| scholarly article | Q13442814 |
| P50 | author | Karen L Posey | Q94454392 |
| Jacqueline T. Hecht | Q17254912 | ||
| P2093 | author name string | Jack Lawler | |
| Paul Bornstein | |||
| Alka C. Veerisetty | |||
| Kurt Hankenson | |||
| P2860 | cites work | Collagen IX is indispensable for timely maturation of cartilage during fracture repair in mice | Q79368430 |
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| Effects of THBS3, SPARC and SPP1 expression on biological behavior and survival in patients with osteosarcoma | Q21260421 | ||
| Mutations in cartilage oligomeric matrix protein causing pseudoachondroplasia and multiple epiphyseal dysplasia affect binding of calcium and collagen I, II, and IX | Q24290529 | ||
| Cartilage oligomeric matrix protein is involved in human limb development and in the pathogenesis of osteoarthritis | Q24307967 | ||
| Cartilage oligomeric matrix protein/thrombospondin 5 supports chondrocyte attachment through interaction with integrins | Q24310801 | ||
| Interaction of cartilage oligomeric matrix protein/thrombospondin 5 with aggrecan | Q24313450 | ||
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| Interactions between the cartilage oligomeric matrix protein and matrilins. Implications for matrix assembly and the pathogenesis of chondrodysplasias | Q24336241 | ||
| Cartilage oligomeric matrix protein-deficient mice have normal skeletal development | Q24537474 | ||
| Diverse mutations in the gene for cartilage oligomeric matrix protein in the pseudoachondroplasia-multiple epiphyseal dysplasia disease spectrum | Q24538707 | ||
| Pseudoachondroplasia and multiple epiphyseal dysplasia: New etiologic developments | Q28206799 | ||
| Pseudoachondroplasia and multiple epiphyseal dysplasia: mutation review, molecular interactions, and genotype to phenotype correlations | Q28215523 | ||
| Characterization of cartilage oligomeric matrix protein (COMP) in human normal and pseudoachondroplasia musculoskeletal tissues | Q28283475 | ||
| MED, COMP, multilayered and NEIN: an overview of multiple epiphyseal dysplasia | Q28289481 | ||
| Mutations in exon 17B of cartilage oligomeric matrix protein (COMP) cause pseudoachondroplasia | Q28297153 | ||
| Beta1 integrins regulate chondrocyte rotation, G1 progression, and cytokinesis | Q28508685 | ||
| Mice lacking alpha 1 (IX) collagen develop noninflammatory degenerative joint disease | Q28509200 | ||
| Mice with a disruption of the thrombospondin 3 gene differ in geometric and biomechanical properties of bone and have accelerated development of the femoral head | Q28513961 | ||
| Cartilage oligomeric matrix protein associates with granulin-epithelin precursor (GEP) and potentiates GEP-stimulated chondrocyte proliferation | Q28569341 | ||
| Functional knockout of the matrilin-3 gene causes premature chondrocyte maturation to hypertrophy and increases bone mineral density and osteoarthritis | Q28586673 | ||
| Serum levels of cartilage oligomeric matrix protein (COMP) increase temporarily after physical exercise in patients with knee osteoarthritis | Q33265934 | ||
| Release of cartilage oligomeric matrix protein (COMP) into joint fluid after knee injury and in osteoarthritis | Q33566010 | ||
| The human thrombospondin 3 gene: analysis of transcription initiation and an alternatively spliced transcript | Q33877613 | ||
| Mutations in the region encoding the von Willebrand factor A domain of matrilin-3 are associated with multiple epiphyseal dysplasia. | Q34085472 | ||
| COMP mutations, chondrocyte function and cartilage matrix | Q34391274 | ||
| Large scale association analysis for identification of genes underlying premature coronary heart disease: cumulative perspective from analysis of 111 candidate genes | Q35053963 | ||
| Unique matrix structure in the rough endoplasmic reticulum cisternae of pseudoachondroplasia chondrocytes | Q35570417 | ||
