces apoptosis | Q28257134 | ||
| The role of TWEAK/Fn14 in the pathogenesis of inflammation and systemic autoimmunity | Q28280794 | ||
| The tumor necrosis factor-like weak inducer of apoptosis (TWEAK)-fibroblast growth factor-inducible 14 (Fn14) signaling system regulates glioma cell survival via NFkappaB pathway activation and BCL-XL/BCL-W expression | Q28299334 | ||
| The ERK1/2 mitogen-activated protein kinase pathway as a master regulator of the G1- to S-phase transition | Q28301955 | ||
| NF-kappaB at the crossroads of life and death | Q29619302 | ||
| A previously unrecognized protein-protein interaction between TWEAK and CD163: potential biological implications | Q33286687 | ||
| Signal transduction and transcription factors in rheumatic disease | Q33598827 | ||
| TWEAK induces liver progenitor cell proliferation | Q33919951 | ||
| Apoptotic cell death in renal injury: the rationale for intervention | Q35022973 | ||
| TWEAK/Fn14 interaction regulates RANTES production, BMP-2-induced differentiation, and RANKL expression in mouse osteoblastic MC3T3-E1 cells | Q35605699 | ||
| Characterization of a renal tubular epithelial cell line which secretes the autologous target antigen of autoimmune experimental interstitial nephritis | Q36219361 | ||
| The cytokine TWEAK modulates renal tubulointerstitial inflammation | Q36666882 | ||
| TWEAKing renal injury | Q36991865 | ||
| Fas transduces dual apoptotic and trophic signals in hematopoietic progenitors | Q39345317 | ||
| Tumor necrosis factor-like weak inducer of apoptosis attenuates the action of insulin in hepatocytes | Q40028462 | ||
| Cytokine cooperation in renal tubular cell injury: the role of TWEAK. | Q40227178 | ||
| IKKbeta/2 induces TWEAK and apoptosis in mammary epithelial cells | Q40300890 | ||
| Proinflammatory effects of TWEAK/Fn14 interactions in glomerular mesangial cells | Q40327315 | ||
| Fas transduces activation signals in normal human T lymphocytes | Q41906015 | ||
| Expression of Smac/Diablo in tubular epithelial cells and during acute renal failure. | Q44581614 | ||
| Activation of cyclin D1 expression by the ERK5 cascade | Q47670094 | ||
| Compensatory kidney growth in estrogen receptor-alpha null mice. | Q51840923 | ||
| Compensatory renal growth in uninephrectomized adult mice is growth hormone dependent. | Q52172565 | ||
| Fas signaling in thyroid carcinomas is diverted from apoptosis to proliferation. | Q53617699 | ||
| P433 | issue | 9B | |
| P304 | page(s) | 3329-3342 | |
| P577 | publication date | 2009-05-01 | |
| P1433 | published in | Journal of Cellular and Molecular Medicine | Q1524063 |
| P1476 | title | Tweak induces proliferation in renal tubular epithelium: a role in uninephrectomy induced renal hyperplasia | |
| P478 | volume | 13 |
| Q44979188 | 3,4-DGE is cytotoxic and decreases HSP27/HSPB1 in podocytes. |
| Q35804184 | Aberrant tubuloglomerular feedback and HIF-1α confer resistance to ischemia after subtotal nephrectomy |
| Q33748559 | BASP1 promotes apoptosis in diabetic nephropathy. |
| Q41480524 | Bcl3: a regulator of NF-κB inducible by TWEAK in acute kidney injury with anti-inflammatory and antiapoptotic properties in tubular cells |
| Q89441236 | Biomeasures and mechanistic modeling highlight PK/PD risks for a monoclonal antibody targeting Fn14 in kidney disease |
| Q33944773 | Combined therapy with renin-angiotensin system and calcium channel blockers in type 2 diabetic hypertensive patients with proteinuria: effects on soluble TWEAK, PTX3, and flow-mediated dilation |
| Q38404587 | Designing drugs that combat kidney damage. |
| Q38985548 | Downregulation of kidney protective factors by inflammation: role of transcription factors and epigenetic mechanisms |
| Q56363330 | Evolution, kidney development, and chronic kidney disease |
