| scholarly article | Q13442814 |
| P50 | author | Bruno Allard | Q86188960 |
| P2860 | cites work | Channelopathies of skeletal muscle excitability | Q28082428 |
| A calcium channel mutation causing hypokalemic periodic paralysis | Q28242375 | ||
| Dihydropyridine receptor mutations cause hypokalemic periodic paralysis | Q28243593 | ||
| Hypokalemic periodic paralysis: In vitro investigation of muscle fiber membrane parameters | Q28265960 | ||
| Altered inactivation of Ca2+ current and Ca2+ release in mouse muscle fibers deficient in the DHP receptor gamma1 subunit | Q28505025 | ||
| Stac3 has a direct role in skeletal muscle-type excitation-contraction coupling that is disrupted by a myopathy-causing mutation | Q30008861 | ||
| Intramembrane charge movement and L-type calcium current in skeletal muscle fibers isolated from control and mdx mice | Q34180105 | ||
| Stac adaptor proteins regulate trafficking and function of muscle and neuronal L-type Ca2+ channels | Q34985729 | ||
| Two atomic constraints unambiguously position the S4 segment relative to S1 and S2 segments in the closed state of Shaker K channel | Q35808791 | ||
| Pathophysiological role of omega pore current in channelopathies | Q36022793 | ||
| A Na+ channel mutation linked to hypokalemic periodic paralysis exposes a proton-selective gating pore | Q36299707 | ||
| A calcium channel mutant mouse model of hypokalemic periodic paralysis | Q36498120 | ||
| K+-dependent paradoxical membrane depolarization and Na+ overload, major and reversible contributors to weakness by ion channel leaks | Q37102151 | ||
| Muscle channelopathies: does the predicted channel gating pore offer new treatment insights for hypokalaemic periodic paralysis? | Q37685726 | ||
| Voltage-sensor mutations in channelopathies of skeletal muscle | Q37692699 | ||
| Biophysics, pathophysiology, and pharmacology of ion channel gating pores | Q38207583 | ||
| Elevated resting H(+) current in the R1239H type 1 Hypokalemic Periodic Paralysis mutated Ca(2+) channel. | Q38600648 | ||
| A proton pore in a potassium channel voltage sensor reveals a focused electric field | Q44757373 | ||
| Voltage-sensing arginines in a potassium channel permeate and occlude cation-selective pores | Q45252796 | ||
| An atypical CaV1.1 mutation reveals a common mechanism for hypokalemic periodic paralysis. | Q46154188 | ||
| Ion permeation through a voltage- sensitive gating pore in brain sodium channels having voltage sensor mutations | Q46615032 | ||
| Na leak with gating pore properties in hypokalemic periodic paralysis V876E mutant muscle Ca channel. | Q47637076 | ||
| Stac3 enhances expression of human CaV1.1 in Xenopus oocytes and reveals gating pore currents in HypoPP mutant channels. | Q48131423 | ||
| When muscle Ca2+ channels carry monovalent cations through gating pores: insights into the pathophysiology of type 1 hypokalaemic periodic paralysis. | Q52343820 | ||
| Whole-cell voltage clamp on skeletal muscle fibers with the silicone-clamp technique. | Q53502303 | ||
| Insulin acts in hypokalemic periodic paralysis by reducing inward rectifier K+ current | Q73114370 | ||
| Improving the characterization of calcium channel gating pore currents with Stac3 | Q87841107 | ||
| P433 | issue | 7 | |
| P304 | page(s) | 897-899 | |
| P577 | publication date | 2018-05-30 | |
| P1433 | published in | The Journal of General Physiology | Q1092259 |
| P1476 | title | Evaluation of mutant muscle Ca2+ channel properties using two different expression systems | |
| P478 | volume | 150 |
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