scholarly article | Q13442814 |
P2093 | author name string | Alan M Weiner | |
Arnold D Bailey | |||
John C Newman | |||
P2860 | cites work | Gene ontology: tool for the unification of biology | Q23781406 |
Translocation of Cockayne syndrome group A protein to the nuclear matrix: possible relevance to transcription-coupled DNA repair | Q24292134 | ||
Functional consequences of mutations in the conserved SF2 motifs and post-translational phosphorylation of the CSB protein | Q24293604 | ||
The ubiquitin ligase activity in the DDB2 and CSA complexes is differentially regulated by the COP9 signalosome in response to DNA damage | Q24301297 | ||
BRCA1/BARD1 inhibition of mRNA 3' processing involves targeted degradation of RNA polymerase II. | Q24302667 | ||
Human transcription-repair coupling factor CSB/ERCC6 is a DNA-stimulated ATPase but is not a helicase and does not disrupt the ternary transcription complex of stalled RNA polymerase II | Q24314310 | ||
Cooperation of the Cockayne syndrome group B protein and poly(ADP-ribose) polymerase 1 in the response to oxidative stress. | Q24316039 | ||
Cockayne syndrome group B protein enhances elongation by RNA polymerase II | Q24317053 | ||
p53 modulation of TFIIH–associated nucleotide excision repair activity | Q24336880 | ||
UV-induced ubiquitination of RNA polymerase II: a novel modification deficient in Cockayne syndrome cells | Q24605185 | ||
Recruitment of the putative transcription-repair coupling factor CSB/ERCC6 to RNA polymerase II elongation complexes | Q24644162 | ||
The cyclooxygenases | Q24801433 | ||
oPOSSUM: identification of over-represented transcription factor binding sites in co-expressed genes | Q24810241 | ||
L2L: a simple tool for discovering the hidden significance in microarray expression data | Q24814110 | ||
Summaries of Affymetrix GeneChip probe level data | Q27860888 | ||
Dynamic lysine methylation on histone H3 defines the regulatory phase of gene transcription | Q27932721 | ||
Requirement of yeast RAD2, a homolog of human XPG gene, for efficient RNA polymerase II transcription. implications for Cockayne syndrome | Q27933687 | ||
TFIIH plays an essential role in RNA polymerase I transcription | Q27934158 | ||
Activation of p53 or loss of the Cockayne syndrome group B repair protein causes metaphase fragility of human U1, U2, and 5S genes | Q28140055 | ||
A summary of mutations in the UV-sensitive disorders: xeroderma pigmentosum, Cockayne syndrome, and trichothiodystrophy | Q28142117 | ||
The orphan nuclear receptor SXR coordinately regulates drug metabolism and efflux | Q28186424 | ||
Enhanceosomes | Q28204862 | ||
ERCC6, a member of a subfamily of putative helicases, is involved in Cockayne's syndrome and preferential repair of active genes | Q28213725 | ||
CSB is a component of RNA pol I transcription | Q28610049 | ||
A summary of mutations in the UV-sensitive disorders: Xeroderma pigmentosum, Cockayne syndrome, and trichothiodystrophy | Q29014018 | ||
Antiproliferative activity of ecteinascidin 743 is dependent upon transcription-coupled nucleotide-excision repair. | Q30998417 | ||
Activation of the p21WAF1/CIP1 promoter independent of p53 by the histone deacetylase inhibitor suberoylanilide hydroxamic acid (SAHA) through the Sp1 sites | Q31750307 | ||
Requirement for yeast RAD26, a homolog of the human CSB gene, in elongation by RNA polymerase II | Q33551567 | ||
Differential requirement for the ATPase domain of the Cockayne syndrome group B gene in the processing of UV-induced DNA damage and 8-oxoguanine lesions in human cells | Q33559326 | ||
The genetic defect in Cockayne syndrome is associated with a defect in repair of UV-induced DNA damage in transcriptionally active DNA. | Q33640104 | ||
Cockayne syndrome and xeroderma pigmentosum | Q34143455 | ||
Role of T antigen interactions with p53 in tumorigenesis | Q34177970 | ||
Histone deacetylase inhibitor up-regulates RECK to inhibit MMP-2 activation and cancer cell invasion. | Q34206478 | ||