| Role of TSP-5/COMP in pseudoachondroplasia | Q35749712 | ||
| The lack of thrombospondin-1 (TSP1) dictates the course of wound healing in double-TSP1/TSP2-null mice | Q35789152 | ||
| The matrilins--adaptor proteins in the extracellular matrix | Q36156037 | ||
| Osteoarthritis associated with mild chondrodysplasia in transgenic mice expressing alpha 1(IX) collagen chains with a central deletion | Q36204092 | ||
| Biochemical and metabolic abnormalities in articular cartilage from osteo-arthritic human hips. II. Correlation of morphology with biochemical and metabolic data | Q36564192 | ||
| Pseudoachondroplasia is caused through both intra- and extracellular pathogenic pathways | Q39735942 | ||
| Thrombospondin-1 is required for normal murine pulmonary homeostasis and its absence causes pneumonia | Q39799595 | ||
| Altered integration of matrilin-3 into cartilage extracellular matrix in the absence of collagen IX. | Q39891469 | ||
| Expression of mutant cartilage oligomeric matrix protein in human chondrocytes induces the pseudoachondroplasia phenotype | Q40346064 | ||
| In vivo human Cartilage oligomeric matrix protein (COMP) promoter activity | Q40363876 | ||
| Tissue distribution and measurement of cartilage oligomeric matrix protein in patients with magnetic resonance imaging-detected bone bruises after acute anterior cruciate ligament tears | Q43716632 | ||
| Chondrocyte cell death and intracellular distribution of COMP and type IX collagen in the pseudoachondroplasia growth plate | Q47380590 | ||
| Reduced expression of thrombospondins and craniofacial dysmorphism in mice overexpressing Fra1. | Q50737986 | ||
| Cartilage oligomeric matrix protein interacts with type IX collagen, and disruptions to these interactions identify a pathogenetic mechanism in a bone dysplasia family. | Q52162571 | ||
| Increased serum levels of non-collagenous matrix proteins (cartilage oligomeric matrix protein and melanoma inhibitory activity) in marathon runners | Q61829685 | ||
| Natural history study of pseudoachondroplasia | Q71308106 | ||
| Increased marrow-derived osteoprogenitor cells and endosteal bone formation in mice lacking thrombospondin 2 | Q73770297 | ||
| Small fragments of cartilage oligomeric matrix protein in synovial fluid and serum as markers for cartilage degradation | Q73949769 | ||
| Calreticulin, PDI, Grp94 and BiP chaperone proteins are associated with retained COMP in pseudoachondroplasia chondrocytes | Q74257251 | ||
| Physiological and pathological secretion of cartilage oligomeric matrix protein by cells in culture | Q77359804 | ||
| Retention of cartilage oligomeric matrix protein (COMP) and cell death in redifferentiated pseudoachondroplasia chondrocytes | Q77922278 | ||
| P433 | issue | 6 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | Cartilage oligomeric matrix protein | Q15320351 |
| Collagen, type IX, alpha 1 | Q21499291 | ||
| Thrombospondin-1 | Q21992233 | ||
| Thrombospondin 3 | Q21992234 | ||
| P304 | page(s) | 1664–1674 | |
| P577 | publication date | 2008-06-01 | |
| P1433 | published in | The American Journal of Pathology | Q4744259 |
| P1476 | title | Skeletal abnormalities in mice lacking extracellular matrix proteins, thrombospondin-1, thrombospondin-3, thrombospondin-5, and type IX collagen | |
| P478 | volume | 172 |
| Q36713354 | Alteration of proteoglycan sulfation affects bone growth and remodeling |
| Q41770377 | An inducible cartilage oligomeric matrix protein mouse model recapitulates human pseudoachondroplasia phenotype |
| Q38007565 | Antiangiogenic and anticancer molecules in cartilage |
| Q28648075 | Antioxidant and anti-inflammatory agents mitigate pathology in a mouse model of pseudoachondroplasia |
| Q50322999 | Antisense Reduction of Mutant COMP Reduces Growth Plate Chondrocyte Pathology |
| Q41905281 | Chop (Ddit3) is essential for D469del-COMP retention and cell death in chondrocytes in an inducible transgenic mouse model of pseudoachondroplasia |
