| scholarly article | Q13442814 |
| P50 | author | Cheng-Xin Gong | Q76062202 |
| P2093 | author name string | Fei Liu | |
| Khalid Iqbal | |||
| P2860 | cites work | Dephosphorylation of Alzheimer paired helical filaments by protein phosphatase-2A and -2B. | Q52209534 |
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| Accumulation of aspartic acid421- and glutamic acid391-cleaved tau in neurofibrillary tangles correlates with progression in Alzheimer disease | Q36426239 | ||
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| Tau exon 10 alternative splicing and tauopathies | Q36789895 | ||
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| The cellular prion protein traps Alzheimer's Aβ in an oligomeric form and disassembles amyloid fibers | Q37157714 | ||
| Physiological regulation of tau phosphorylation during hibernation | Q37229319 | ||
| Misfolded tau protein and disease modifying pathways in transgenic rodent models of human tauopathies | Q37399956 | ||
| The intersections between O-GlcNAcylation and phosphorylation: implications for multiple signaling pathways | Q37479201 | ||
| Proteolytic processing of tau. | Q37775653 | ||
| Tau gene mutation K257T causes a tauopathy similar to Pick's disease | Q38442790 | ||
| Disruption of microtubule network by Alzheimer abnormally hyperphosphorylated tau. | Q38686402 | ||
| Granular tau oligomers as intermediates of tau filaments | Q39202279 | ||
| Increased dosage of Dyrk1A alters alternative splicing factor (ASF)-regulated alternative splicing of tau in Down syndrome | Q39956895 | ||
| Molecular characterization of the minimal protease resistant tau unit of the Alzheimer's disease paired helical filament | Q40871850 | ||
| An experimental rat model of sporadic Alzheimer's disease and rescue of cognitive impairment with a neurotrophic peptide | Q41866952 | ||
| Defective brain microtubule assembly in Alzheimer's disease | Q41893869 | ||
| GSK-3beta phosphorylation of functionally distinct tau isoforms has differential, but mild effects | Q42061829 | ||
| Analysis of tau phosphorylation and truncation in a mouse model of human tauopathy | Q42146033 | ||
| Tau is a candidate gene for chromosome 17 frontotemporal dementia | Q42456776 | ||
| The myeloid leukemia-associated protein SET is a potent inhibitor of protein phosphatase 2A | Q24310186 | ||
| Caspase cleavage of tau: linking amyloid and neurofibrillary tangles in Alzheimer's disease | Q24312611 | ||
| Purification and characterization of two putative HLA class II associated proteins: PHAPI and PHAPII | Q24315134 | ||
| Mechanism of inhibition of PP2A activity and abnormal hyperphosphorylation of tau by I2(PP2A)/SET | Q24318458 | ||
| Tau protein binds to microtubules through a flexible array of distributed weak sites | Q24323891 | ||
| Hyperphosphorylation induces self-assembly of tau into tangles of paired helical filaments/straight filaments | Q24599152 | ||
| Can, a putative oncogene associated with myeloid leukemogenesis, may be activated by fusion of its 3' half to different genes: characterization of the set gene | Q24629998 | ||
| Reduced O-GlcNAcylation links lower brain glucose metabolism and tau pathology in Alzheimer's disease | Q24656640 | ||
| Synapse loss and microglial activation precede tangles in a P301S tauopathy mouse model | Q28115206 | ||
| Accumulation of abnormally phosphorylated tau precedes the formation of neurofibrillary tangles in Alzheimer's disease | Q28242505 | ||
| Association of missense and 5'-splice-site mutations in tau with the inherited dementia FTDP-17 | Q28274687 | ||
| Phosphorylation of tau at both Thr 231 and Ser 262 is required for maximal inhibition of its binding to microtubules | Q28282209 | ||
| The microtubule-associated protein tau is extensively modified with O-linked N-acetylglucosamine | Q28296295 | ||
| Alzheimer's disease: paired helical filaments and cytomembranes | Q28301717 | ||
| Multiple isoforms of human microtubule-associated protein tau: sequences and localization in neurofibrillary tangles of Alzheimer's disease | Q28910345 | ||
| Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins | Q29547593 | ||
| Tau suppression in a neurodegenerative mouse model improves memory function | Q29615831 | ||
| Abnormal phosphorylation of the microtubule-associated protein tau (tau) in Alzheimer cytoskeletal pathology | Q29617284 | ||
| Cell death and endoplasmic reticulum stress: disease relevance and therapeutic opportunities | Q29617768 | ||
| Enrichment and site mapping of O-linked N-acetylglucosamine by a combination of chemical/enzymatic tagging, photochemical cleavage, and electron transfer dissociation mass spectrometry | Q30433713 | ||
| O-GlcNAc cycling mutants modulate proteotoxicity in Caenorhabditis elegans models of human neurodegenerative diseases | Q30528027 | ||
| Caspase-cleavage of tau is an early event in Alzheimer disease tangle pathology | Q30633722 | ||
| Rapid acquisition of beta-sheet structure in the prion protein prior to multimer formation | Q31946943 | ||
