| scholarly article | Q13442814 |
| P2093 | author name string | A Roher | |
| E G Gray | |||
| M Paula-Barbosa | |||
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| P433 | issue | 2 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | Alzheimer's disease | Q11081 |
| P304 | page(s) | 91-110 | |
| P577 | publication date | 1987-01-01 | |
| P1433 | published in | Neuropathology and Applied Neurobiology | Q7002494 |
| P1476 | title | Alzheimer's disease: paired helical filaments and cytomembranes | |
| P478 | volume | 13 |
| Q38025108 | ACS chemical neuroscience molecule spotlight on ELND006: another γ-secretase inhibitor fails in the clinic |
| Q28215096 | Aberrant glycosylation modulates phosphorylation of tau by protein kinase A and dephosphorylation of tau by protein phosphatase 2A and 5 |
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| Q36192471 | Advances on biological markers in early diagnosis of Alzheimer disease. |
| Q34116321 | Ageing of the brain |
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| Q28215968 | Alzheimer's disease-do tauists and baptists finally shake hands? |
| Q41423732 | Alzheimer's disease-like phosphorylation of the microtubule-associated protein tau by glycogen synthase kinase-3 in transfected mammalian cells. |
| Q53191493 | Alzheimer's disease. Paired helical filaments and the cytoskeleton. |
| Q33706311 | Amyloid and lipids in the pathology of Alzheimer disease. |
| Q44429258 | Anionic Micelles and Vesicles Induce Tau Fibrillization in Vitro |
| Q41240079 | Binding of the three-repeat domain of tau to phospholipid membranes induces an aggregated-like state of the protein. |
| Q35890955 | Biochemical and morphological characterization of the AβPP/PS/tau triple transgenic mouse model and its relevance to sporadic Alzheimer's disease |
| Q28542628 | Biochemical assessment of precuneus and posterior cingulate gyrus in the context of brain aging and Alzheimer's disease |
| Q22252362 | Brain cholinesterases: II. The molecular and cellular basis of Alzheimer's disease |
| Q46858641 | C-terminal truncation modulates both nucleation and extension phases of tau fibrillization |
| Q35211845 | Changes in the ageing brain in health and disease |
| Q41925867 | Chloroquine myopathy suggests that tau is degraded in lysosomes: implication for the formation of paired helical filaments in Alzheimer's disease |
| Q30524309 | Cognitive deterioration and associated pathology induced by chronic low-level aluminum ingestion in a translational rat model provides an explanation of Alzheimer's disease, tests for susceptibility and avenues for treatment |
| Q34449486 | Decreased nuclear beta-catenin, tau hyperphosphorylation and neurodegeneration in GSK-3beta conditional transgenic mice |
| Q28276894 | Dendritic changes in Alzheimer's disease and factors that may underlie these changes |
| Q40891013 | Differential assembly of human tau isoforms in the presence of arachidonic acid |
| Q57972066 | Differential effects of apolipoprotein E isoforms on phosphorylation at specific sites on tau by glycogen synthase kinase-3β identified by nano-electrospray mass spectrometry |
| Q44441688 | Discovery and characterization of stable and toxic Tau/phospholipid oligomeric complexes |
| Q53310562 | Distribution and expression of the alpha-tubulin mRNA in the hippocampus and the temporal cortex in Alzheimer's disease. |
| Q43662734 | Functional differences of tau isoforms containing 3 or 4 C-terminal repeat regions and the influence of oxidative stress |
| Q37010838 | Histopathological and molecular heterogeneity among individuals with dementia associated with Presenilin mutations |
| Q49129166 | Human, but not bovine, oxidized cerebral spinal fluid lipoproteins disrupt neuronal microtubules |
| Q42555418 | Hyperphosphorylation-induced tau oligomers |
| Q34314365 | Hypothesis: microtubules, a key to Alzheimer disease |
| Q45969642 | Increased association between rough endoplasmic reticulum membranes and mitochondria in transgenic mice that express P301L tau. |
