scholarly article | Q13442814 |
P356 | DOI | 10.1128/JVI.01223-19 |
P698 | PubMed publication ID | 31597765 |
P50 | author | Mikhail Matrosovich | Q80203681 |
Luca Hensen | Q90616758 | ||
P2093 | author name string | Hans-Dieter Klenk | |
Tatyana Matrosovich | |||
Katrin Roth | |||
P2860 | cites work | IFITM3 directly engages and shuttles incoming virus particles to lysosomes | Q91045937 |
Interferon-Induced Transmembrane Proteins Mediate Viral Evasion in Acute and Chronic Hepatitis C Virus Infection | Q91813039 | ||
Stability of infectious influenza A viruses to treatment at low pH and heating | Q93590259 | ||
IFITM3 restricts influenza A virus entry by blocking the formation of fusion pores following virus-endosome hemifusion | Q27329190 | ||
Recognition of sulphated and fucosylated receptor sialosides by A/Vietnam/1194/2004 (H5N1) influenza virus | Q27685500 | ||
Receptor binding and membrane fusion in virus entry: the influenza hemagglutinin | Q27861017 | ||
Experimental adaptation of an influenza H5 HA confers respiratory droplet transmission to a reassortant H5 HA/H1N1 virus in ferrets | Q29619972 | ||
A DNA transfection system for generation of influenza A virus from eight plasmids | Q29642855 | ||
Avian-virus-like receptor specificity of the hemagglutinin impedes influenza virus replication in cultures of human airway epithelium. | Q30359078 | ||
Avian influenza virus (H5N1): a threat to human health. | Q30361041 | ||
Acid-induced membrane fusion by the hemagglutinin protein and its role in influenza virus biology. | Q30364511 | ||
Role of Substitutions in the Hemagglutinin in the Emergence of the 1968 Pandemic Influenza Virus. | Q30379214 | ||
H1N1 Swine Influenza Viruses Differ from Avian Precursors by a Higher pH Optimum of Membrane Fusion. | Q30381890 | ||
2009 pandemic influenza A (H1N1): pathology and pathogenesis of 100 fatal cases in the United States | Q30389767 | ||
Constitutively Expressed IFITM3 Protein in Human Endothelial Cells Poses an Early Infection Block to Human Influenza Viruses | Q30393767 | ||
Distribution of viral RNA, sialic acid receptor, and pathology in H5N1 avian influenza patients. | Q30394986 | ||
pH Optimum of Hemagglutinin-Mediated Membrane Fusion Determines Sensitivity of Influenza A Viruses to the Interferon-Induced Antiviral State and IFITMs | Q30400470 | ||
Human pulmonary microvascular endothelial cells support productive replication of highly pathogenic avian influenza viruses: possible involvement in the pathogenesis of human H5N1 virus infection | Q30409253 | ||
Enhanced growth of influenza vaccine seed viruses in vero cells mediated by broadening the optimal pH range for virus membrane fusion | Q30409570 | ||
Amphotericin B increases influenza A virus infection by preventing IFITM3-mediated restriction | Q30415173 | ||
Increased acid stability of the hemagglutinin protein enhances H5N1 influenza virus growth in the upper respiratory tract but is insufficient for transmission in ferrets | Q30432710 | ||
Receptor specificity of influenza viruses from birds and mammals: new data on involvement of the inner fragments of the carbohydrate chain. | Q30985426 | ||
Clinical, virological, and histopathological manifestations of fatal human infections by avian influenza A(H7N9) virus | Q33409780 | ||
Single HA2 mutation increases the infectivity and immunogenicity of a live attenuated H5N1 intranasal influenza vaccine candidate lacking NS1. | Q33872175 | ||
Role of receptor binding specificity in influenza A virus transmission and pathogenesis | Q34324562 | ||
High basal expression of interferon-stimulated genes in human bronchial epithelial (BEAS-2B) cells contributes to influenza A virus resistance | Q34337520 | ||
Identification, characterization, and natural selection of mutations driving airborne transmission of A/H5N1 virus | Q35146036 | ||
Inhibitors of the interferon response enhance virus replication in vitro. | Q35414495 | ||
Avian Influenza Virus Infection of Immortalized Human Respiratory Epithelial Cells Depends upon a Delicate Balance between Hemagglutinin Acid Stability and Endosomal pH. | Q35536208 | ||
A(H7N9) virus results in early induction of proinflammatory cytokine responses in both human lung epithelial and endothelial cells and shows increased human adaptation compared with avian H5N1 virus | Q35640967 | ||