Cockayne syndrome: Review of 140 cases | Q34232807 | ||
A weak signal for strong responses: interferon-alpha/beta revisited | Q34238056 | ||
Gene expression profiling in Werner syndrome closely resembles that of normal aging | Q34267712 | ||
Gene regulation and DNA damage in the ageing human brain | Q34326236 | ||
Dark matter in the genome: evidence of widespread transcription detected by microarray tiling experiments | Q34386366 | ||
Ecteinascidin-743 inhibits activated but not constitutive transcription. | Q34524124 | ||
Whole-genome expression analysis of snf/swi mutants of Saccharomyces cerevisiae | Q35110235 | ||
Impact of aging on DNA methylation | Q35119503 | ||
PARP goes transcription | Q35155777 | ||
Interference of transcriptional activation by the antineoplastic drug ecteinascidin-743. | Q35802653 | ||
A flame burning within | Q35907508 | ||
Insights into host responses against pathogens from transcriptional profiling. | Q36087019 | ||
Nucleotide excision repair in chromatin and the right of entry | Q36165675 | ||
Induction of interferon-stimulated gene expression and antiviral responses require protein deacetylase activity. | Q36447938 | ||
Incipient Alzheimer's disease: microarray correlation analyses reveal major transcriptional and tumor suppressor responses | Q36605276 | ||
Complete absence of Cockayne syndrome group B gene product gives rise to UV-sensitive syndrome but not Cockayne syndrome | Q37593459 | ||
Transcriptional regulation of cyclooxygenase-1 by histone deacetylase inhibitors in normal human astrocyte cells | Q38291503 | ||
Transcriptional profiles of unirradiated or UV-irradiated human cells expressing either the cancer-prone XPB/CS allele or the noncancer-prone XPB/TTD allele. | Q38332883 | ||
Biochemical and biological characterization of wild-type and ATPase-deficient Cockayne syndrome B repair protein. | Q38336695 | ||
ATP-dependent chromatin remodeling by the Cockayne syndrome B DNA repair-transcription-coupling factor. | Q39455992 | ||
BRCA1/BARD1 ubiquitinate phosphorylated RNA polymerase II. | Q40424257 | ||
The transcriptional response after oxidative stress is defective in Cockayne syndrome group B cells | Q40667908 | ||
The cockayne syndrome group B gene product is involved in cellular repair of 8-hydroxyadenine in DNA. | Q44024895 | ||
Susceptibility of multidrug resistance tumor cells to apoptosis induction by histone deacetylase inhibitors | Q44321035 | ||
Both Rb/p16INK4a inactivation and telomerase activity are required to immortalize human epithelial cells. | Q44816904 | ||
The CSB protein actively wraps DNA. | Q45153429 | ||
Cockayne syndrome, between transcription and DNA repair defects | Q74657696 | ||
P433 | issue | 25 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | Cockayne syndrome | Q914389 |
P304 | page(s) | 9613-8 | |
P577 | publication date | 2006-06-20 | |
P1433 | published in | Proceedings of the National Academy of Sciences of the United States of America | Q1146531 |
P1476 | title | Cockayne syndrome group B protein (CSB) plays a general role in chromatin maintenance and remodeling | |
P478 | volume | 103 |
Q36120408 | A simple gene set-based method accurately predicts the synergy of drug pairs |
Q39694111 | A ubiquitin-binding domain in Cockayne syndrome B required for transcription-coupled nucleotide excision repair |
Q37021545 | A ubiquitylation site in Cockayne syndrome B required for repair of oxidative DNA damage, but not for transcription-coupled nucleotide excision repair |
Q37609639 | ALS-associated mutation FUS-R521C causes DNA damage and RNA splicing defects |
Q34699754 | ATP-dependent chromatin remodeling by Cockayne syndrome protein B and NAP1-like histone chaperones is required for efficient transcription-coupled DNA repair |
Q35865561 | ATP-dependent chromatin remodeling factors and DNA damage repair |
Q28755346 | An abundant evolutionarily conserved CSB-PiggyBac fusion protein expressed in Cockayne syndrome |
