| scholarly article | Q13442814 |
| P356 | DOI | 10.1073/PNAS.1612441113 |
| P8608 | Fatcat ID | release_pj4qnvhdrrea3pgbl32gz7iuly |
| P932 | PMC publication ID | 5047181 |
| P698 | PubMed publication ID | 27621462 |
| P2093 | author name string | Eric N Olson | |
| Alexander Polster | |||
| Benjamin R Nelson | |||
| Kurt G Beam | |||
| P2860 | cites work | Stac3 is a component of the excitation-contraction coupling machinery and mutated in Native American myopathy | Q24294570 |
| Absence of the beta subunit (cchb1) of the skeletal muscle dihydropyridine receptor alters expression of the alpha 1 subunit and eliminates excitation-contraction coupling | Q24680364 | ||
| Modulation of L-type Ca2+ current but not activation of Ca2+ release by the gamma1 subunit of the dihydropyridine receptor of skeletal muscle | Q24802046 | ||
| Excitation-contraction coupling in skeletal muscle of a mouse lacking the dihydropyridine receptor subunit gamma1 | Q28366204 | ||
| Absence of the gamma subunit of the skeletal muscle dihydropyridine receptor increases L-type Ca2+ currents and alters channel inactivation properties | Q28509060 | ||
| Excitation-contraction uncoupling and muscular degeneration in mice lacking functional skeletal muscle ryanodine-receptor gene | Q28587644 | ||
| Skeletal muscle-specific T-tubule protein STAC3 mediates voltage-induced Ca2+ release and contractility | Q28588104 | ||
| The Ca2+ channel alpha2delta-1 subunit determines Ca2+ current kinetics in skeletal muscle but not targeting of alpha1S or excitation-contraction coupling | Q28593153 | ||
| Non-Ca2+-conducting Ca2+ channels in fish skeletal muscle excitation-contraction coupling | Q33778038 | ||
| Reduced Ca2+ current, charge movement, and absence of Ca2+ transients in skeletal muscle deficient in dihydropyridine receptor beta 1 subunit | Q34041027 | ||
| Differential regulation of skeletal muscle L-type Ca2+ current and excitation-contraction coupling by the dihydropyridine receptor beta subunit | Q34170119 | ||
| Involvement of a heptad repeat in the carboxyl terminus of the dihydropyridine receptor beta1a subunit in the mechanism of excitation-contraction coupling in skeletal muscle | Q34186764 | ||
| Molecular organization of transverse tubule/sarcoplasmic reticulum junctions during development of excitation-contraction coupling in skeletal muscle | Q34445587 | ||
| Restoration of excitation-contraction coupling and slow calcium current in dysgenic muscle by dihydropyridine receptor complementary DNA. | Q34560693 | ||
| Stac adaptor proteins regulate trafficking and function of muscle and neuronal L-type Ca2+ channels | Q34985729 | ||
| Auxiliary subunits: essential components of the voltage-gated calcium channel complex. | Q35172799 | ||
| Functional impact of the ryanodine receptor on the skeletal muscle L-type Ca(2+) channel | Q36444962 | ||
| Alpha2delta1 dihydropyridine receptor subunit is a critical element for excitation-coupled calcium entry but not for formation of tetrads in skeletal myotubes | Q36510485 | ||
| Distinct Components of Retrograde Ca(V)1.1-RyR1 Coupling Revealed by a Lethal Mutation in RyR1. | Q36644359 | ||
| Intramembrane charge movement restored in dysgenic skeletal muscle by injection of dihydropyridine receptor cDNAs | Q41200795 | ||
| Functional and structural approaches to the study of excitation-contraction coupling | Q41649828 | ||
| Proper restoration of excitation-contraction coupling in the dihydropyridine receptor beta1-null zebrafish relaxed is an exclusive function of the beta1a subunit | Q41820912 | ||
| Structural characterization of the 1,4-dihydropyridine receptor of the voltage-dependent Ca2+ channel from rabbit skeletal muscle. Evidence for two distinct high molecular weight subunits | Q41962726 | ||
| Skeletal muscle L-type Ca(2+) current modulation in gamma1-deficient and wildtype murine myotubes by the gamma1 subunit and cAMP. | Q43908348 | ||
