| scholarly article | Q13442814 |
| P2093 | author name string | Yoram Rudy | |
| Livia Hool | |||
| Namit Gaur | |||
| P2860 | cites work | Sex, age, and regional differences in L-type calcium current are important determinants of arrhythmia phenotype in rabbit hearts with drug-induced long QT type 2 | Q28751737 |
| Arrhythmias in the congenital long QT syndrome: how often is torsade de pointes pause dependent? | Q33920608 | ||
| The role of stochastic and modal gating of cardiac L-type Ca2+ channels on early after-depolarizations | Q33947478 | ||
| Mechanism of ventricular arrhythmias in the long QT syndrome: on hermeneutics | Q34087469 | ||
| Action potential and contractility changes in [Na(+)](i) overloaded cardiac myocytes: a simulation study | Q34173006 | ||
| Molecular and cellular mechanisms of cardiac arrhythmias | Q34175373 | ||
| Ionic charge conservation and long-term steady state in the Luo-Rudy dynamic cell model | Q34177010 | ||
| Regulation and modulation of calcium channels in cardiac, skeletal, and smooth muscle cells | Q34340353 | ||
| Mechanisms of arrhythmogenic delayed and early afterdepolarizations in ferret ventricular muscle | Q34545690 | ||
| Redox signaling in oxygen sensing by vessels | Q34749340 | ||
| Computational biology in the study of cardiac ion channels and cell electrophysiology. | Q36015209 | ||
| Acute hypoxia differentially regulates K(+) channels. Implications with respect to cardiac arrhythmia. | Q36052206 | ||
| Antiarrhythmic action of beta-blockers: potential mechanisms | Q36167672 | ||
| The cardiac persistent sodium current: an appealing therapeutic target? | Q37028768 | ||
| The mechanism of acute hypoxic pulmonary vasoconstriction: the tale of two channels | Q40545374 | ||
| Modulation of recombinant human cardiac L-type Ca2+ channel alpha1C subunits by redox agents and hypoxia | Q40981937 | ||
| Early afterdepolarizations: mechanism of induction and block. A role for L-type Ca2+ current | Q41256047 | ||
| Differential regulation of the slow and rapid components of guinea-pig cardiac delayed rectifier K+ channels by hypoxia | Q42453233 | ||
| Hypoxia increases the sensitivity of the L-type Ca(2+) current to beta-adrenergic receptor stimulation via a C2 region-containing protein kinase C isoform | Q42496978 | ||
| Hypoxia alters the sensitivity of the L-type Ca(2+) channel to alpha-adrenergic receptor stimulation in the presence of beta-adrenergic receptor stimulation | Q42504872 | ||
| Reduction of Ca(2+) channel activity by hypoxia in human and porcine coronary myocytes. | Q42514176 | ||
| Decreasing cellular hydrogen peroxide with catalase mimics the effects of hypoxia on the sensitivity of the L-type Ca2+ channel to beta-adrenergic receptor stimulation in cardiac myocytes | Q42526610 | ||
| Pharmacological separation of early afterdepolarizations from arrhythmogenic substrate in DeltaKPQ Scn5a murine hearts modelling human long QT 3 syndrome | Q42729116 | ||
| Hypoxia causes downregulation of protein and RNA synthesis in noncontracting Mammalian cardiomyocytes. | Q43966254 | ||
| Calmodulin kinase II and arrhythmias in a mouse model of cardiac hypertrophy | Q44123766 | ||
| O2 sensing in the human ductus arteriosus: regulation of voltage-gated K+ channels in smooth muscle cells by a mitochondrial redox sensor | Q44145585 | ||
| Electrophysiologic effects of acute myocardial ischemia: a theoretical study of altered cell excitability and action potential duration | Q46239877 | ||
| Beta-adrenergic and cholinergic modulation of inward rectifier K+ channel function and phosphorylation in guinea-pig ventricle | Q46305956 | ||
| A dynamic model of the cardiac ventricular action potential. I. Simulations of ionic currents and concentration changes | Q46410988 | ||
| Role of NAD(P)H oxidase in the regulation of cardiac L-type Ca2+ channel function during acute hypoxia | Q46507230 | ||
| Redox reaction modulates transient and persistent sodium current during hypoxia in guinea pig ventricular myocytes | Q46689827 | ||
| Regulation of caveolar cardiac sodium current by a single Gsalpha histidine residue. | Q46748407 | ||
