| scholarly article | Q13442814 |
| P50 | author | Ping Wang | Q90460136 |
| P2093 | author name string | Rongqian Wu | |
| Fangming Zhang | |||
| Mian Zhou | |||
| Xiaoling Qiang | |||
| P2860 | cites work | Physiology and immunology of the cholinergic antiinflammatory pathway | Q24681833 |
| Epidemiology of severe sepsis in the United States: analysis of incidence, outcome, and associated costs of care | Q27860820 | ||
| The sympathetic nerve--an integrative interface between two supersystems: the brain and the immune system | Q28140631 | ||
| The epidemiology of sepsis in the United States from 1979 through 2000 | Q29547760 | ||
| Efficacy and safety of recombinant human activated protein C for severe sepsis | Q29547839 | ||
| Chemokines--chemotactic cytokines that mediate inflammation | Q29616433 | ||
| The pathophysiology and treatment of sepsis | Q29619103 | ||
| Ghrelin inhibits sympathetic nervous activity in sepsis. | Q30968697 | ||
| The inflammatory reflex | Q34165907 | ||
| Drotrecogin alfa (activated): does current evidence support treatment for any patients with severe sepsis? | Q34568205 | ||
| Nonsynaptic noradrenaline release in neuro-immune responses | Q34685868 | ||
| Novel strategies for the treatment of sepsis | Q35118750 | ||
| Role of inflammatory mediators in the pathophysiology of acute respiratory distress syndrome | Q35638490 | ||
| Ghrelin attenuates sepsis-induced acute lung injury and mortality in rats | Q36063608 | ||
| Sympathetic excitotoxicity in sepsis: pro-inflammatory priming of macrophages by norepinephrine. | Q36170206 | ||
| Clinical implications of cytokines in the critical-care unit | Q37546161 | ||
| Cardiac dysfunction in severe sepsis and septic shock | Q37562775 | ||
| Sepsis and septic shock--a review of laboratory models and a proposal | Q40290200 | ||
| Mechanism of hepatocellular dysfunction during hyperdynamic sepsis | Q41224068 | ||
| Regulation of tumor necrosis factor production by adrenaline and beta-adrenergic agonists | Q41622626 | ||
| Mechanism of hepatocellular dysfunction during early sepsis. Key role of increased gene expression and release of proinflammatory cytokines tumor necrosis factor and interleukin-6. | Q43662373 | ||
| The role of Kupffer cell alpha(2)-adrenoceptors in norepinephrine-induced TNF-alpha production | Q43690393 | ||
| Norepinephrine-induced hepatocellular dysfunction in early sepsis is mediated by activation of alpha2-adrenoceptors | Q43737851 | ||
| Differential expression of cytochrome P450 isoforms in the lungs of septic animals | Q44931788 | ||
| Modulatory effects of glucocorticoids and catecholamines on human interleukin-12 and interleukin-10 production: clinical implications | Q48924980 | ||
| National study of emergency department visits for sepsis, 1992 to 2001. | Q50787890 | ||
| Adrenomedullin and Adrenomedullin Binding Protein-1 Prevent Acute Lung Injury after Gut Ischemia-Reperfusion | Q58876470 | ||
| Sustained elevation in circulating catecholamine levels during polymicrobial sepsis | Q70907243 | ||
| Differential alterations in systemic and regional oxygen delivery and consumption during the early and late stages of sepsis | Q73099863 | ||
| Gut-derived norepinephrine plays a critical role in producing hepatocellular dysfunction during early sepsis | Q73230238 | ||
| Regulation of cytokine and chemokine production by transmitters and co-transmitters of the autonomic nervous system | Q77512461 | ||
| Kupffer cells are responsible for producing inflammatory cytokines and hepatocellular dysfunction during early sepsis | Q77753642 | ||
| Is gut the major source of proinflammatory cytokine release during polymicrobial sepsis? | Q78145079 | ||
| Seasonal variation in the epidemiology of sepsis | Q79431696 | ||
| Ghrelin down-regulates proinflammatory cytokines in sepsis through activation of the vagus nerve | Q80161124 | ||
| ST91 [2-(2,6-diethylphenylamino)-2-imidazoline hydrochloride]-mediated spinal antinociception and synergy with opioids persists in the absence of functional alpha-2A- or alpha-2C-adrenergic receptors | Q81250624 | ||
| P433 | issue | 3 | |
| P921 | main subject | inflammation | Q101991 |
| sepsis | Q183134 | ||
| P304 | page(s) | 289–296 | |
| P577 | publication date | 2010-03-01 | |
| P1433 | published in | Journal of Molecular Medicine | Q6295593 |
| P1476 | title | Antagonism of alpha2A-adrenoceptor: a novel approach to inhibit inflammatory responses in sepsis | |
| P478 | volume | 88 |
| Q36261528 | Activation of α2 adrenoceptor attenuates lipopolysaccharide-induced hepatic injury |
| Q37233638 | Advances in sepsis-associated liver dysfunction |
| Q47131337 | Altered Neuroendocrine Immune Responses, a Two-Sword Weapon against Traumatic Inflammation |
| Q38058384 | Clinical review: The liver in sepsis |
| Q41976208 | Dexmedetomidine Protects Rat Liver against Ischemia-Reperfusion Injury Partly by the α2A-Adrenoceptor Subtype and the Mechanism Is Associated with the TLR4/NF-κB Pathway. |
| Q34028610 | Divergent neuroendocrine responses to localized and systemic inflammation. |
| Q38513417 | Participation of α2 -adrenoceptors in sodium appetite inhibition during sickness behaviour following administration of lipopolysaccharide |
| Q34730803 | Yohimbine promotes cardiac NE release and prevents LPS-induced cardiac dysfunction via blockade of presynaptic α2A-adrenergic receptor |
| Q55339088 | α2A-adrenergic blockade attenuates septic cardiomyopathy by increasing cardiac norepinephrine concentration and inhibiting cardiac endothelial activation. |
Search more.