| Q41942487 | Comparative analysis with collagen type II distinguishes cartilage oligomeric matrix protein as a primary TGFβ-responsive gene |
| Q36832626 | Comparative proteomic analysis of normal and collagen IX null mouse cartilage reveals altered extracellular matrix composition and novel components of the collagen IX interactome. |
| Q33883058 | Comprehensive profiling of cartilage extracellular matrix formation and maturation using sequential extraction and label-free quantitative proteomics |
| Q37601200 | Disorders of the growth plate |
| Q24630445 | Emerging functions of matricellular proteins |
| Q35604546 | Enhanced activity of transforming growth factor β1 (TGF-β1) bound to cartilage oligomeric matrix protein |
| Q37991256 | Evidence for FGF23 involvement in a bone-kidney axis regulating bone mineralization and systemic phosphate and vitamin D homeostasis. |
| Q36430269 | Fine mapping a locus controlling leg morphology in the domestic dog |
| Q40493272 | Histomorphometric Parameters of the Growth Plate and Trabecular Bone in Wild-Type and Trefoil Factor Family 3 (Tff3)-Deficient Mice Analyzed by Free and Open-Source Image Processing Software. |
| Q58799604 | Hydra Mesoglea Proteome Identifies Thrombospondin as a Conserved Component Active in Head Organizer Restriction |
| Q37410009 | Hypothalamic dysfunction of the thrombospondin receptor α2δ-1 underlies the overeating and obesity triggered by brain-derived neurotrophic factor deficiency. |
| Q33597026 | Insights into shell deposition in the Antarctic bivalve Laternula elliptica: gene discovery in the mantle transcriptome using 454 pyrosequencing. |
| Q35849106 | Intermolecular interactions of thrombospondins drive their accumulation in extracellular matrix. |
| Q35542423 | Matricellular proteins in cardiac adaptation and disease |
| Q37332408 | Novel regulators of Fgf23 expression and mineralization in Hyp bone |
| Q39197382 | Novel therapeutic interventions for pseudoachondroplasia |
| Q92476218 | Patterns of spon1b:GFP expression during early zebrafish brain development |
| Q27312155 | Positional cloning of zinc finger domain transcription factor Zfp69, a candidate gene for obesity-associated diabetes contributed by mouse locus Nidd/SJL |
| Q34407214 | Pseudoachondroplasia/COMP - translating from the bench to the bedside |
| Q33565464 | RNAi reduces expression and intracellular retention of mutant cartilage oligomeric matrix protein |
| Q38656989 | Regulation of cellular redox signaling by matricellular proteins in vascular biology, immunology, and cancer |
| Q58777324 | TRIP-1 Promotes the Assembly of an ECM That Contains Extracellular Vesicles and Factors That Modulate Angiogenesis |
| Q27654058 | The crystal structure of the signature domain of cartilage oligomeric matrix protein: implications for collagen, glycosaminoglycan and integrin binding |
| Q28744006 | The evolution of thrombospondins and their ligand-binding activities |
| Q37424208 | The interaction of Thrombospondins with extracellular matrix proteins |
| Q35230013 | The thrombospondins. |
| Q47149542 | Thrombospondin-1 Partly Mediates the Cartilage Protective Effect of Adipose-Derived Mesenchymal Stem Cells in Osteoarthritis. |
| Q35493987 | Thrombospondin-1 regulates bone homeostasis through effects on bone matrix integrity and nitric oxide signaling in osteoclasts |
| Q37424213 | Thrombospondin-2 and SPARC/osteonectin are critical regulators of bone remodeling |
| Q60917607 | Thrombospondin-3 augments injury-induced cardiomyopathy by intracellular integrin inhibition and sarcolemmal instability |
| Q37738777 | Thrombospondins and novel TSR-containing proteins, R-spondins, regulate bone formation and remodeling |
| Q33964749 | Type IX collagen interacts with fibronectin providing an important molecular bridge in articular cartilage. |
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