| Hypoxia increases persistent sodium current in rat ventricular myocytes. | Q51556510 | ||
| A conservation principle and its effect on the formulation of Na-Ca exchanger current in cardiac cells. | Q52252561 | ||
| Splice variants reveal the region involved in oxygen sensing by recombinant human L-type Ca(2+) channels. | Q52863647 | ||
| Hypoxia inhibits the recombinant alpha 1C subunit of the human cardiac L-type Ca2+ channel. | Q52868122 | ||
| Distinct voltage-dependent regulation of a heart-delayed IK by protein kinases A and C. | Q53854275 | ||
| Sudden Cardiac Death | Q56593283 | ||
| Inactivation-resistant channels underlying the persistent sodium current in rat ventricular myocytes | Q72055880 | ||
| Systemic administration of calmodulin antagonist W-7 or protein kinase A inhibitor H-8 prevents torsade de pointes in rabbits | Q73267709 | ||
| In vivo mechanisms precipitating torsades de pointes in a canine model of drug-induced long-QT1 syndrome | Q80721442 | ||
| P433 | issue | 12 | |
| P921 | main subject | hypoxia | Q105688 |
| P304 | page(s) | 1196-1203 | |
| P577 | publication date | 2009-10-29 | |
| P1433 | published in | Circulation Research | Q2599020 |
| P1476 | title | Contributions of ion channel currents to ventricular action potential changes and induction of early afterdepolarizations during acute hypoxia | |
| P478 | volume | 105 |
| Q37441977 | A new method to detect rapid oxygen changes around cells: how quickly do calcium channels sense oxygen in cardiomyocytes? |
| Q41917870 | Adrenergic signaling controls RGK-dependent trafficking of cardiac voltage-gated L-type Ca2+ channels through PKD1. |
| Q28067531 | Altered Calcium Handling and Ventricular Arrhythmias in Acute Ischemia |
| Q42138057 | Arrhythmogenic right ventricular cardiomyopathy: considerations from in silico experiments |
| Q39328451 | Carbon monoxide effects on human ventricle action potential assessed by mathematical simulations. |
| Q33721787 | Cardiac BIN1 folds T-tubule membrane, controlling ion flux and limiting arrhythmia |
| Q90444009 | Cardiac function dependence on carbon monoxide |
| Q36992757 | Constitutive Intracellular Na+ Excess in Purkinje Cells Promotes Arrhythmogenesis at Lower Levels of Stress Than Ventricular Myocytes From Mice With Catecholaminergic Polymorphic Ventricular Tachycardia |
| Q92226110 | Delayed afterdepolarization-induced triggered activity in cardiac purkinje cells mediated through cytosolic calcium diffusion waves |
| Q60050731 | Determinants of early afterdepolarization properties in ventricular myocyte models |
| Q54265842 | Endothelin-1 stimulates the expression of L-type Ca2+ channels in neonatal rat cardiomyocytes via the extracellular signal-regulated kinase 1/2 pathway. |
| Q34213428 | High purity human-induced pluripotent stem cell-derived cardiomyocytes: electrophysiological properties of action potentials and ionic currents. |
| Q89787652 | Hypoxia Produces Pro-arrhythmic Late Sodium Current in Cardiac Myocytes by SUMOylation of NaV1.5 Channels |
| Q55119914 | Impact of Different Serum Potassium Levels on Postresuscitation Heart Function and Hemodynamics in Patients with Nontraumatic Out-of-Hospital Cardiac Arrest. |
| Q43777125 | Ionic mechanisms of electrophysiological properties and repolarization abnormalities in rabbit Purkinje fibers |
| Q38767537 | NHE inhibition does not improve Na(+) or Ca(2+) overload during reperfusion: using modeling to illuminate the mechanisms underlying a therapeutic failure |
| Q44946056 | Ranolazine is an Effective and Safe Treatment of Adults with Symptomatic Premature Ventricular Contractions due to Triggered Ectopy |
| Q46402706 | The cardiac L-type calcium channel alpha subunit is a target for direct redox modification during oxidative stress-the role of cysteine residues in the alpha interacting domain. |
| Q41374450 | The relative influences of phosphometabolites and pH on action potential morphology during myocardial reperfusion: a simulation study |
| Q36000078 | β-Adrenergic stimulation and rapid pacing mutually promote heterogeneous electrical failure and ventricular fibrillation in the globally ischemic heart |
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