scholarly article | Q13442814 |
P50 | author | Wenting Wang | Q60331741 |
Sheng-Xi Wu | Q81472605 | ||
Baolin Guo | Q92093871 | ||
Marie-Sophie van der Goes | Q96096679 | ||
P2093 | author name string | Guoping Feng | |
Qian Chen | |||
Chenchen Li | |||
Ying Zang | |||
Annie Y Yao | |||
Charles R Gerfen | |||
Congyi Lu | |||
Dongqing Wang | |||
James Hawrot | |||
Katherine Lyman | |||
Qiangge Zhang | |||
Xian Gao | |||
Zhanyan Fu | |||
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Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice | Q21198691 | ||
Haploinsufficiency of the autism-associated Shank3 gene leads to deficits in synaptic function, social interaction, and social communication | Q21198697 | ||
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Neuroligins and neurexins link synaptic function to cognitive disease | Q22251092 | ||
Mutations in the gene encoding the synaptic scaffolding protein SHANK3 are associated with autism spectrum disorders | Q24337114 | ||
Identification of a novel cortactin SH3 domain-binding protein and its localization to growth cones of cultured neurons | Q24522731 | ||
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Disruption of the ProSAP2 gene in a t(12;22)(q24.1;q13.3) is associated with the 22q13.3 deletion syndrome | Q24535749 | ||
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Identification of a recurrent breakpoint within the SHANK3 gene in the 22q13.3 deletion syndrome | Q24656220 | ||
Molecular characterisation of the 22q13 deletion syndrome supports the role of haploinsufficiency of SHANK3/PROSAP2 in the major neurological symptoms | Q24676539 | ||
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Autism-associated neuroligin-3 mutations commonly impair striatal circuits to boost repetitive behaviors | Q26269833 | ||
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Coupling of mGluR/Homer and PSD-95 complexes by the Shank family of postsynaptic density proteins | Q28141190 | ||
The Shank family of scaffold proteins | Q28144084 | ||
Proline-rich synapse-associated proteins ProSAP1 and ProSAP2 interact with synaptic proteins of the SAPAP/GKAP family | Q28145992 | ||
22q13 deletion syndrome | Q28200653 | ||
Projection subtypes of rat neostriatal matrix cells revealed by intracellular injection of biocytin | Q28264640 | ||
Molecular and phenotypic characterization of ring chromosome 22 | Q28264937 | ||
Mice with Shank3 Mutations Associated with ASD and Schizophrenia Display Both Shared and Distinct Defects | Q28271310 | ||
Adult restoration of Shank3 expression rescues selective autistic-like phenotypes | Q28273413 | ||
L-type calcium channels: the low down | Q28288051 | ||
An architectural framework that may lie at the core of the postsynaptic density | Q28293971 | ||
Clinical utility gene card for: deletion 22q13 syndrome | Q28300606 | ||
Smaller dendritic spines, weaker synaptic transmission, but enhanced spatial learning in mice lacking Shank1. | Q28512788 | ||
Neuronal Ca(V)1.3alpha(1) L-type channels activate at relatively hyperpolarized membrane potentials and are incompletely inhibited by dihydropyridines | Q28582608 | ||
Parallel Organization of Functionally Segregated Circuits Linking Basal Ganglia and Cortex | Q29391304 | ||
The mGluR theory of fragile X mental retardation | Q29615060 | ||
A translational profiling approach for the molecular characterization of CNS cell types | Q29615252 | ||
D1 and D2 dopamine receptor-regulated gene expression of striatonigral and striatopallidal neurons | Q29615480 | ||
PDZ domain proteins of synapses | Q29616399 | ||
A gene expression atlas of the central nervous system based on bacterial artificial chromosomes | Q29617753 | ||
A synaptic trek to autism | Q29622832 | ||
Regulation of dendritic spine morphology and synaptic function by Shank and Homer | Q30014842 | ||
Altered Striatal Synaptic Function and Abnormal Behaviour in Shank3 Exon4-9 Deletion Mouse Model of Autism | Q30362074 | ||
Impaired Dendritic Development and Memory in Sorbs2 Knock-Out Mice. | Q30376404 | ||
SHANK3 mutations identified in autism lead to modification of dendritic spine morphology via an actin-dependent mechanism. | Q30428959 | ||
Balanced activity in basal ganglia projection pathways is critical for contraversive movements | Q30434712 | ||
Loss of predominant Shank3 isoforms results in hippocampus-dependent impairments in behavior and synaptic transmission | Q30438299 | ||
SHANK3 overexpression causes manic-like behaviour with unique pharmacogenetic properties | Q30438816 | ||
Reduced excitatory neurotransmission and mild autism-relevant phenotypes in adolescent Shank3 null mutant mice | Q30461096 | ||
Repeated cortico-striatal stimulation generates persistent OCD-like behavior | Q30574071 | ||
Concurrent activation of striatal direct and indirect pathways during action initiation. | Q30579307 | ||
Functional neuroimaging and the neuroanatomy of obsessive-compulsive disorder | Q33181070 | ||
Ca(2+) permeable AMPA receptor induced long-term potentiation requires PI3/MAP kinases but not Ca/CaM-dependent kinase II. | Q33405238 | ||