| Mapping sites of potential proximity between the dihydropyridine receptor and RyR1 in muscle using a cyan fluorescent protein-yellow fluorescent protein tandem as a fluorescence resonance energy transfer probe | Q44997701 | ||
| Dyspedic mouse skeletal muscle expresses major elements of the triadic junction but lacks detectable ryanodine receptor protein and function | Q45256995 | ||
| Organization of calcium channel beta1a subunits in triad junctions in skeletal muscle | Q46827134 | ||
| Enhanced dihydropyridine receptor channel activity in the presence of ryanodine receptor. | Q52519047 | ||
| Native American myopathy: congenital myopathy with cleft palate, skeletal anomalies, and susceptibility to malignant hyperthermia | Q81454581 | ||
| P433 | issue | 39 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | SH3 and cysteine rich domain 3 | Q21989583 |
| P304 | page(s) | 10986-10991 | |
| P577 | publication date | 2016-09-12 | |
| P1433 | published in | Proceedings of the National Academy of Sciences of the United States of America | Q1146531 |
| P1476 | title | Stac3 has a direct role in skeletal muscle-type excitation-contraction coupling that is disrupted by a myopathy-causing mutation | |
| P478 | volume | 113 |
| Q57752434 | Allosteric regulators selectively prevent Ca-feedback of Ca and Na channels |
| Q53839183 | Calcium-induced release of calcium in muscle: 50 years of work and the emerging consensus. |
| Q45869436 | Clinicopathologic Conference: A Newborn With Hypotonia, Cleft Palate, Micrognathia, and Bilateral Club Feet. |
| Q49388884 | Congenital myopathies: disorders of excitation-contraction coupling and muscle contraction |
| Q30008804 | Congenital myopathy results from misregulation of a muscle Ca2+ channel by mutant Stac3. |
| Q91897792 | Crystal structures of Ca2+-calmodulin bound to NaV C-terminal regions suggest role for EF-hand domain in binding and inactivation |
| Q47304950 | De novo reconstitution reveals the proteins required for skeletal muscle voltage-induced Ca2+ release |
| Q89294511 | Duplex signaling by CaM and Stac3 enhances CaV1.1 function and provides insights into congenital myopathy |
| Q60960134 | Dyadic Plasticity in Cardiomyocytes |
| Q88922983 | Evaluation of mutant muscle Ca2+ channel properties using two different expression systems |
| Q92719884 | Excitation-contraction coupling in skeletal muscle: recent progress and unanswered questions |
| Q59882254 | Genetic epidemiology of malignant hyperthermia in the UK |
| Q37672288 | How and why are calcium currents curtailed in the skeletal muscle voltage-gated calcium channels? |
| Q39052152 | Pharmacogenetics and pathophysiology of CACNA1S mutations in malignant hyperthermia |
| Q48177519 | STAC proteins associate to the IQ domain of CaV1.2 and inhibit calcium-dependent inactivation. |
| Q49950374 | STAC2 negatively regulates osteoclast formation by targeting the RANK signaling complex |
| Q57752412 | STAC3 incorporation into skeletal muscle triads occurs independent of the dihydropyridine receptor |
| Q30008782 | STAC3 stably interacts through its C1 domain with CaV1.1 in skeletal muscle triads |
| Q57752447 | STAC3 variants cause a congenital myopathy with distinctive dysmorphic features and malignant hyperthermia susceptibility |
| Q57752435 | Stac Proteins Suppress Ca-Dependent Inactivation of Neuronal L-type Ca Channels |
| Q52691686 | Stac proteins associate with the critical domain for excitation-contraction coupling in the II-III loop of CaV1.1. |
| Q48131423 | Stac3 enhances expression of human CaV1.1 in Xenopus oocytes and reveals gating pore currents in HypoPP mutant channels. |
| Q46130508 | Structural insights into binding of STAC proteins to voltage-gated calcium channels |
| Q97884777 | Trafficking of neuronal calcium channels |
| Q37678236 | Will you still need me (Ca2+ , TnT, and DHPR), will you still cleave me (calpain), when I'm 64? |
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