Endocytosis of influenza viruses | Q35862726 | ||
Receptor-binding profiles of H7 subtype influenza viruses in different host species | Q35868067 | ||
Primate lentiviruses are differentially inhibited by interferon-induced transmembrane proteins. | Q36090006 | ||
Determinants of [Cl-] in recycling and late endosomes and Golgi complex measured using fluorescent ligands | Q36323160 | ||
Second messengers regulate endosomal acidification in Swiss 3T3 fibroblasts | Q36531965 | ||
Amino acid residues in the fusion peptide pocket regulate the pH of activation of the H5N1 influenza virus hemagglutinin protein. | Q37145184 | ||
Amino acid residues at positions 222 and 227 of the hemagglutinin together with the neuraminidase determine binding of H5 avian influenza viruses to sialyl Lewis X. | Q37270231 | ||
Hemagglutinin-dependent tropism of H5N1 avian influenza virus for human endothelial cells. | Q37452450 | ||
Epidemiology of low pathogenic avian influenza viruses in wild birds | Q37556389 | ||
Influenza Virus Overcomes Cellular Blocks To Productively Replicate, Impacting Macrophage Function | Q37560736 | ||
Neuraminidase is important for the initiation of influenza virus infection in human airway epithelium | Q37596932 | ||
Highly Pathogenic Influenza A(H5Nx) Viruses with Altered H5 Receptor-Binding Specificity | Q37664374 | ||
Endothelial cell tropism is a determinant of H5N1 pathogenesis in mammalian species | Q37716213 | ||
Pathogenesis of influenza virus infections: the good, the bad and the ugly | Q38019628 | ||
Endothelial activation and dysfunction in the pathogenesis of influenza A virus infection | Q38122282 | ||
IFITMs restrict the replication of multiple pathogenic viruses | Q38146443 | ||
Receptor binding and pH stability - how influenza A virus hemagglutinin affects host-specific virus infection | Q38155706 | ||
Entry of influenza A virus: host factors and antiviral targets. | Q38162340 | ||
Pathogenesis of influenza-induced acute respiratory distress syndrome. | Q38163209 | ||
Influenza pathobiology and pathogenesis in avian species | Q38228594 | ||
Influenza virus and endothelial cells: a species specific relationship | Q38292659 | ||
IFITM-Family Proteins: The Cell's First Line of Antiviral Defense | Q38323041 | ||
pH regulation in early endosomes and interferon-inducible transmembrane proteins control avian retrovirus fusion | Q38710609 | ||
Solid-phase assays of receptor-binding specificity. | Q39360142 | ||
The Interplay between the Host Receptor and Influenza Virus Hemagglutinin and Neuraminidase | Q39438794 | ||
Mutations affecting the stability of the haemagglutinin molecule impair the immunogenicity of live attenuated H3N2 intranasal influenza vaccine candidates lacking NS1. | Q39575832 | ||
Evolution, global spread, and pathogenicity of highly pathogenic avian influenza H5Nx clade 2.3.4.4. | Q40062832 | ||
Unique Infectious Strategy of H5N1 Avian Influenza Virus Is Governed by the Acid-Destabilized Property of Hemagglutinin | Q40144005 | ||
Influenza H5N1 virus infection of polarized human alveolar epithelial cells and lung microvascular endothelial cells. | Q42569973 | ||
Influenza Hemagglutinin Protein Stability, Activation, and Pandemic Risk. | Q54230830 | ||
Host and viral determinants of influenza A virus species specificity | Q59349660 | ||
Receptor-binding properties of influenza viruses isolated from gulls | Q59356757 | ||
Interferon-induced transmembrane protein 3 blocks fusion of sensitive but not resistant viruses by partitioning into virus-carrying endosomes | Q61818410 | ||
Infection of the endothelium by influenza viruses | Q81094162 | ||
P433 | issue | 1 | |
P577 | publication date | 2019-12-12 | |
P1433 | published in | Journal of Virology | Q1251128 |
P1476 | title | HA-Dependent Tropism of H5N1 and H7N9 Influenza Viruses to Human Endothelial Cells Is Determined by Reduced Stability of the HA, Which Allows the Virus To Cope with Inefficient Endosomal Acidification and Constitutively Expressed IFITM3 | |
P478 | volume | 94 |
Q90393978 | Topology, Antiviral Functional Residues and Mechanism of IFITM1 | cites work | P2860 |
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