| Tau interacts with Golgi membranes and mediates their association with microtubules | Q33256814 | ||
| O-linked beta-N-acetylglucosamine (O-GlcNAc): Extensive crosstalk with phosphorylation to regulate signaling and transcription in response to nutrients and stress | Q33625265 | ||
| Anesthesia induces phosphorylation of tau | Q33691128 | ||
| Identification of O-GlcNAc sites within peptides of the Tau protein and their impact on phosphorylation | Q33822354 | ||
| O-GlcNAcylation regulates phosphorylation of tau: a mechanism involved in Alzheimer's disease | Q34333004 | ||
| Phosphoprotein phosphatase activities in Alzheimer disease brain | Q34356873 | ||
| Cellular tau pathology and immunohistochemical study of tau isoforms in sporadic tauopathies | Q42503599 | ||
| Regulation of phosphorylation of tau by protein kinases in rat brain | Q42504485 | ||
| Inhibitors of protein phosphatase-2A from human brain structures, immunocytological localization and activities towards dephosphorylation of the Alzheimer type hyperphosphorylated tau. | Q42819489 | ||
| Tauopathy in Drosophila: neurodegeneration without neurofibrillary tangles | Q43642001 | ||
| Decrease in levels and rates of synthesis of tubulin and actin in developing rat brain | Q43690915 | ||
| Interaction of tau isoforms with Alzheimer's disease abnormally hyperphosphorylated tau and in vitro phosphorylation into the disease-like protein | Q43701640 | ||
| Mapping O-GlcNAc modification sites on tau and generation of a site-specific O-GlcNAc tau antibody | Q43713127 | ||
| Promotion of hyperphosphorylation by frontotemporal dementia tau mutations | Q44931294 | ||
| Inhibitors of protein phosphatase-2A: topography and subcellular localization | Q44974814 | ||
| Early N-terminal changes and caspase-6 cleavage of tau in Alzheimer's disease. | Q45050796 | ||
| Glycogen synthase kinase 3 beta induces caspase-cleaved tau aggregation in situ | Q45115056 | ||
| Tau truncation during neurofibrillary tangle evolution in Alzheimer's disease. | Q45302891 | ||
| Increased association between rough endoplasmic reticulum membranes and mitochondria in transgenic mice that express P301L tau. | Q45969642 | ||
| Reduction of soluble Abeta and tau, but not soluble Abeta alone, ameliorates cognitive decline in transgenic mice with plaques and tangles | Q46332461 | ||
| O-GlcNAcylation modulates the self-aggregation ability of the fourth microtubule-binding repeat of tau. | Q46451411 | ||
| Contributions of protein phosphatases PP1, PP2A, PP2B and PP5 to the regulation of tau phosphorylation | Q46785508 | ||
| The potential for beta-structure in the repeat domain of tau protein determines aggregation, synaptic decay, neuronal loss, and coassembly with endogenous Tau in inducible mouse models of tauopathy. | Q46804886 | ||
| Ultrastructural neuronal pathology in transgenic mice expressing mutant (P301L) human tau. | Q47878702 | ||
| Reversible paired helical filament-like phosphorylation of tau is an adaptive process associated with neuronal plasticity in hibernating animals. | Q48232761 | ||
| Anesthesia leads to tau hyperphosphorylation through inhibition of phosphatase activity by hypothermia. | Q48232865 | ||
| The DeltaK280 mutation in MAP tau favors exon 10 skipping in vivo | Q48318340 | ||
| Mutations in the tau gene that cause an increase in three repeat tau and frontotemporal dementia | Q48375000 | ||
| Relation of hippocampal phospho-SAPK/JNK granules in Alzheimer's disease and tauopathies to granulovacuolar degeneration bodies | Q48377189 | ||
| Brain levels of microtubule-associated protein tau are elevated in Alzheimer's disease: a radioimmuno-slot-blot assay for nanograms of the protein | Q48446922 | ||
| Truncated tau from sporadic Alzheimer's disease suffices to drive neurofibrillary degeneration in vivo. | Q48510520 | ||
| Concurrent alterations of O-GlcNAcylation and phosphorylation of tau in mouse brains during fasting | Q48573957 | ||
| Hereditary Pick's disease with the G272V tau mutation shows predominant three-repeat tau pathology | Q48805535 | ||
| Glycosylation of microtubule-associated protein tau: an abnormal posttranslational modification in Alzheimer's disease | Q48959233 | ||
| Non-proline-dependent protein kinases phosphorylate several sites found in tau from Alzheimer disease brain | Q49160674 | ||
| Alzheimer's disease hyperphosphorylated tau sequesters normal tau into tangles of filaments and disassembles microtubules. | Q52200995 | ||
| Restoration of biological activity of Alzheimer abnormally phosphorylated tau by dephosphorylation with protein phosphatase-2A, -2B and -1. | Q52201253 | ||
| Phosphatase activity toward abnormally phosphorylated tau: decrease in Alzheimer disease brain. | Q52206922 | ||
| P304 | page(s) | 112 | |
| P577 | publication date | 2013-08-15 | |
| P1433 | published in | Frontiers in Neurology | Q15817039 |
| P1476 | title | Hyperphosphorylation-induced tau oligomers | |
| P478 | volume | 4 |
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