| P50 | author | Huda Zoghbi | Q1633764 |
| | Aya Ito-Ishida | Q79147547 |
| P2093 | author name string | Wei Wang | |
| | Hui Lu | |
| | Jianrong Tang | |
| | Kerstin Ure | |
| | Ling-Jie He | |
| | Wu Chen | |
| | Yehezkel Sztainberg | |
| | Zhenyu Wu | |
| P2860 | cites work | Alterations of GABAergic signaling in autism spectrum disorders | Q21296802 |
| | Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2 | Q22337290 |
| | Reduced cortical activity due to a shift in the balance between excitation and inhibition in a mouse model of Rett syndrome | Q24530286 |
| | MeCP2, a key contributor to neurological disease, activates and represses transcription | Q24647533 |
| | Loss of MeCP2 in aminergic neurons causes cell-autonomous defects in neurotransmitter synthesis and specific behavioral abnormalities | Q24652603 |
| | Common circuit defect of excitatory-inhibitory balance in mouse models of autism | Q26269924 |
| | Reversal of phenotypes in MECP2 duplication mice using genetic rescue or antisense oligonucleotides | Q27314897 |
| | A mouse Mecp2-null mutation causes neurological symptoms that mimic Rett syndrome | Q28504458 |
| | MeCP2 controls excitatory synaptic strength by regulating glutamatergic synapse number | Q28507266 |
| | Model of autism: increased ratio of excitation/inhibition in key neural systems | Q29547530 |
| | Neocortical excitation/inhibition balance in information processing and social dysfunction | Q29616196 |
| | Dysfunction in GABA signalling mediates autism-like stereotypies and Rett syndrome phenotypes | Q29616326 |
| | Deficiency of methyl-CpG binding protein-2 in CNS neurons results in a Rett-like phenotype in mice | Q29616328 |
| | Reversal of neurological defects in a mouse model of Rett syndrome | Q29616452 |
| | Loss of MeCP2 in cholinergic neurons causes part of RTT-like phenotypes via α7 receptor in hippocampus | Q30355025 |
| | Loss of MeCP2 in Parvalbumin-and Somatostatin-Expressing Neurons in Mice Leads to Distinct Rett Syndrome-like Phenotypes | Q30369619 |
| | Manipulations of MeCP2 in glutamatergic neurons highlight their contributions to Rett and other neurological disorders | Q30378670 |
| | Cellular origins of auditory event-related potential deficits in Rett syndrome | Q30424295 |
| | Wild-type microglia arrest pathology in a mouse model of Rett syndrome | Q30424614 |
| | Female Mecp2(+/-) mice display robust behavioral deficits on two different genetic backgrounds providing a framework for pre-clinical studies | Q30445073 |
| | Glutamate co-release at GABA/glycinergic synapses is crucial for the refinement of an inhibitory map. | Q30493499 |
| | Postnatal NMDA receptor ablation in corticolimbic interneurons confers schizophrenia-like phenotypes | Q33556077 |
| | X-chromosome inactivation patterns are unbalanced and affect the phenotypic outcome in a mouse model of rett syndrome | Q33911020 |
| | Equalizing excitation-inhibition ratios across visual cortical neurons | Q33983822 |
| | A computational model for cerebral cortical dysfunction in autism spectrum disorders | Q35014681 |
| | A role for glia in the progression of Rett's syndrome | Q35712588 |
| | Role of glial amino acid transporters in synaptic transmission and brain energetics | Q35836052 |
| | Wild-type microglia do not reverse pathology in mouse models of Rett syndrome | Q36386922 |
| | An AT-hook domain in MeCP2 determines the clinical course of Rett syndrome and related disorders | Q36810668 |
| | Single rodent mesohabenular axons release glutamate and GABA | Q36832519 |
| | Deletion of Mecp2 in Sim1-expressing neurons reveals a critical role for MeCP2 in feeding behavior, aggression, and the response to stress. | Q37001615 |
| | Mouse models of MeCP2 disorders share gene expression changes in the cerebellum and hypothalamus | Q37223275 |
| | Cocaine exposure in utero alters synaptic plasticity in the medial prefrontal cortex of postnatal rats | Q37361940 |
| | Neuroimaging and neurogenetics of epilepsy in humans | Q37590504 |
| | The clinical recognition and differential diagnosis of Rett syndrome | Q39545713 |
| | Mood regulation. GABA/glutamate co-release controls habenula output and is modified by antidepressant treatment. | Q42465895 |
| | GABAergic hub neurons orchestrate synchrony in developing hippocampal networks | Q43228823 |
| | GABAergic signaling at mossy fiber synapses in neonatal rat hippocampus. | Q46890616 |
| | Conditional deletion of Mecp2 in parvalbumin-expressing GABAergic cells results in the absence of critical period plasticity | Q47802651 |
| | Evidence against GABA release from glutamatergic mossy fiber terminals in the developing hippocampus. | Q48095807 |
| | Genetic ablation of VIAAT in glycinergic neurons causes a severe respiratory phenotype and perinatal death. | Q48658091 |
| | Amino acids: analytical aspects | Q74017457 |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | Rett syndrome | Q917357 |
| P577 | publication date | 2016-06-21 | |
| P1433 | published in | eLife | Q2000008 |
| P1476 | title | Restoration of Mecp2 expression in GABAergic neurons is sufficient to rescue multiple disease features in a mouse model of Rett syndrome. | |
| P478 | volume | 5 | |