scholarly article | Q13442814 |
P50 | author | Marylyn Addo | Q66714025 |
Marcus Altfeld | Q72531060 | ||
Daniel E Kaufmann | Q87677945 | ||
Eric S Rosenberg | Q89548046 | ||
Mathias Lichterfeld | Q91496240 | ||
Elizabeth T Kalife | Q114576937 | ||
Daryld A Strick | Q114576938 | ||
Bruce D. Walker | Q23770361 | ||
P2093 | author name string | Mary N Johnston | |
Bradford S Wagner | |||
Paul K Lee | |||
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Tat-specific cytotoxic T lymphocytes select for SIV escape variants during resolution of primary viraemia | Q57075698 | ||
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Structured antiretroviral treatment interruptions in chronically HIV-1-infected subjects | Q33949316 | ||
High-level HIV-1 viremia suppresses viral antigen-specific CD4(+) T cell proliferation | Q33950164 | ||
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Treatment of primary HIV-1 infection with cyclosporin A coupled with highly active antiretroviral therapy | Q34789249 | ||
The functional CD8 T cell response to HIV becomes type-specific in progressive disease | Q34815358 | ||
Viral persistence alters CD8 T-cell immunodominance and tissue distribution and results in distinct stages of functional impairment | Q34857689 | ||
Enhanced detection of human immunodeficiency virus type 1-specific T-cell responses to highly variable regions by using peptides based on autologous virus sequences | Q35149407 | ||
Cellular immune responses and viral diversity in individuals treated during acute and early HIV-1 infection. | Q36369035 | ||
HIV-1 viremia prevents the establishment of interleukin 2-producing HIV-specific memory CD4+ T cells endowed with proliferative capacity | Q36371562 | ||
Diminished Proliferation of Human Immunodeficiency Virus-Specific CD4+T Cells Is Associated with Diminished Interleukin-2 (IL-2) Production and Is Recovered by Exogenous IL-2 | Q36474240 | ||
Characteristics of the specific cell-mediated immune response in human immunodeficiency virus infection | Q36889122 | ||
Persistent recognition of autologous virus by high-avidity CD8 T cells in chronic, progressive human immunodeficiency virus type 1 infection. | Q36943282 | ||
Infection with Multiple Human Immunodeficiency Virus Type 1 Variants Is Associated with Faster Disease Progression | Q37059806 | ||
Transient mobilization of human immunodeficiency virus (HIV)-specific CD4 T-helper cells fails to control virus rebounds during intermittent antiretroviral therapy in chronic HIV type 1 infection | Q39601164 | ||
Predictors of Plasma Human Immunodeficiency Virus Type 1 RNA Control after Discontinuation of Highly Active Antiretroviral Therapy Initiated at Acute Infection Combined with Structured Treatment Interruptions and Immune‐Based Therapies | Q40548790 | ||
A controlled trial of granulocyte macrophage-colony stimulating factor during interruption of HAART. | Q40556224 | ||
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Impact of hepatitis G virus co-infection on the course of hepatitis C virus infection before and after liver transplantation | Q42991527 | ||
Effect of coinfection with GB virus C on survival among patients with HIV infection | Q43034157 | ||
Influence of combinations of human major histocompatibility complex genes on the course of HIV-1 infection | Q43542210 | ||
A prospective trial of structured treatment interruptions in human immunodeficiency virus infection | Q44767301 | ||
Skewed representation of functionally distinct populations of virus-specific CD4 T cells in HIV-1-infected subjects with progressive disease: changes after antiretroviral therapy | Q45721932 | ||
Structured treatment interruption in patients with multidrug-resistant human immunodeficiency virus | Q45722048 | ||
HIV-1 superinfection despite broad CD8+ T-cell responses containing replication of the primary virus | Q45729968 | ||
Effect of prolonged discontinuation of successful antiretroviral therapy on CD4+ T cell decline in human immunodeficiency virus-infected patients: implications for intermittent therapeutic strategies | Q45731190 | ||
Discontinuation of antiretroviral therapy commenced early during the course of human immunodeficiency virus type 1 infection, with or without adjunctive vaccination | Q45731214 | ||
Enhancement of human immunodeficiency virus type 1-specific CD4 and CD8 T cell responses in chronically infected persons after temporary treatment interruption | Q45740983 | ||
Natural history of human immunodeficiency virus type 1 viremia after seroconversion and proximal to AIDS in a large cohort of homosexual men. Multicenter AIDS Cohort Study | Q45743784 | ||
Antiretroviral-drug resistance among patients recently infected with HIV. | Q46277555 | ||
HIV evolution: CTL escape mutation and reversion after transmission | Q47211742 | ||
Humoral immunity to HIV-1: kinetics of antibody responses in chronic infection reflects capacity of immune system to improve viral set point | Q47371942 | ||
HIV-1-specific cytotoxicity is preferentially mediated by a subset of CD8(+) T cells producing both interferon-gamma and tumor necrosis factor-alpha | Q47835000 | ||
Comparison of overlapping peptide sets for detection of antiviral CD8 and CD4 T cell responses | Q48002526 | ||
P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
P6216 | copyright status | copyrighted | Q50423863 |
P433 | issue | 2 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | acute HIV infection | Q12777087 |
P304 | page(s) | e36 | |
P577 | publication date | 2004-11-01 | |
P1433 | published in | PLOS MEDICINE | Q1686921 |
P1476 | title | Limited durability of viral control following treated acute HIV infection | |
P478 | volume | 1 |
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Q36471285 | Attenuated disease in SIV-infected macaques treated with a monoclonal antibody against FasL. |
Q35175897 | Augmented HIV-specific interferon-gamma responses, but impaired lymphoproliferation during interruption of antiretroviral treatment initiated in primary HIV infection |
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Q28766705 | Complement lysis activity in autologous plasma is associated with lower viral loads during the acute phase of HIV-1 infection |
Q28472597 | Control of viremia and prevention of AIDS following immunotherapy of SIV-infected macaques with peptide-pulsed blood |
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Q41762711 | HIV controllers maintain a population of highly efficient Th1 effector cells in contrast to patients treated in the long term. |
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