| Q43573882 | Evolutionary Nephrology |
| Q41763293 | Histone lysine crotonylation during acute kidney injury in mice |
| Q34450860 | Inflammatory cytokines and survival factors from serum modulate tweak-induced apoptosis in PC-3 prostate cancer cells |
| Q36354235 | Inhibition of the TWEAK/Fn14 pathway attenuates renal disease in nephrotoxic serum nephritis |
| Q38842507 | MicroRNA and receptor mediated signaling pathways as potential therapeutic targets in heart failure |
| Q94527020 | Microvesicles from indoxyl sulfate-treated endothelial cells induce vascular calcification in vitro |
| Q39398586 | Mitogen-Activated Protein Kinase 14 Promotes AKI. |
| Q36129845 | One dose of cyclosporine A is protective at initiation of folic acid-induced acute kidney injury in mice |
| Q38102743 | Progress in the development of animal models of acute kidney injury and its impact on drug discovery |
| Q38113962 | Regulation of PI-3-Kinase and Akt Signaling in T Lymphocytes and Other Cells by TNFR Family Molecules |
| Q38021183 | Role of TWEAK in lupus nephritis: a bench-to-bedside review |
| Q38992322 | Roles of tumour necrosis factor-related weak inducer of apoptosis/fibroblast growth factor-inducible 14 pathway in lupus nephritis |
| Q27347256 | Soluble Fn14 Is Detected and Elevated in Mouse and Human Kidney Disease |
| Q34751855 | Soluble TWEAK and PTX3 in nondialysis CKD patients: impact on endothelial dysfunction and cardiovascular outcomes |
| Q43801106 | Soluble TWEAK levels are independently associated with coronary artery disease severity in patients with stage 2-3 kidney disease. |
| Q51538354 | TLR4-mediated inflammation is a key pathogenic event leading to kidney damage and fibrosis in cyclosporine nephrotoxicity. |
| Q30479123 | TNF superfamily: a growing saga of kidney injury modulators. |
| Q38925875 | TNF-related weak inducer of apoptosis (TWEAK) regulates junctional proteins in tubular epithelial cells via canonical NF-κB pathway and ERK activation |
| Q64266102 | TRAIL, OPG, and TWEAK in kidney disease: biomarkers or therapeutic targets? |
| Q39406226 | TWEAK (tumor necrosis factor-like weak inducer of apoptosis) activates CXCL16 expression during renal tubulointerstitial inflammation |
| Q28749137 | TWEAK activates the non-canonical NFkappaB pathway in murine renal tubular cells: modulation of CCL21 |
| Q26996775 | TWEAK and the progression of renal disease: clinical translation |
| Q40640099 | TWEAK favors phosphate-induced calcification of vascular smooth muscle cells through canonical and non-canonical activation of NFκB. |
| Q55490942 | TWEAK increases CD74 expression and sensitizes to DDT proinflammatory actions in tubular cells. |
| Q57787023 | TWEAK promotes endothelial progenitor cell vasculogenesis to alleviate acute myocardial infarction via the Fn14-NF-κB signaling pathway |
| Q27318227 | TWEAK promotes peritoneal inflammation |
| Q37893443 | TWEAK, a multifunctional cytokine in kidney injury |
| Q38194552 | TWEAK-Fn14 Cytokine-Receptor Axis: A New Player of Myocardial Remodeling and Cardiac Failure |
| Q35073736 | TWEAK-Fn14 as a mediator of acute kidney injury |
| Q38171480 | TWEAK/Fn14 and Non-Canonical NF-kappaB Signaling in Kidney Disease |
| Q84067885 | TWEAK/Fn14 promotes the proliferation and collagen synthesis of rat cardiac fibroblasts via the NF-кB pathway |
| Q38129961 | The TWEAK-Fn14 system as a potential drug target |
| Q38016615 | The TWEAK/Fn14 pathway as an aggravating and perpetuating factor in inflammatory diseases: focus on inflammatory bowel diseases |
| Q62932708 | The inflammatory cytokine TWEAK decreases PGC-1α expression and mitochondrial function in acute kidney injury |
| Q39515008 | The inflammatory cytokines TWEAK and TNFα reduce renal klotho expression through NFκB. |
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