Distribution of viral RNA, sialic acid receptor, and pathology in H5N1 avian influenza patients.

scientific article

Distribution of viral RNA, sialic acid receptor, and pathology in H5N1 avian influenza patients. is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1111/J.1600-0463.2010.02676.X
P698PubMed publication ID20955463

P50authorPrasert AuewarakulQ43156881
P2093author name stringPilaipan Puthavathana
Ornpreya Suptawiwat
Yuwarat Monteerarat
Yotthachai Piwpankaew
Mongkol Uipresertkul
P2860cites workFatal avian influenza A H5N1 in a dogQ24606768
Avian influenza H5N1 in naturally infected domestic catQ28218088
Probable tiger-to-tiger transmission of avian influenza H5N1.Q35873055
Avian influenza H5N1 in tigers and leopardsQ35881097
Influenza A H5N1 replication sites in humansQ36022034
Apoptosis and pathogenesis of avian influenza A (H5N1) virus in humansQ37334291
H5N1 transmission and disease: observations from the frontlinesQ38965703
P433issue11
P921main subjectpathologyQ7208
avian influenzaQ43987
P304page(s)895-902
P577publication date2010-09-02
P1433published inActa Pathologica et Microbiologica ScandinavicaQ15756643
P1476titleDistribution of viral RNA, sialic acid receptor, and pathology in H5N1 avian influenza patients
P478volume118

Reverse relations

cites work (P2860)
Q30416451A novel pathogenic mechanism of highly pathogenic avian influenza H5N1 viruses involves hemagglutinin mediated resistance to serum innate inhibitors
Q41816698Adaptation of novel H7N9 influenza A virus to human receptors
Q59791236Complete Genomic Sequences of Highly Pathogenic H5N1 Avian Influenza Viruses Obtained Directly from Human Autopsy Specimens
Q35226000Distribution of sialic acid receptors and influenza A virus of avian and swine origin in experimentally infected pigs
Q90616762HA-Dependent Tropism of H5N1 and H7N9 Influenza Viruses to Human Endothelial Cells Is Determined by Reduced Stability of the HA, Which Allows the Virus To Cope with Inefficient Endosomal Acidification and Constitutively Expressed IFITM3
Q43902374How many trimers? Modeling influenza virus fusion yields a minimum aggregate size of six trimers, three of which are fusogenic
Q57068751Infection of pulmonary vascular endothelial cells by H5N1 avian influenza virus and its role in pathogenesis
Q26800158Modulating the Innate Immune Response to Influenza A Virus: Potential Therapeutic Use of Anti-Inflammatory Drugs
Q38399543Pathological study of archival lung tissues from five fatal cases of avian H5N1 influenza in Vietnam.
Q42276866The expression pattern and histological distribution of sialyltransferases ST3Gal III in yellow chicken
Q37136609Towards multiscale modeling of influenza infection
Q45365675Viral load of the highly pathogenic avian influenza H5N1 virus in infected human tissues
Q30353598Virus-specific factors associated with zoonotic and pandemic potential.

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