Generation of designer receptors exclusively activated by designer drugs (DREADDs) using directed molecular evolution | Q33523314 | ||
Distinct roles of GABAergic interneurons in the regulation of striatal output pathways | Q33720464 | ||
Inhibitory control of neostriatal projection neurons by GABAergic interneurons | Q33861738 | ||
Activation of group I mGluRs is necessary for induction of long-term depression at striatal synapses | Q34100802 | ||
Insights into Autism Spectrum Disorder Genomic Architecture and Biology from 71 Risk Loci | Q34495555 | ||
Dopaminergic control of corticostriatal long-term synaptic depression in medium spiny neurons is mediated by cholinergic interneurons. | Q34521936 | ||
Retro-orbital injections in mice. | Q35171751 | ||
Mutations causing syndromic autism define an axis of synaptic pathophysiology | Q35587150 | ||
Dynamic aspects of CNS synapse formation. | Q35652924 | ||
Metabotropic glutamate receptor-dependent long-term depression is impaired due to elevated ERK signaling in the ΔRG mouse model of tuberous sclerosis complex | Q35748464 | ||
MAGUKs in synapse assembly and function: an emerging view | Q35750293 | ||
Neuroligin 1 modulates striatal glutamatergic neurotransmission in a pathway and NMDAR subunit-specific manner | Q35898012 | ||
Decreased probability of neurotransmitter release underlies striatal long-term depression and postnatal development of corticostriatal synapses | Q36054269 | ||
Learning From Animal Models of Obsessive-Compulsive Disorder | Q36249163 | ||
Identification and differential subcellular localization of the neuronal class C and class D L-type calcium channel alpha 1 subunits | Q36383289 | ||
The role of the GluR2 subunit in AMPA receptor function and synaptic plasticity. | Q36854587 | ||
Prospective investigation of autism and genotype-phenotype correlations in 22q13 deletion syndrome and SHANK3 deficiency | Q37001013 | ||
Direct-pathway striatal neurons regulate the retention of decision-making strategies | Q37048139 | ||
Scaffolding proteins at the postsynaptic density: shank as the architectural framework | Q37168233 | ||
Habits, rituals, and the evaluative brain | Q37193050 | ||
Neurotransmitter roles in synaptic modulation, plasticity and learning in the dorsal striatum | Q37680104 | ||
Modeling autism by SHANK gene mutations in mice | Q38098737 | ||
Striatal circuits, habits, and implications for obsessive-compulsive disorder. | Q38252883 | ||
Striatal direct and indirect pathways control decision-making behavior. | Q38271951 | ||
Distinct roles for direct and indirect pathway striatal neurons in reinforcement. | Q41894840 | ||
Autism-like Deficits in Shank3-Deficient Mice Are Rescued by Targeting Actin Regulators | Q42018247 | ||
Long-term synaptic depression in the striatum: physiological and pharmacological characterization | Q42453585 | ||
Coordinated Regulation of Synaptic Plasticity at Striatopallidal and Striatonigral Neurons Orchestrates Motor Control | Q42484851 | ||
Endocannabinoid-mediated rescue of striatal LTD and motor deficits in Parkinson's disease models | Q42508054 | ||
Drd1a-tdTomato BAC transgenic mice for simultaneous visualization of medium spiny neurons in the direct and indirect pathways of the basal ganglia. | Q42525723 | ||
Application of a translational profiling approach for the comparative analysis of CNS cell types | Q42585641 | ||
The pathophysiology of restricted repetitive behavior | Q42729995 | ||
Novel de novo SHANK3 mutation in autistic patients | Q43768017 | ||
Postsynaptic endocannabinoid release is critical to long-term depression in the striatum | Q43971441 | ||
Selective alleviation of compulsive lever-pressing in rats by D1, but not D2, blockade: possible implications for the involvement of D1 receptors in obsessive-compulsive disorder | Q44259226 | ||
Selective blockade of spontaneous motor stereotypy via intrastriatal pharmacological manipulation | Q44384952 | ||
G-protein-coupled receptor modulation of striatal CaV1.3 L-type Ca2+ channels is dependent on a Shank-binding domain. | Q45249950 | ||
Functional state of corticostriatal synapses determines their expression of short- and long-term plasticity | Q46255311 | ||
Dopamine modulation of state-dependent endocannabinoid release and long-term depression in the striatum. | Q46279749 | ||
Shank expression is sufficient to induce functional dendritic spine synapses in aspiny neurons. | Q46425942 | ||
Combined activation of L-type Ca2+ channels and synaptic transmission is sufficient to induce striatal long-term depression. | Q48130770 | ||
An MRI study of the basal ganglia in autism | Q48173058 | ||
OCD-Like behaviors caused by a neuropotentiating transgene targeted to cortical and limbic D1+ neurons | Q48188537 | ||
A functional magnetic resonance imaging study of tic suppression in Tourette syndrome | Q48485554 | ||
P433 | issue | 5 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | autism | Q38404 |
P304 | page(s) | 1978-1990 | |
P577 | publication date | 2017-04-17 | |
P1433 | published in | Journal of Clinical Investigation | Q3186904 |
P1476 | title | Striatopallidal dysfunction underlies repetitive behavior in Shank3-deficient model of autism | |
P478 | volume | 127 |