| scholarly article | Q13442814 |
| P50 | author | Marina Botto | Q56247130 |
| P2093 | author name string | H Terence Cook | |
| Katherine A Vernon | |||
| Matthew C Pickering | |||
| Marieta M Ruseva | |||
| Marina Botto | |||
| Talat H Malik | |||
| P2860 | cites work | Three kidneys, two diseases, one antibody? | Q33396530 |
| Membrano-proliferative glomerulonephritis, atypical hemolytic uremic syndrome, and a new complement factor H mutation: report of a case | Q33401621 | ||
| Prevention of C5 activation ameliorates spontaneous and experimental glomerulonephritis in factor H-deficient mice | Q34537391 | ||
| Familial C3 glomerulopathy associated with CFHR5 mutations: clinical characteristics of 91 patients in 16 pedigrees | Q35030648 | ||
| Hereditary porcine membranoproliferative glomerulonephritis type II is caused by factor H deficiency | Q35552728 | ||
| Acute presentation and persistent glomerulonephritis following streptococcal infection in a patient with heterozygous complement factor H-related protein 5 deficiency | Q36057084 | ||
| Loss of properdin exacerbates C3 glomerulopathy resulting from factor H deficiency | Q36509318 | ||
| C3 glomerulopathy: consensus report | Q37348685 | ||
| Toward a working definition of C3 glomerulopathy by immunofluorescence | Q38394841 | ||
| Factor H facilitates the clearance of GBM bound iC3b by controlling C3 activation in fluid phase | Q41443111 | ||
| Accelerated nephrotoxic nephritis is exacerbated in C1q-deficient mice | Q43612859 | ||
| Targeted deletion of 5'HS2 of the murine beta-globin LCR reveals that it is not essential for proper regulation of the beta-globin locus. | Q52205977 | ||
| Uncontrolled C3 activation causes membranoproliferative glomerulonephritis in mice deficient in complement factor H | Q57089510 | ||
| Composition of nephritic factor-generated glomerular deposits in membranoproliferative glomerulonephritis type 2 | Q73947390 | ||
| Mouse podocyte complement factor H: the functional analog to human complement receptor 1 | Q79896677 | ||
| The alternative pathway of complement is activated in the glomeruli and tubulointerstitium of mice with adriamycin nephropathy | Q80375869 | ||
| Identification of a mutation in complement factor H-related protein 5 in patients of Cypriot origin with glomerulonephritis | Q24615589 | ||
| Factor I is required for the development of membranoproliferative glomerulonephritis in factor H-deficient mice | Q24651118 | ||
| Atypical haemolytic uraemic syndrome with underlying glomerulopathies. A case series and a review of the literature | Q26864297 | ||
| The development of atypical hemolytic uremic syndrome depends on complement C5 | Q28300378 | ||
| Dual roles for glucokinase in glucose homeostasis as determined by liver and pancreatic beta cell-specific gene knock-outs using Cre recombinase | Q28508079 | ||
| A highly efficient recombineering-based method for generating conditional knockout mutations | Q29615157 | ||
| Complement factor H mutations and gene polymorphisms in haemolytic uraemic syndrome: the C-257T, the A2089G and the G2881T polymorphisms are strongly associated with the disease | Q33357336 | ||
| Phenotypic expression of factor H mutations in patients with atypical hemolytic uremic syndrome. | Q33370511 | ||
| Primary glomerulonephritis with isolated C3 deposits: a new entity which shares common genetic risk factors with haemolytic uraemic syndrome | Q33372850 | ||
| Spontaneous hemolytic uremic syndrome triggered by complement factor H lacking surface recognition domains | Q33375335 | ||
| Translational mini-review series on complement factor H: genetics and disease associations of human complement factor H. | Q33377799 | ||
| Translational mini-review series on complement factor H: renal diseases associated with complement factor H: novel insights from humans and animals. | Q33378236 | ||
| Complement factor H deficiency and posttransplantation glomerulonephritis with isolated C3 deposits | Q33379098 | ||
| Atypical hemolytic uremic syndrome associated with mutations in complement regulator genes | Q33392010 | ||
| Thrombotic microangiopathy mimicking membranoproliferative glomerulonephritis. | Q33396467 | ||
| P433 | issue | 5 | |
| P407 | language of work or name | English | Q1860 |
| P304 | page(s) | 1334-1342 | |
| P577 | publication date | 2015-09-15 | |
| P1433 | published in | Journal of the American Society of Nephrology | Q17123893 |
| P1476 | title | Partial Complement Factor H Deficiency Associates with C3 Glomerulopathy and Thrombotic Microangiopathy | |
| P478 | volume | 27 |
| Q55255568 | A computational model for the evaluation of complement system regulation under homeostasis, disease, and drug intervention. |
| Q37546464 | Association among Complement Factor H Autoantibodies, Deletions of CFHR, and the Risk of Atypical Hemolytic Uremic Syndrome |
| Q50049007 | Complement in cancer: untangling an intricate relationship. |
| Q58603887 | Complements are involved in alcoholic fatty liver disease, hepatitis and fibrosis |
| Q28072499 | Diacylglycerol Kinase-ε: Properties and Biological Roles |
| Q38991026 | Endothelial cells: source, barrier, and target of defensive mediators |
| Q90871287 | Endothelium structure and function in kidney health and disease |
| Q61651844 | Hyperfunctional complement C3 promotes C5-dependent atypical hemolytic uremic syndrome in mice |
| Q55347709 | Mesenchymal Stem Cells Control Complement C5 Activation by Factor H in Lupus Nephritis. |
| Q38770928 | Mutations in Complement Factor H Impair Alternative Pathway Regulation on Mouse Glomerular Endothelial Cells in Vitro |
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