Q42503209 | An altered redox balance mediates the hypersensitivity of Cockayne syndrome primary fibroblasts to oxidative stress. |
Q37038917 | Bidirectional transcription of trinucleotide repeats: roles for excision repair |
Q34554610 | Blinded by the UV light: how the focus on transcription-coupled NER has distracted from understanding the mechanisms of Cockayne syndrome neurologic disease |
Q90727995 | CSB affected on the sensitivity of lung cancer cells to platinum-based drugs through the global decrease of let-7 and miR-29 |
Q36839881 | Clinical implications of the basic defects in Cockayne syndrome and xeroderma pigmentosum and the DNA lesions responsible for cancer, neurodegeneration and aging |
Q35049796 | Cockayne Syndrome B Protects Against Methamphetamine-Enhanced Oxidative DNA Damage in Murine Fetal Brain and Postnatal Neurodevelopmental Deficits |
Q39294995 | Cockayne Syndrome group B protein interacts with TRF2 and regulates telomere length and stability |
Q37638150 | Cockayne Syndrome group B protein stimulates NEIL2 DNA glycosylase activity. |
Q35166110 | Cockayne syndrome B protein antagonizes OGG1 in modulating CAG repeat length in vivo |
Q38712278 | Cockayne syndrome B protein regulates recruitment of the Elongin A ubiquitin ligase to sites of DNA damage. |
Q34469204 | Cockayne syndrome group B protein regulates DNA double-strand break repair and checkpoint activation |
Q35974689 | Cockayne syndrome, underlying molecular defects and p53. |
Q36676801 | Cockayne syndrome-derived neurons display reduced synapse density and altered neural network synchrony |
Q28078628 | DNA Damage: From Chronic Inflammation to Age-Related Deterioration |
Q35006896 | Disorders of nucleotide excision repair: the genetic and molecular basis of heterogeneity |
Q36690915 | Do all of the neurologic diseases in patients with DNA repair gene mutations result from the accumulation of DNA damage? |
Q39525558 | Downregulation of Cockayne syndrome B protein reduces human 8-oxoguanine DNA glycosylase-1 expression and repair of UV radiation-induced 8-oxo-7,8-dihydro-2'-deoxyguanine. |
Q35849782 | Dysmyelination not demyelination causes neurological symptoms in preweaned mice in a murine model of Cockayne syndrome. |
Q34407717 | Dysregulation of gene expression as a cause of Cockayne syndrome neurological disease |
Q28307320 | Ecteinascidin 743 interferes with the activity of EWS-FLI1 in Ewing sarcoma cells |
Q34503412 | Elements That Regulate the DNA Damage Response of Proteins Defective in Cockayne Syndrome |
Q60958004 | Epigenetic Regulation of Skin Cells in Natural Aging and Premature Aging Diseases |
Q24657549 | Epigenetic principles and mechanisms underlying nervous system functions in health and disease |
Q36142076 | Evidence for premature aging due to oxidative stress in iPSCs from Cockayne syndrome |
Q33794136 | Gender and cell-type-specific effects of the transcription-coupled repair protein, ERCC6/CSB, on repeat expansion in a mouse model of the fragile X-related disorders |
Q38078209 | Genetic syndromes caused by mutations in epigenetic genes |
Q36711222 | Human U2 snRNA genes exhibit a persistently open transcriptional state and promoter disassembly at metaphase |
Q35647709 | Identification of Novel Proteins Co-Purifying with Cockayne Syndrome Group B (CSB) Reveals Potential Roles for CSB in RNA Metabolism and Chromatin Dynamics. |
Q24682525 | Increased apoptosis, p53 up-regulation, and cerebellar neuronal degeneration in repair-deficient Cockayne syndrome mice |
Q26749169 | Mechanisms of FUS mutations in familial amyotrophic lateral sclerosis |
Q90387567 | Mechanistic insights into the regulation of transcription and transcription-coupled DNA repair by Cockayne syndrome protein B |
Q36203024 | Meta-analysis of microarray results: challenges, opportunities, and recommendations for standardization |
Q33821544 | Meta-analysis of muscle transcriptome data using the MADMuscle database reveals biologically relevant gene patterns |