| Q44833066 | Inflammatory components in human Alzheimer's disease and after active amyloid-β42 immunization |
| Q35870566 | Interaction of tau protein with model lipid membranes induces tau structural compaction and membrane disruption |
| Q48163824 | Isoform transition from four-repeat to three-repeat tau underlies dendrosomatic and regional progression of neurofibrillary pathology |
| Q28760169 | Isolation and chemical characterization of Alzheimer's disease paired helical filament cytoskeletons: differentiation from amyloid plaque core protein |
| Q33859830 | Mechanisms of tau-induced neurodegeneration |
| Q48159838 | Microtubule destruction induces tau liberation and its subsequent phosphorylation |
| Q47689049 | Muscarinic therapies in Alzheimer's disease; from palliative treatments to disease modification |
| Q91645039 | NLRP3 inflammasome as a novel therapeutic target for Alzheimer's disease |
| Q53296687 | New biochemical insights to unravel the pathogenesis of Alzheimer's lesions. |
| Q30481896 | Nucleation-dependent tau filament formation: the importance of dimerization and an estimation of elementary rate constants |
| Q26749061 | Overview of Alzheimer's Disease and Some Therapeutic Approaches Targeting Aβ by Using Several Synthetic and Herbal Compounds |
| Q48615183 | Phospholipids alter tau conformation, phosphorylation, proteolysis, and association with microtubules: implication for tau function under normal and degenerative conditions |
| Q42028884 | Phosphorylation of tau by glycogen synthase kinase 3beta affects the ability of tau to promote microtubule self-assembly |
| Q41333041 | Posttranslational modifications of α-tubulin in alzheimer disease |
| Q36481949 | Presenilin-1 280Glu-->Ala mutation alters C-terminal APP processing yielding longer abeta peptides: implications for Alzheimer's disease |
| Q31111749 | Pseudophosphorylation and glycation of tau protein enhance but do not trigger fibrillization in vitro |
| Q45711453 | Role of glycosylation in hyperphosphorylation of tau in Alzheimer's disease |
| Q37087223 | Small-molecule mediated neuroprotection in an in situ model of tauopathy |
| Q48590331 | Specific monoclonal antibodies against normal microtubule-associated protein-2 (MAP2) epitopes present in Alzheimer pathological structures do not recognize paired helical filaments |
| Q34505647 | Strategies for diminishing katanin-based loss of microtubules in tauopathic neurodegenerative diseases |
| Q42157648 | Structural transitions in tau k18 on micelle binding suggest a hierarchy in the efficacy of individual microtubule-binding repeats in filament nucleation |
| Q37349368 | Structure-activity relationship of cyanine tau aggregation inhibitors. |
| Q37199229 | Subjects harboring presenilin familial Alzheimer's disease mutations exhibit diverse white matter biochemistry alterations |
| Q28077671 | Synaptopathies: synaptic dysfunction in neurological disorders - A review from students to students |
| Q54395401 | Synergistic action of polyanionic and non-polar cofactors in fibrillation of human islet amyloid polypeptide. |
| Q27004724 | Tangles, Toxicity, and Tau Secretion in AD - New Approaches to a Vexing Problem |
| Q40146754 | Tau aggregation and toxicity in a cell culture model of tauopathy |
| Q36970350 | Tau as a therapeutic target in neurodegenerative disease |
| Q34203739 | Tau binds to lipid membrane surfaces via short amphipathic helices located in its microtubule-binding repeats |
| Q34953869 | Tau in Alzheimer disease and related tauopathies |
| Q41868884 | Tau in Alzheimer's disease and Down's syndrome is insoluble and abnormally phosphorylated |
| Q37781878 | The elimination of accumulated and aggregated proteins: a role for aggrephagy in neurodegeneration |
| Q52535194 | The lipid peroxidation product 4-hydroxynonenal inhibits neurite outgrowth, disrupts neuronal microtubules, and modifies cellular tubulin. |
| Q64287035 | The role of annealing and fragmentation in human tau aggregation dynamics |
| Q33880243 | The role of the lipid bilayer in tau aggregation |
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