Q92340005 | Molecular basis of chromatin remodeling by Rhp26, a yeast CSB ortholog |
Q36963083 | Multiple interaction partners for Cockayne syndrome proteins: implications for genome and transcriptome maintenance |
Q39931897 | Mutation update for the CSB/ERCC6 and CSA/ERCC8 genes involved in Cockayne syndrome. |
Q33636033 | NAP1L1 accelerates activation and decreases pausing to enhance nucleosome remodeling by CSB |
Q37308518 | Nucleic acid binding activity of human Cockayne syndrome B protein and identification of Ca(2+) as a novel metal cofactor |
Q91859408 | Poly(ADP-ribose) polymerase 1 (PARP1) promotes oxidative stress-induced association of Cockayne syndrome group B protein with chromatin |
Q57755410 | Polymorphism of DNA repair genes OGG1, XRCC1, XPD and ERCC6 in bladder cancer in Belarus |
Q35266600 | Reciprocally regulated chromatin association of Cockayne syndrome protein B and p53 protein |
Q59799382 | Regulation of the Intranuclear Distribution of the Cockayne Syndrome Proteins |
Q91628294 | Rescue of premature aging defects in Cockayne syndrome stem cells by CRISPR/Cas9-mediated gene correction |
Q38867513 | Sources and consequences of oxidative damage from mitochondria and neurotransmitter signaling |
Q26995830 | Structure, function and regulation of CSB: a multi-talented gymnast |
Q35516084 | Testicular nuclear receptor 4 (TR4) regulates UV light-induced responses via Cockayne syndrome B protein-mediated transcription-coupled DNA repair |
Q34426620 | Tethering of the conserved piggyBac transposase fusion protein CSB-PGBD3 to chromosomal AP-1 proteins regulates expression of nearby genes in humans. |
Q36701021 | The CSB chromatin remodeler and CTCF architectural protein cooperate in response to oxidative stress |
Q36328938 | The Cellular Response to Oxidatively Induced DNA Damage and Polymorphism of Some DNA Repair Genes Associated with Clinicopathological Features of Bladder Cancer |
Q52560605 | The Cellular Response to Transcription-Blocking DNA Damage. |
Q58738685 | The Cockayne syndrome protein B is involved in the repair of 5-AZA-2'-deoxycytidine-induced DNA lesions |
Q35909014 | The Repeat Expansion Diseases: The dark side of DNA repair |
Q34717528 | The chromatin remodeling factor CSB recruits histone acetyltransferase PCAF to rRNA gene promoters in active state for transcription initiation |
Q35923273 | The conserved Cockayne syndrome B-piggyBac fusion protein (CSB-PGBD3) affects DNA repair and induces both interferon-like and innate antiviral responses in CSB-null cells. |
Q37398643 | The molecular basis of chromatin dynamics during nucleotide excision repair |
Q24297071 | The role of CSA in the response to oxidative DNA damage in human cells |
Q40412328 | The role of Cockayne Syndrome Protein B in transcription regulation |
Q36882150 | The role of Cockayne Syndrome group B (CSB) protein in base excision repair and aging. |
Q34649133 | The role of genetics in the establishment and maintenance of the epigenome. |
Q35151644 | The sequence-specific transcription factor c-Jun targets Cockayne syndrome protein B to regulate transcription and chromatin structure. |
Q39042789 | Transcription factors that influence RNA polymerases I and II: To what extent is mechanism of action conserved? |
Q37048164 | Transcription-coupled nucleotide excision repair in mammalian cells: molecular mechanisms and biological effects |
Q39291784 | UV-induced association of the CSB remodeling protein with chromatin requires ATP-dependent relief of N-terminal autorepression. |
Q28292625 | UVSSA and USP7, a new couple in transcription-coupled DNA repair |
Q36755008 | Valosin-containing Protein (VCP)/p97 Segregase Mediates Proteolytic Processing of Cockayne Syndrome Group B (CSB) in Damaged Chromatin |
Q38078156 | What role (if any) does the highly conserved CSB-PGBD3 fusion protein play in Cockayne syndrome? |
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